Shinji Toki

1.7k total citations
44 papers, 1.2k citations indexed

About

Shinji Toki is a scholar working on Immunology, Physiology and Surgery. According to data from OpenAlex, Shinji Toki has authored 44 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Immunology, 20 papers in Physiology and 11 papers in Surgery. Recurrent topics in Shinji Toki's work include IL-33, ST2, and ILC Pathways (18 papers), Asthma and respiratory diseases (18 papers) and Eosinophilic Esophagitis (11 papers). Shinji Toki is often cited by papers focused on IL-33, ST2, and ILC Pathways (18 papers), Asthma and respiratory diseases (18 papers) and Eosinophilic Esophagitis (11 papers). Shinji Toki collaborates with scholars based in United States, Japan and Canada. Shinji Toki's co-authors include R. Stokes Peebles, Dawn C. Newcomb, Kasia Goleniewska, Weisong Zhou, Kelli L. Boyd, Melissa H. Bloodworth, Jian Zhang, Matthew T. Stier, Baohua Zhou and Sara Reiss and has published in prestigious journals such as Journal of Clinical Investigation, The Journal of Immunology and PLoS ONE.

In The Last Decade

Shinji Toki

42 papers receiving 1.2k citations

Peers

Shinji Toki
Matthew T. Stier United States
Yoshiki Shiraishi Japan
Josiane S. Neves Brazil
Gavin Lewis United States
Joachim Maxeiner Germany
Nina Dehzad Germany
Marie Carlson Sweden
J M Cavaillon France
Jennifer J. McIntire United States
Yue Jia China
Matthew T. Stier United States
Shinji Toki
Citations per year, relative to Shinji Toki Shinji Toki (= 1×) peers Matthew T. Stier

Countries citing papers authored by Shinji Toki

Since Specialization
Citations

This map shows the geographic impact of Shinji Toki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shinji Toki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shinji Toki more than expected).

Fields of papers citing papers by Shinji Toki

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shinji Toki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shinji Toki. The network helps show where Shinji Toki may publish in the future.

