Stephen E. Goldfinger
- Molecular Biology top 10%
- Nephrology top 2%
- Immunology top 10%
- Rheumatology top 10%
- Surgery
- Co-authors
- J. Edwin SeegmillerJames R. KlinenbergDavid W. AllingDavid C. DaleCharles A. DinarelloSheldon WolffWilliam D. HeizerVincent G. Zannoni
- Topics
- Gout, Hyperuricemia, Uric Acid (6 papers)Child Nutrition and Feeding Issues (3 papers)Inflammasome and immune disorders (3 papers)
- Partner nations
- United StatesPolandMalaysia
In The Last Decade
Stephen E. Goldfinger
25 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 136
- Molecular Biology 719
- Nephrology 325
- Immunology 256
- Rheumatology 155
- Surgery 143
Countries citing papers authored by Stephen E. Goldfinger
This map shows the geographic impact of Stephen E. Goldfinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stephen E. Goldfinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stephen E. Goldfinger more than expected).
Fields of papers citing papers by Stephen E. Goldfinger
This network shows the impact of papers produced by Stephen E. Goldfinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stephen E. Goldfinger. The network helps show where Stephen E. Goldfinger may publish in the future.
Co-authorship network of co-authors of Stephen E. Goldfinger
This figure shows the co-authorship network connecting the top 25 collaborators of Stephen E. Goldfinger. A scholar is included among the top collaborators of Stephen E. Goldfinger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stephen E. Goldfinger. Stephen E. Goldfinger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | The inherited autoinflammatory syndrome: a decade of discovery. | 24 |
| 2 | 1 | |
| 3 | 21 | |
| 4 | Your good health : how to stay well, and what to do when you're not | 1 |
| 5 | 31 | |
| 6 | 5 | |
| 7 | 0 | |
| 8 | 3 | |
| 9 | 9 | |
| 10 | 275 | |
| 11 | 3 | |
| 12 | 251 | |
| 13 | 13 | |
| 14 | 21 | |
| 15 | 8 | |
| 16 | 2 | |
| 17 | 8 | |
| 18 | 56 | |
| 19 | 90 | |
| 20 | 44 |
About Stephen E. Goldfinger
Stephen E. Goldfinger is a scholar working on Nephrology, Gastroenterology and Health Information Management, having authored 28 papers that have together received 1.3k indexed citations. Recurring topics across this work include Gout, Hyperuricemia, Uric Acid (6 papers), Child Nutrition and Feeding Issues (3 papers) and Inflammasome and immune disorders (3 papers). The work is most often cited by research in Nephrology (325 citations), Clinical Biochemistry (128 citations) and Immunology (256 citations). Stephen E. Goldfinger has collaborated with scholars based in United States, Poland and Malaysia. Frequent co-authors include J. Edwin Seegmiller, James R. Klinenberg, David W. Alling, David C. Dale, Charles A. Dinarello, Sheldon Wolff, William D. Heizer, Vincent G. Zannoni, Kenneth L. Melmon and Marion E. Webster. Their work appears in journals such as New England Journal of Medicine, JAMA and Journal of Clinical Investigation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.