Sofia Mosesova

3.6k total citations · 3 hit papers
16 papers, 2.6k citations indexed

About

Sofia Mosesova is a scholar working on Physiology, Immunology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Sofia Mosesova has authored 16 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Physiology, 6 papers in Immunology and 5 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Sofia Mosesova's work include Asthma and respiratory diseases (10 papers), Respiratory and Cough-Related Research (4 papers) and IL-33, ST2, and ILC Pathways (4 papers). Sofia Mosesova is often cited by papers focused on Asthma and respiratory diseases (10 papers), Respiratory and Cough-Related Research (4 papers) and IL-33, ST2, and ILC Pathways (4 papers). Sofia Mosesova collaborates with scholars based in United States, Canada and United Kingdom. Sofia Mosesova's co-authors include Joseph R. Arron, Jeffrey M. Harris, Nicola A. Hanania, Lawren C. Wu, David F. Choy, Heleen Scheerens, John G. Matthews, Sean P. Bohen, Merdad V. Parsey and Phillip E. Korenblat and has published in prestigious journals such as New England Journal of Medicine, American Journal of Respiratory and Critical Care Medicine and Journal of Allergy and Clinical Immunology.

In The Last Decade

Sofia Mosesova

16 papers receiving 2.6k citations

Hit Papers

Lebrikizumab Treatment in Adults with Asthma 2011 2026 2016 2021 2011 2013 2012 250 500 750 1000

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sofia Mosesova United States 10 2.2k 1.5k 929 589 416 16 2.6k
Guiquan Jia United States 11 1.7k 0.8× 1.4k 0.9× 754 0.8× 311 0.5× 321 0.8× 16 2.3k
Merdad V. Parsey United States 11 1.2k 0.5× 841 0.6× 724 0.8× 287 0.5× 237 0.6× 16 1.8k
Aarti Shikotra United Kingdom 14 807 0.4× 455 0.3× 426 0.5× 204 0.3× 155 0.4× 19 1.1k
Paul Stryszak United States 15 598 0.3× 460 0.3× 343 0.4× 275 0.5× 143 0.3× 20 1.3k
Brian Leaker United Kingdom 23 428 0.2× 556 0.4× 345 0.4× 145 0.2× 129 0.3× 66 1.5k
Abhya Gupta Germany 13 459 0.2× 920 0.6× 171 0.2× 94 0.2× 85 0.2× 39 1.4k
Stanisława Bazan‐Socha Poland 18 334 0.2× 343 0.2× 216 0.2× 140 0.2× 92 0.2× 107 990
Zsuzsanna Szalai Hungary 19 454 0.2× 595 0.4× 258 0.3× 945 1.6× 194 0.5× 51 2.5k
Ieuan Jones United Kingdom 13 308 0.1× 171 0.1× 223 0.2× 206 0.3× 244 0.6× 29 1.0k
Sylvie Abi-Younes United States 9 348 0.2× 137 0.1× 386 0.4× 184 0.3× 88 0.2× 9 969

Countries citing papers authored by Sofia Mosesova

Since Specialization
Citations

This map shows the geographic impact of Sofia Mosesova's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sofia Mosesova with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sofia Mosesova more than expected).

Fields of papers citing papers by Sofia Mosesova

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sofia Mosesova. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sofia Mosesova. The network helps show where Sofia Mosesova may publish in the future.

