Stephen H. Schilling

635 total citations
10 papers, 555 citations indexed

About

Stephen H. Schilling is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Oncology. According to data from OpenAlex, Stephen H. Schilling has authored 10 papers receiving a total of 555 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 2 papers in Pathology and Forensic Medicine and 2 papers in Oncology. Recurrent topics in Stephen H. Schilling's work include TGF-β signaling in diseases (5 papers), NF-κB Signaling Pathways (2 papers) and Bone Metabolism and Diseases (2 papers). Stephen H. Schilling is often cited by papers focused on TGF-β signaling in diseases (5 papers), NF-κB Signaling Pathways (2 papers) and Bone Metabolism and Diseases (2 papers). Stephen H. Schilling collaborates with scholars based in United States, Australia and Bulgaria. Stephen H. Schilling's co-authors include Rik Derynck, Xiao‐Fan Wang, Kristin A. Knouse, Lisa Choy, M. Balooch, Ellen Filvaroff, Sally J. Marshall, Grayson W. Marshall, Tamara Alliston and R.K. Nalla and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Stephen H. Schilling

9 papers receiving 546 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Stephen H. Schilling United States 8 404 108 86 53 49 10 555
Wai Fook Leong Singapore 11 330 0.8× 159 1.5× 41 0.5× 25 0.5× 51 1.0× 14 497
Shan-Chuan Zhao United States 8 487 1.2× 101 0.9× 35 0.4× 18 0.3× 39 0.8× 9 648
T-C He United States 5 332 0.8× 65 0.6× 94 1.1× 21 0.4× 16 0.3× 6 609
Nicole Stuendl Austria 15 223 0.6× 108 1.0× 51 0.6× 16 0.3× 21 0.4× 17 456
Christine Stewart United States 8 483 1.2× 196 1.8× 105 1.2× 11 0.2× 53 1.1× 10 713
Wenkan Zhang China 14 221 0.5× 120 1.1× 46 0.5× 14 0.3× 38 0.8× 27 465
Rieko Yagi United States 9 533 1.3× 67 0.6× 110 1.3× 33 0.6× 12 0.2× 12 766
Eva Luegmayr Austria 11 245 0.6× 203 1.9× 30 0.3× 13 0.2× 125 2.6× 14 502
Jelena Kocić Serbia 12 285 0.7× 86 0.8× 57 0.7× 8 0.2× 28 0.6× 20 558
Sandra Parenti Italy 14 228 0.6× 75 0.7× 26 0.3× 9 0.2× 19 0.4× 27 430

Countries citing papers authored by Stephen H. Schilling

Since Specialization
Citations

This map shows the geographic impact of Stephen H. Schilling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stephen H. Schilling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stephen H. Schilling more than expected).

Fields of papers citing papers by Stephen H. Schilling

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stephen H. Schilling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stephen H. Schilling. The network helps show where Stephen H. Schilling may publish in the future.

Co-authorship network of co-authors of Stephen H. Schilling

This figure shows the co-authorship network connecting the top 25 collaborators of Stephen H. Schilling. A scholar is included among the top collaborators of Stephen H. Schilling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stephen H. Schilling. Stephen H. Schilling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Schilling, Stephen H.. (2011). DNA as patentable subject matter and a narrow framework for addressing the perceived problems caused by gene patents.. PubMed. 61(3). 731–73.
2.
Radiloff, Daniel R., Timothy P. Wakeman, Junjie Feng, et al.. (2011). Trefoil factor 1 acts to suppress senescence induced by oncogene activation during the cellular transformation process. Proceedings of the National Academy of Sciences. 108(16). 6591–6596. 22 indexed citations
3.
Schilling, Stephen H., Anita B. Hjelmeland, Daniel R. Radiloff, et al.. (2009). NDRG4 Is Required for Cell Cycle Progression and Survival in Glioblastoma Cells. Journal of Biological Chemistry. 284(37). 25160–25169. 47 indexed citations
4.
Jamaï, Aziz, Patrice A. Salomé, Stephen H. Schilling, Andreas P.M. Weber, & C. Robertson McClung. (2009). Arabidopsis Photorespiratory Serine Hydroxymethyltransferase Activity Requires the Mitochondrial Accumulation of Ferredoxin-Dependent Glutamate Synthase. The Plant Cell. 21(2). 595–606. 70 indexed citations
5.
Schilling, Stephen H., Anita B. Hjelmeland, Jeremy N. Rich, & Xiao‐Fan Wang. (2008). 3 TGF-β: A Multipotential Cytokine. Cold Spring Harbor Monograph Archive. 50. 45–77. 1 indexed citations
6.
Schilling, Stephen H., et al.. (2008). TGFβ‐stimulated Smad1/5 phosphorylation requires the ALK5 L45 loop and mediates the pro‐migratory TGFβ switch. The EMBO Journal. 28(2). 88–98. 150 indexed citations
7.
Schilling, Stephen H., Michael Datto, & Xiao‐Fan Wang. (2006). A Phosphatase Controls the Fate of Receptor-Regulated Smads. Cell. 125(5). 838–840. 15 indexed citations
8.
Chow, Edward Kai‐Hua, Ryan M. O’Connell, Stephen H. Schilling, et al.. (2005). TLR agonists regulate PDGF‐B production and cell proliferation through TGF‐β/type I IFN crosstalk. The EMBO Journal. 24(23). 4071–4081. 39 indexed citations
9.
Balooch, Guive, M. Balooch, R.K. Nalla, et al.. (2005). TGF-β regulates the mechanical properties and composition of bone matrix. Proceedings of the National Academy of Sciences. 102(52). 18813–18818. 181 indexed citations
10.
Hjelmeland, Anita B., Stephen H. Schilling, Xing Guo, L. Darryl Quarles, & Xiao‐Fan Wang. (2005). Loss of Smad3-Mediated Negative Regulation of Runx2 Activity Leads to an Alteration in Cell Fate Determination. Molecular and Cellular Biology. 25(21). 9460–9468. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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