Stacy Carl-McGrath

1.1k total citations
17 papers, 926 citations indexed

About

Stacy Carl-McGrath is a scholar working on Oncology, Molecular Biology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Stacy Carl-McGrath has authored 17 papers receiving a total of 926 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Oncology, 4 papers in Molecular Biology and 4 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Stacy Carl-McGrath's work include Peptidase Inhibition and Analysis (6 papers), Hormonal Regulation and Hypertension (4 papers) and Renin-Angiotensin System Studies (3 papers). Stacy Carl-McGrath is often cited by papers focused on Peptidase Inhibition and Analysis (6 papers), Hormonal Regulation and Hypertension (4 papers) and Renin-Angiotensin System Studies (3 papers). Stacy Carl-McGrath collaborates with scholars based in Germany, China and Uganda. Stacy Carl-McGrath's co-authors include Christoph Röcken, Uwe Lendeckel, Matthias Ebert, Albert Roessner, Matthias Ebert, Marco Arndt, J. Kohl, Matthias P. Ebert, Regine Schneider‐Stock and M. Ebert and has published in prestigious journals such as Clinical Cancer Research, The American Journal of Surgical Pathology and The Journal of Pathology.

In The Last Decade

Stacy Carl-McGrath

17 papers receiving 916 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Stacy Carl-McGrath Germany 15 417 376 172 161 128 17 926
Yuenian Eric Shi United States 14 431 1.0× 335 0.9× 84 0.5× 480 3.0× 34 0.3× 23 1.0k
Marco Arndt Germany 16 600 1.4× 607 1.6× 765 4.4× 232 1.4× 75 0.6× 37 1.7k
E. Spitzer Germany 15 499 1.2× 412 1.1× 55 0.3× 109 0.7× 52 0.4× 26 910
Liang Hu China 19 669 1.6× 309 0.8× 76 0.4× 321 2.0× 19 0.1× 31 1.2k
Hiroaki Shimoyamada Japan 20 426 1.0× 218 0.6× 133 0.8× 211 1.3× 172 1.3× 46 1.1k
Ramprasad Srinivasan United States 16 353 0.8× 213 0.6× 128 0.7× 127 0.8× 21 0.2× 34 1.2k
Hui‐Jia Dong United States 12 468 1.1× 199 0.5× 45 0.3× 177 1.1× 29 0.2× 16 1.1k
Zhenling Yao United States 14 478 1.1× 188 0.5× 42 0.2× 195 1.2× 76 0.6× 27 1.2k
L. M. Ellis United States 12 585 1.4× 371 1.0× 50 0.3× 244 1.5× 26 0.2× 20 1.1k
G F Barnard United States 18 673 1.6× 540 1.4× 40 0.2× 344 2.1× 79 0.6× 26 1.5k

Countries citing papers authored by Stacy Carl-McGrath

Since Specialization
Citations

This map shows the geographic impact of Stacy Carl-McGrath's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stacy Carl-McGrath with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stacy Carl-McGrath more than expected).

Fields of papers citing papers by Stacy Carl-McGrath

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stacy Carl-McGrath. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stacy Carl-McGrath. The network helps show where Stacy Carl-McGrath may publish in the future.

