Stanley Venitt

779 total citations
18 papers, 631 citations indexed

About

Stanley Venitt is a scholar working on Cancer Research, Molecular Biology and Genetics. According to data from OpenAlex, Stanley Venitt has authored 18 papers receiving a total of 631 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Cancer Research, 6 papers in Molecular Biology and 4 papers in Genetics. Recurrent topics in Stanley Venitt's work include Carcinogens and Genotoxicity Assessment (10 papers), DNA Repair Mechanisms (6 papers) and Effects and risks of endocrine disrupting chemicals (2 papers). Stanley Venitt is often cited by papers focused on Carcinogens and Genotoxicity Assessment (10 papers), DNA Repair Mechanisms (6 papers) and Effects and risks of endocrine disrupting chemicals (2 papers). Stanley Venitt collaborates with scholars based in United Kingdom, Switzerland and Germany. Stanley Venitt's co-authors include David H. Phillips, C. Crofton-Sleigh, Francesco De Matteis, Lewis L. Smith, Alan Hewer, Adrian Davies, Michael R. Stratton, William Warren, Patrick J. Biggs and E.M. Tarmy and has published in prestigious journals such as Biochemical and Biophysical Research Communications, Environmental Health Perspectives and Journal of Medicinal Chemistry.

In The Last Decade

Stanley Venitt

18 papers receiving 597 citations

Peers

Stanley Venitt

GENET

ONCOL

HTM

C. Crofton-Sleigh United Kingdom

Susumu Akasaka Japan

J G Liehr United States

Wendy S. Kennan United States

Harri Vainio Finland

LORRAINE C. GRAND United States

Kim J. Rich United Kingdom

Indra Dwivedy India

Toyohiko Aoki Japan

G. L. Gleason United States

C. Crofton-Sleigh United Kingdom

Stanley Venitt

240 ×0.9

200 ×0.8

235 ×1.0

GENET

85 ×0.6

ONCOL

85 ×0.8

HTM

Citations per year, relative to Stanley Venitt Stanley Venitt (= 1×) peers C. Crofton-Sleigh

Countries citing papers authored by Stanley Venitt

Since Specialization

Citations

This map shows the geographic impact of Stanley Venitt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stanley Venitt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stanley Venitt more than expected).

Fields of papers citing papers by Stanley Venitt

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stanley Venitt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stanley Venitt. The network helps show where Stanley Venitt may publish in the future.

Co-authorship network of co-authors of Stanley Venitt

This figure shows the co-authorship network connecting the top 25 collaborators of Stanley Venitt. A scholar is included among the top collaborators of Stanley Venitt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stanley Venitt. Stanley Venitt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Martin, Francis L., et al.. (1999). The DNA repair inhibitors hydroxyurea and cytosine arabinoside enhance the sensitivity of the alkaline single-cell gel electrophoresis (`comet') assay in metabolically-competent MCL-5 cells. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 445(1). 21–43. 66 indexed citations

Blain, Peter G., et al.. (1998). Consideration of short-term carcinogenicity tests using transgenic mouse models. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 403(1-2). 259–263. 5 indexed citations

Martin, Francis L., Wolfgang Pfau, Kathleen J. Cole, et al.. (1998). Morphological Transformation of C3H/M2 Mouse Fibroblasts by Extracts of Human Mammary Lipid. Biochemical and Biophysical Research Communications. 251(1). 182–189. 10 indexed citations

Venitt, Stanley, C. Crofton-Sleigh, Mavis Agbandje, Terence C. Jenkins, & Stephen Neidle. (1998). Anthracene-9,10-diones as Potential Anticancer Agents: Bacterial Mutation Studies of Amido-Substituted Derivatives Reveal an Unexpected Lack of Mutagenicity. Journal of Medicinal Chemistry. 41(19). 3748–3752. 33 indexed citations

Spigelman, Allan D., et al.. (1997). The use of 32P-postlabelling in studies of the nature and origin of DNA adducts formed by bile from patients with familial adenomatous polyposis and from normal patients. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 378(1-2). 113–125. 9 indexed citations

Venitt, Stanley. (1996). Mechanisms of Spontaneous Human Cancers. Environmental Health Perspectives. 104. 633–633. 4 indexed citations

Phillips, David H., et al.. (1994). Reduced genotoxicity of [D₅-ethyl]-tamoxifen implicates α-hydroxylation of the ethyl group as a major pathway of tamoxifen activation to a liver carcinogen. Carcinogenesis. 15(8). 1487–1492. 66 indexed citations

Biggs, Patrick J., William Warren, Stanley Venitt, & Michael R. Stratton. (1993). Does a genotoxic carcinogen contribute to human breast cancer?. Mutagenesis. 8(4). 275–283. 83 indexed citations

Spigelman, Allan D., et al.. (1992). DNA adducts detected by ³²P-postlabelling, in the intestine of rats given bile from patients with familial adenomatous polyposis and from unaffected controls. Carcinogenesis. 13(4). 731–735. 22 indexed citations

10.

Matteis, Francesco De, Adrian Davies, Lewis L. Smith, et al.. (1992). Genotoxic potential of tamoxifen and analogues in female Fischer F344/n rats, DBA/2 and C57BL/6 mice and in human MCL-5 cells. Carcinogenesis. 13(12). 2197–2203. 207 indexed citations

11.

Spigelman, Allan D., et al.. (1991). DNA adducts, detected by ³²P-postlabelling, in the foregut of patients with familial adenomatous polyposis and in unaffected controls. Carcinogenesis. 12(9). 1727–1732. 32 indexed citations

12.

Venitt, Stanley, et al.. (1989). A study of the heterogeneity of bacterial fluctuation-test data and the effects of auxotrophic-growth enhancement. Mutagenesis. 4(2). 126–132. 3 indexed citations

13.

Crofton-Sleigh, C., et al.. (1987). A forward mutation assay using ampicillin-resistance in Escherichia coli designed for investigating the mutagenicity of biological samples. Mutagenesis. 2(6). 455–467. 5 indexed citations

14.

Venitt, Stanley, et al.. (1986). Testing human faecal extracts for genotoxic activity with the SOS Chromotest: the importance of controlling for faecal enzyme activity. Mutagenesis. 1(2). 143–149. 7 indexed citations

15.

Venitt, Stanley, et al.. (1986). Further studies on the detection of mutagenic and genotoxic activity in human faeces: aerobic and anaerobic fluctuation tests with S. typhimurium and E. coli, and the SOS Chromotest. Mutagenesis. 1(1). 49–64. 13 indexed citations

16.

Gingold, Elliot B., et al.. (1983). Mutagenic activity of Kathon, an industrial biocide and cosmetics preservative containing 5-chloro-2-methyl-4-isothiazolin-3-one and 2-methyl-4-isothiazolin-3-one. Mutation Research Letters. 119(1). 35–43. 10 indexed citations

17.

Venitt, Stanley, et al.. (1983). The development of anaerobic methods for bacterial mutation assays: aerobic and anaerobic fluctuation tests of human faecal extracts and reference mutagens. Carcinogenesis. 4(3). 339–345. 19 indexed citations

18.

Venitt, Stanley & E.M. Tarmy. (1972). The selective excision of arylalkylated products from the DNA of Escherichia coli treated with the carcinogen 7-bromomethylbenz()anthracene. Biochimica et Biophysica Acta (BBA) - Nucleic Acids and Protein Synthesis. 287(1). 38–51. 37 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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