Co-authorship network of co-authors of Shinji Toki

This figure shows the co-authorship network connecting the top 25 collaborators of Shinji Toki. A scholar is included among the top collaborators of Shinji Toki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shinji Toki. Shinji Toki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Thomas, Christopher M., Jian Zhang, Shinji Toki, et al.. (2025). CFTR negatively reprograms Th2 cell responses, and CFTR potentiation restrains allergic airway inflammation. JCI Insight. 10(9). 2 indexed citations
2.
Caslin, Heather L., W. Reid Bolus, Christopher M. Thomas, et al.. (2023). Bovine Serum Albumin Elicits IL-33–Dependent Adipose Tissue Eosinophilia: Potential Relevance to Ovalbumin-induced Models of Allergic Disease. ImmunoHorizons. 7(12). 842–852.
3.
Shiroshita, Akihiro, et al.. (2023). Expanding the Scope: In-depth Review of Interaction in Regression Models. PubMed. 6(2). 25–32. 1 indexed citations
4.
5.
Bastarache, Julie A., et al.. (2023). Late Breaking Abstract - Liraglutide attenuates sepsis-induced endothelial dysfunction and Acute Lung Injury. PA5233–PA5233. 1 indexed citations
6.
Norlander, Allison E., Melissa H. Bloodworth, Shinji Toki, et al.. (2021). Prostaglandin I2 signaling licenses Treg suppressive function and prevents pathogenic reprogramming. Journal of Clinical Investigation. 131(7). 14 indexed citations
7.
Zhou, Weisong, Jian Zhang, Shinji Toki, et al.. (2020). COX Inhibition Increases Alternaria-Induced Pulmonary Group 2 Innate Lymphoid Cell Responses and IL-33 Release in Mice. The Journal of Immunology. 205(4). 1157–1166. 19 indexed citations
8.
Toki, Shinji, Kasia Goleniewska, Jian Zhang, et al.. (2020). TSLP and IL‐33 reciprocally promote each other's lung protein expression and ILC2 receptor expression to enhance innate type‐2 airway inflammation. Allergy. 75(7). 1606–1617. 120 indexed citations
9.
Palmer, Lauren D., K. Nichole Maloney, Kelli L. Boyd, et al.. (2019). The Innate Immune Protein S100A9 Protects from T-Helper Cell Type 2–mediated Allergic Airway Inflammation. American Journal of Respiratory Cell and Molecular Biology. 61(4). 459–468. 25 indexed citations
10.
Toki, Shinji, et al.. (2018). TSLP and IL-33 Reciprocally Regulate Each Other’s Lung Protein Expression and Receptor Expression on ILC2 following Aeroallergen Challenge in Mice. Journal of Allergy and Clinical Immunology. 141(2). AB2–AB2. 3 indexed citations
11.
Toki, Shinji, Kasia Goleniewska, Sara Reiss, et al.. (2018). Glucagon-like peptide 1 signaling inhibits allergen-induced lung IL-33 release and reduces group 2 innate lymphoid cell cytokine production in vivo. Journal of Allergy and Clinical Immunology. 142(5). 1515–1528.e8. 70 indexed citations
12.
Stier, Matthew T., Hubaida Fuseini, Jeffrey A. Yung, et al.. (2017). Testosterone Attenuates Group 2 Innate Lymphoid Cell-Mediated Airway Inflammation. Cell Reports. 21(9). 2487–2499. 197 indexed citations
13.
Toki, Shinji, Kasia Goleniewska, Sara Reiss, et al.. (2016). The histone deacetylase inhibitor trichostatin A suppresses murine innate allergic inflammation by blocking group 2 innate lymphoid cell (ILC2) activation. Thorax. 71(7). 633–645. 46 indexed citations
14.
Zhou, Weisong, Shinji Toki, Jian Zhang, et al.. (2015). Prostaglandin I2 Signaling and Inhibition of Group 2 Innate Lymphoid Cell Responses. American Journal of Respiratory and Critical Care Medicine. 193(1). 31–42. 109 indexed citations
15.
Toki, Shinji, Reed A. Omary, Kevin J. Wilson, et al.. (2013). A comprehensive analysis of transfection-assisted delivery of iron oxide nanoparticles to dendritic cells. Nanomedicine Nanotechnology Biology and Medicine. 9(8). 1235–1244. 15 indexed citations
16.
Sevin, Carla M., Dawn C. Newcomb, Shinji Toki, et al.. (2013). Deficiency of gp91 phox Inhibits Allergic Airway Inflammation. American Journal of Respiratory Cell and Molecular Biology. 49(3). 396–402. 20 indexed citations
17.
Zhou, Weisong, M.M. Huckabee, Dawn C. Newcomb, et al.. (2012). Prostaglandin I2 Signaling Drives Th17 Differentiation and Exacerbates Experimental Autoimmune Encephalomyelitis. PLoS ONE. 7(5). e33518–e33518. 29 indexed citations
18.
Toki, Shinji, et al.. (2011). Prostaglandin I2 Receptor (IP) Signaling Inhibits TLR4 Cell Surface Expression and Reduces LPS-induced Lung Inflammation. Journal of Allergy and Clinical Immunology. 127(2). AB92–AB92. 1 indexed citations
19.
Hasegawa, Takanori, et al.. (2009). Efficacy ofLactobacillus plantarumStrain HSK201 in Relief from Japanese Cedar Pollinosis. Bioscience Biotechnology and Biochemistry. 73(12). 2626–2631. 14 indexed citations
20.
Toki, Shinji, Shinji Kagaya, Miwa Shinohara, et al.. (2008). <i>Lactobacillus rhamnosus</i> GG and <i>Lactobacillus casei</i> Suppress <i>Escherichia coli</i>-Induced Chemokine Expression in Intestinal Epithelial Cells. International Archives of Allergy and Immunology. 148(1). 45–58. 32 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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