Co-authorship network of co-authors of Stacy Carl-McGrath

This figure shows the co-authorship network connecting the top 25 collaborators of Stacy Carl-McGrath. A scholar is included among the top collaborators of Stacy Carl-McGrath based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stacy Carl-McGrath. Stacy Carl-McGrath is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Carl-McGrath, Stacy, Friedrich‐Wilhelm Röhl, Uwe Lendeckel, et al.. (2009). Differential Expression of SPARC in Intestinal-type Gastric Cancer Correlates with Tumor Progression and Nodal Spread. Translational Oncology. 2(4). 310–320. 25 indexed citations
3.
Carl-McGrath, Stacy, et al.. (2008). Proteomics of Pancreatic Cancer. Pancreas. 36(4). 329–336. 36 indexed citations
4.
Carl-McGrath, Stacy, Matthias Ebert, Uwe Lendeckel, & Christoph Röcken. (2007). Expression of the local angiotensin II system in gastric cancer may facilitate lymphatic invasion and nodal spread. Cancer Biology & Therapy. 6(8). 1229–1237. 41 indexed citations
5.
Carl-McGrath, Stacy, Regine Schneider‐Stock, Matthias Ebert, & Christoph Röcken. (2007). Differential expression and localisation of gasdermin-like (GSDML), a novel member of the cancer-associated GSDMDC protein family, in neoplastic and non-neoplastic gastric, hepatic, and colon tissues. Pathology. 40(1). 13–24. 69 indexed citations
6.
Röcken, Christoph, Konrad Neumann, Stacy Carl-McGrath, et al.. (2007). The Gene Polymorphism of the Angiotensin I-Converting Enzyme Correlates with Tumor Size and Patient Survival in Colorectal Cancer Patients. Neoplasia. 9(9). 716–722. 44 indexed citations
7.
Röcken, Christoph, Friedrich‐Wilhelm Röhl, Uwe Lendeckel, et al.. (2007). The Angiotensin II/Angiotensin II Receptor System Correlates with Nodal Spread in Intestinal Type Gastric Cancer. Cancer Epidemiology Biomarkers & Prevention. 16(6). 1206–1212. 60 indexed citations
8.
Carl-McGrath, Stacy, Uwe Lendeckel, M. Ebert, & Christoph Röcken. (2006). Ectopeptidases in tumour biology: a review.. PubMed. 21(12). 1339–53. 64 indexed citations
9.
Röcken, Christoph, Jonathan D. Licht, Albert Roessner, & Stacy Carl-McGrath. (2005). Canalicular immunostaining of aminopeptidase N (CD13) as a diagnostic marker for hepatocellular carcinoma. Journal of Clinical Pathology. 58(10). 1069–1075. 37 indexed citations
10.
Ebert, Matthias PA, Claus Schäfer, Jie Chen, et al.. (2005). Protective role of heat shock protein 27 in gastric mucosal injury. The Journal of Pathology. 207(2). 177–184. 26 indexed citations
11.
Röcken, Christoph, Uwe Lendeckel, Jutta Dierkes, et al.. (2005). The Number of Lymph Node Metastases in Gastric Cancer Correlates with the Angiotensin I–Converting Enzyme Gene Insertion/Deletion Polymorphism. Clinical Cancer Research. 11(7). 2526–2530. 87 indexed citations
12.
Carl-McGrath, Stacy, Uwe Lendeckel, Matthias Ebert, Albert Roessner, & Christoph Röcken. (2005). The disintegrin-metalloproteinases ADAM9, ADAM12, and ADAM15 are upregulated in gastric cancer. International Journal of Oncology. 26(1). 17–24. 138 indexed citations
13.
Lendeckel, Uwe, et al.. (2004). Angiotensin I-converting Enzyme (CD143) Is Down-regulated in Focal Nodular Hyperplasia of the Liver. The American Journal of Surgical Pathology. 28(1). 84–88. 1 indexed citations
14.
Lendeckel, Uwe, et al.. (2004). Increased expression of ADAM family members in human breast cancer and breast cancer cell lines. Journal of Cancer Research and Clinical Oncology. 131(1). 41–48. 132 indexed citations
15.
Carl-McGrath, Stacy, et al.. (2004). The ectopeptidases CD10, CD13, CD26, and CD143 are upregulated in gastric cancer. International Journal of Oncology. 25(5). 1223–32. 54 indexed citations
16.
Röcken, Christoph, et al.. (2004). Ectopeptidases are differentially expressed in hepatocellular carcinomas. International Journal of Oncology. 24(3). 487–95. 14 indexed citations
17.
Röcken, Christoph & Stacy Carl-McGrath. (2001). Pathology and Pathogenesis of Hepatocellular Carcinoma. Digestive Diseases. 19(4). 269–278. 65 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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