Steven Yea

1.7k total citations
9 papers, 598 citations indexed

About

Steven Yea is a scholar working on Molecular Biology, Epidemiology and Genetics. According to data from OpenAlex, Steven Yea has authored 9 papers receiving a total of 598 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 2 papers in Epidemiology and 2 papers in Genetics. Recurrent topics in Steven Yea's work include Kruppel-like factors research (5 papers), Cancer-related gene regulation (4 papers) and Genetic Syndromes and Imprinting (2 papers). Steven Yea is often cited by papers focused on Kruppel-like factors research (5 papers), Cancer-related gene regulation (4 papers) and Genetic Syndromes and Imprinting (2 papers). Steven Yea collaborates with scholars based in United States, China and Spain. Steven Yea's co-authors include Scott L. Friedman, Goutham Narla, Johnny Loke, Martin J. Walsh, Mirko Tarocchi, John J. Sninsky, Guo J, Feng Hong, Hongjin Huang and Michaela S. Banck and has published in prestigious journals such as Journal of Biological Chemistry, Gastroenterology and Hepatology.

In The Last Decade

Steven Yea

8 papers receiving 593 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Steven Yea United States	8	384	197	138	98	77	9	598
Hasmik Mkrtchyan United States	8	130 0.3×	190 1.0×	94 0.7×	22 0.2×	57 0.7×	12	392
Masumi Yoshie Japan	8	191 0.5×	51 0.3×	94 0.7×	35 0.4×	108 1.4×	15	392
Yuichi Tsuchiya Japan	10	528 1.4×	176 0.9×	37 0.3×	19 0.2×	223 2.9×	21	805
Zhao-na Yang China	8	255 0.7×	98 0.5×	47 0.3×	17 0.2×	89 1.2×	8	460
Sandra Schmidhofer Germany	8	233 0.6×	110 0.6×	27 0.2×	26 0.3×	29 0.4×	9	412
Jamil Haque United States	9	303 0.8×	89 0.5×	218 1.6×	27 0.3×	52 0.7×	9	580
Robert Sherwin United States	4	267 0.7×	117 0.6×	47 0.3×	97 1.0×	74 1.0×	7	478
Sandrine Cariou France	7	257 0.7×	123 0.6×	278 2.0×	18 0.2×	44 0.6×	7	538
Takashi Morizono Japan	11	458 1.2×	95 0.5×	79 0.6×	188 1.9×	299 3.9×	15	777

Countries citing papers authored by Steven Yea

Since Specialization

Citations

This map shows the geographic impact of Steven Yea's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Steven Yea with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Steven Yea more than expected).

Fields of papers citing papers by Steven Yea

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Steven Yea. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Steven Yea. The network helps show where Steven Yea may publish in the future.

Co-authorship network of co-authors of Steven Yea

This figure shows the co-authorship network connecting the top 25 collaborators of Steven Yea. A scholar is included among the top collaborators of Steven Yea based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Steven Yea. Steven Yea is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Yea, Steven, et al.. (2013). The miR-183~96~182 cluster promotes tumorigenesis in a mouse model of medulloblastoma. Journal of Biomedical Research. 27(6). 486–486. 20 indexed citations

2.

Ghiassi–Nejad, Zahra, Andrew J. Paris, Steven Yea, et al.. (2010). Tumor suppressor activity of KLF6 mediated by downregulation of the PTTG1 oncogene. FEBS Letters. 584(5). 1006–1010. 16 indexed citations

3.

J, Guo, Hong Feng, Johnny Loke, et al.. (2009). A DDX5 S480A Polymorphism Is Associated with Increased Transcription of Fibrogenic Genes in Hepatic Stellate Cells. Journal of Biological Chemistry. 285(8). 5428–5437. 31 indexed citations

4.

Yea, Steven, Goutham Narla, Xiao Zhao, et al.. (2008). Ras Promotes Growth by Alternative Splicing-Mediated Inactivation of the KLF6 Tumor Suppressor in Hepatocellular Carcinoma. Gastroenterology. 134(5). 1521–1531. 84 indexed citations

5.

J, Guo, Johnny Loke, Feng Zheng, et al.. (2008). Functional linkage of cirrhosis‐predictive single nucleotide polymorphisms of toll‐like receptor 4 to hepatic stellate cell responses†‡. Hepatology. 49(3). 960–968. 183 indexed citations

6.

Lemmer, Eric R., Ying‐Bei Chen, Steven Yea, et al.. (2006). Dysregulation of hedgehog pathway signaling in hepatocellular carcinoma. Cancer Research. 66. 630–630.

7.

Narla, Goutham, Ying‐Bei Chen, Eldad A. Hod, et al.. (2006). Downregulation of KLF6 is an early event in hepatocarcinogenesis, and stimulates proliferation while reducing differentiation. Journal of Hepatology. 46(4). 645–654. 71 indexed citations

8.

Li, Dan, Steven Yea, SiDe Li, et al.. (2005). Krüppel-like Factor-6 Promotes Preadipocyte Differentiation through Histone Deacetylase 3-dependent Repression of DLK1. Journal of Biological Chemistry. 280(29). 26941–26952. 144 indexed citations

9.

Li, Dan, Steven Yea, Georgia Dolios, et al.. (2005). Regulation of Kruppel-like Factor 6 Tumor Suppressor Activity by Acetylation. Cancer Research. 65(20). 9216–9225. 49 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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