Co-authorship network of co-authors of Sofia Mosesova

This figure shows the co-authorship network connecting the top 25 collaborators of Sofia Mosesova. A scholar is included among the top collaborators of Sofia Mosesova based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sofia Mosesova. Sofia Mosesova is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Grievink, Hendrika W., Jules A. A. C. Heuberger, Fen Huang, et al.. (2019). DNL104, a Centrally Penetrant RIPK1 Inhibitor, Inhibits RIP1 Kinase Phosphorylation in a Randomized Phase I Ascending Dose Study in Healthy Volunteers. Clinical Pharmacology & Therapeutics. 107(2). 406–414. 56 indexed citations
2.
Cabanski, Christopher R., Houston Gilbert, & Sofia Mosesova. (2018). Can Graphics Tell Lies? A Tutorial on How To Visualize Your Data. Clinical and Translational Science. 11(4). 371–377. 2 indexed citations
3.
4.
Fischer, Saloumeh K, et al.. (2016). Characterization of Il-17Aa and Il-17Ff in Rheumatoid Arthritis and Multiple Sclerosis. Bioanalysis. 8(22). 2317–2327. 27 indexed citations
5.
Noonan, Michael, Phillip E. Korenblat, Sofia Mosesova, et al.. (2013). Dose-ranging study of lebrikizumab in asthmatic patients not receiving inhaled steroids. Journal of Allergy and Clinical Immunology. 132(3). 567–574.e12. 140 indexed citations
6.
Hanania, Nicola A., Sally E. Wenzel, Karin Rosén, et al.. (2013). Exploring the Effects of Omalizumab in Allergic Asthma. American Journal of Respiratory and Critical Care Medicine. 187(8). 804–811. 669 indexed citations breakdown →
7.
Gauvreau, Gail M., L.‐P. Boulet, Donald W. Cockcroft, et al.. (2013). OX40L blockade and allergen‐induced airway responses in subjects with mild asthma. Clinical & Experimental Allergy. 44(1). 29–37. 81 indexed citations
8.
Jia, Guiquan, Richard W. Erickson, David F. Choy, et al.. (2012). Periostin is a systemic biomarker of eosinophilic airway inflammation in asthmatic patients. Journal of Allergy and Clinical Immunology. 130(3). 647–654.e10. 455 indexed citations breakdown →
9.
Scheerens, Heleen, Joseph R. Arron, David F. Choy, et al.. (2012). Lebrikizumab Treatment Reduces Serum Periostin Levels In Asthma Patients With Elevated Baseline Levels Of Periostin. A3960–A3960. 9 indexed citations
10.
Scheerens, Heleen, Joseph R. Arron, David F. Choy, et al.. (2012). Lebrikizumab reduces serum periostin in asthma patients with elevated baseline periostin. 40. 2167. 3 indexed citations
11.
McClintock, Dana, Jonathan Corren, Nicola A. Hanania, et al.. (2012). Lebrikizumab, An Anti-IL-13 Monoclonal Antibody, Reduces Severe Asthma Exacerbations Over 32 Weeks In Adults With Inadequately Controlled Asthma. A3959–A3959. 8 indexed citations
12.
Scheerens, Heleen, Wendy S. Putnam, Yanan Zheng, et al.. (2012). Treatment With MEMP1972A, An Anti-M1 Prime Monoclonal Antibody, Reduced Serum IgE In Healthy Volunteers And Patients With Allergic Rhinitis. A6791–A6791. 9 indexed citations
13.
Hanania, Nicola A., Robert F. Lemanske, Phillip E. Korenblat, et al.. (2011). Efficacy of an anti-IL13 monoclonal antibody, lebrikizumab, in adults with inadequately controlled asthma is enhanced in those with high periostin levels. European Respiratory Journal. 38(Suppl 55). 3426–3426. 2 indexed citations
14.
Corren, Jonathan, Robert F. Lemanske, Nicola A. Hanania, et al.. (2011). Lebrikizumab Treatment in Adults with Asthma. New England Journal of Medicine. 365(12). 1088–1098. 1097 indexed citations breakdown →
15.
Arron, Joseph R., Richard W. Erickson, David F. Choy, et al.. (2011). Periostin Is A Systemic Biomarker Of Eosinophilic Airway Inflammation In Asthma. A4455–A4455. 5 indexed citations
16.
Steiner, Stefan H., et al.. (2007). A Bubble Mixture Experiment Project for Use in an Advanced Design of Experiments Class. Journal of Statistics Education. 15(1). 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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