S.J. Hong

740 total citations
37 papers, 646 citations indexed

About

S.J. Hong is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, S.J. Hong has authored 37 papers receiving a total of 646 indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Molecular Biology, 19 papers in Cellular and Molecular Neuroscience and 8 papers in Physiology. Recurrent topics in S.J. Hong's work include Ion channel regulation and function (31 papers), Neuroscience and Neural Engineering (10 papers) and Neuroscience and Neuropharmacology Research (9 papers). S.J. Hong is often cited by papers focused on Ion channel regulation and function (31 papers), Neuroscience and Neural Engineering (10 papers) and Neuroscience and Neuropharmacology Research (9 papers). S.J. Hong collaborates with scholars based in Taiwan and Japan. S.J. Hong's co-authors include C.C. Chang, C. C. Chang, Ming‐Jai Su, Lih‐Chu Chiou, Wan‐Wan Lin, Kunihiro Tsuji, Chih‐Yu Huang and Sang Hee Lee and has published in prestigious journals such as The Journal of Physiology, Journal of Neurophysiology and Brain Research.

In The Last Decade

S.J. Hong

37 papers receiving 638 citations

Peers

S.J. Hong

CMN

GENET

PHYSI

Masaakira Kano Japan

Frances M. Sansone United States

Andrew Palma United States

Roland Bournaud France

Maria R. Castro Costa United States

S M Schuetze United States

John V. Milligan Canada

Nancy Osorio France

Randal Numann United States

Rocio K. Finol‐Urdaneta Australia

Masaakira Kano Japan

S.J. Hong

328 ×0.6

254 ×0.9

CMN

33 ×0.4

GENET

49 ×0.7

PHYSI

27 ×0.4

Citations per year, relative to S.J. Hong S.J. Hong (= 1×) peers Masaakira Kano

Countries citing papers authored by S.J. Hong

Since Specialization

Citations

This map shows the geographic impact of S.J. Hong's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S.J. Hong with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S.J. Hong more than expected).

Fields of papers citing papers by S.J. Hong

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by S.J. Hong. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S.J. Hong. The network helps show where S.J. Hong may publish in the future.

Co-authorship network of co-authors of S.J. Hong

This figure shows the co-authorship network connecting the top 25 collaborators of S.J. Hong. A scholar is included among the top collaborators of S.J. Hong based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with S.J. Hong. S.J. Hong is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Hong, S.J., et al.. (2003). Dependence of cyclopiazonic-acid-induced muscle contractures on extracellular Ca²⁺. Canadian Journal of Physiology and Pharmacology. 81(12). 1101–1109. 3 indexed citations

Hong, S.J.. (2000). Inhibition of mouse neuromuscular transmission and contractile function by okadaic acid and cantharidin. British Journal of Pharmacology. 130(6). 1211–1218. 8 indexed citations

Hong, S.J. & C.C. Chang. (1999). Inhibition of quantal release from motor nerve by wortmannin. British Journal of Pharmacology. 128(1). 142–148. 14 indexed citations

Hong, S.J. & C.C. Chang. (1998). Novel inhibition of contractility by wortmannin in skeletal muscle. British Journal of Pharmacology. 124(5). 849–856. 8 indexed citations

Hong, S.J., et al.. (1996). Inhibition of neuromuscular transmission in the myenteric plexus of guinea‐pig ileum by ω‐conotoxins GVIA, MVIIA, MVIIC and SVIB. British Journal of Pharmacology. 118(3). 797–803. 18 indexed citations

Hong, S.J. & C.C. Chang. (1995). Calcium channel subtypes for the sympathetic and parasympathetic nerves of guinea‐pig atria. British Journal of Pharmacology. 116(1). 1577–1582. 37 indexed citations

Hong, S.J., Sang Hee Lee, & C.C. Chang. (1995). Physiological and regenerative acetylcholine release form motor nerve: Differential inhibitions by vesamicol and ω-agatoxin IVA. Neuroscience. 67(1). 169–175. 5 indexed citations

Hong, S.J., et al.. (1994). Facilitation of nicotinic receptor desensitization at mouse motor endplate by a receptor-operated Ca2+ channel blocker, SK&F 96365. European Journal of Pharmacology. 265(1-2). 35–42. 8 indexed citations

Hong, S.J., Wan‐Wan Lin, & C. C. Chang. (1994). Inhibition of the sodium channel by SK&F 96365, an inhibitor of the receptor-operated calcium channel, in mouse diaphragm. Journal of Biomedical Science. 1(3). 172–178. 15 indexed citations

10.

Hong, S.J. & C.C. Chang. (1993). Transmitter‐mediated local contracture of the endplate region of the focally innervated mouse diaphragm treated with anticholinesterase. British Journal of Pharmacology. 109(4). 1178–1185. 9 indexed citations

11.

Hong, S.J. & C.C. Chang. (1991). Run‐down of neuromuscular transmission during repetitive nerve activity by nicotinic antagonists is not due to desensitization of the postsynaptic receptor. British Journal of Pharmacology. 102(4). 817–822. 29 indexed citations

12.

Hong, S.J., et al.. (1990). Nicotinic actions of oxotremorine on murine skeletal muscle. Evidence against muscarinic modulation of acetylcholine release. Brain Research. 534(1-2). 142–148. 7 indexed citations

13.

Hong, S.J. & C.C. Chang. (1990). Facilitation by 3,4‐diaminopyridine of regenerative acetylcholine release from mouse motor nerve. British Journal of Pharmacology. 101(4). 793–798. 20 indexed citations

14.

Chang, C.C., Chih‐Yu Huang, & S.J. Hong. (1990). Organic calcium channel antagonists provoke acetylcholine receptor autodesensitization on train stimulation of motor nerve. Neuroscience. 38(3). 731–742. 10 indexed citations

15.

Chang, C.C., et al.. (1989). Nicardipine inhibits axon conduction but causes dual changes of acetylcholine release in the mouse motor nerve. Canadian Journal of Physiology and Pharmacology. 67(12). 1493–1498. 3 indexed citations

16.

Hong, S.J., et al.. (1989). Antagonism by tubocurarine and verapamil of the regenerative acetylcholine release from mouse motor nerve. European Journal of Pharmacology. 162(1). 11–17. 18 indexed citations

17.

Hong, S.J., et al.. (1988). Neuromuscular block by verapamil and diltiazem and inhibition of acetylcholine release. Brain Research. 454(1-2). 332–339. 23 indexed citations

18.

Chang, C.C., et al.. (1988). Reversals of the neostigmine‐induced tetanic fade and endplate potential run‐down with respect to the autoregulation of transmitter release. British Journal of Pharmacology. 95(4). 1255–1261. 19 indexed citations

19.

Chiou, Lih‐Chu, et al.. (1987). Does endogenous adenosine modulate the release of acetylcholine from motor nerve during single and repetitive stimulations in the mouse diaphragm?. The Japanese Journal of Pharmacology. 44(4). 373–380. 4 indexed citations

20.

Chang, C.C. & S.J. Hong. (1987). Dissociation of the end-plate potential run-down and the tetanic fade from the postsynaptic inhibition of acetylcholine receptor by α-neurotoxins. Experimental Neurology. 98(3). 509–517. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact