Matthew T. Stier

1.5k total citations
25 papers, 1.1k citations indexed

About

Matthew T. Stier is a scholar working on Immunology, Surgery and Physiology. According to data from OpenAlex, Matthew T. Stier has authored 25 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Immunology, 12 papers in Surgery and 6 papers in Physiology. Recurrent topics in Matthew T. Stier's work include IL-33, ST2, and ILC Pathways (14 papers), Eosinophilic Esophagitis (10 papers) and Immune Cell Function and Interaction (7 papers). Matthew T. Stier is often cited by papers focused on IL-33, ST2, and ILC Pathways (14 papers), Eosinophilic Esophagitis (10 papers) and Immune Cell Function and Interaction (7 papers). Matthew T. Stier collaborates with scholars based in United States, Canada and South Korea. Matthew T. Stier's co-authors include R. Stokes Peebles, Dawn C. Newcomb, Kasia Goleniewska, Melissa H. Bloodworth, Shinji Toki, Kelli L. Boyd, Weisong Zhou, Jacqueline Cephus, Jian Zhang and Baohua Zhou and has published in prestigious journals such as The Journal of Experimental Medicine, Nature Immunology and The Journal of Immunology.

In The Last Decade

Matthew T. Stier

24 papers receiving 1.1k citations

Peers

Matthew T. Stier

IMMUN

SURGE

PHYSI

PRM

EPIDE

Hadi Maazi United States

Benjamin P. Hurrell United States

Gavin Lewis United States

Laura Denney United Kingdom

Anupriya Khare United States

Mahesh Raundhal United States

Zhixuan Loh Australia

Fernando Botelho Canada

Dana Colbert United States

R.P. Schleimer United States

Hadi Maazi United States

Matthew T. Stier

802 ×1.1

IMMUN

447 ×0.9

SURGE

388 ×1.1

PHYSI

97 ×0.6

PRM

179 ×1.3

EPIDE

Citations per year, relative to Matthew T. Stier Matthew T. Stier (= 1×) peers Hadi Maazi

Countries citing papers authored by Matthew T. Stier

Since Specialization

Citations

This map shows the geographic impact of Matthew T. Stier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew T. Stier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew T. Stier more than expected).

Fields of papers citing papers by Matthew T. Stier

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew T. Stier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew T. Stier. The network helps show where Matthew T. Stier may publish in the future.

Co-authorship network of co-authors of Matthew T. Stier

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew T. Stier. A scholar is included among the top collaborators of Matthew T. Stier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew T. Stier. Matthew T. Stier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Stier, Matthew T., Changbeom Sim, C. Mark Nichols, et al.. (2026). Metabolic adaptations rewire CD4+ T cells in a subset-specific manner in human critical illness with and without sepsis. Nature Immunology. 27(2). 236–249.

Wu, Wei, Matthew T. Stier, John W. Stokes, et al.. (2023). Immune characterization of a xenogeneic human lung cross-circulation support system. Science Advances. 9(13). eade7647–eade7647. 3 indexed citations

Stier, Matthew T. & R. Stokes Peebles. (2020). Protocols for Studying Murine ILC Development. Methods in molecular biology. 2121. 7–22. 1 indexed citations

Meyer, Anne R., Amy C. Engevik, Matthew T. Stier, et al.. (2020). Group 2 Innate Lymphoid Cells Coordinate Damage Response in the Stomach. Gastroenterology. 159(6). 2077–2091.e8. 64 indexed citations

Stier, Matthew T., et al.. (2020). Differential Type 2 cytokine responses and group 2 innate lymphoid cell (ILC2) activation among 44 strains of mice in the Collaborative Cross following Alternaria extract challenge. Journal of Allergy and Clinical Immunology. 145(2). AB2–AB2. 1 indexed citations

Stier, Matthew T. & R. Stokes Peebles. (2018). Host and Viral Determinants of Respiratory Syncytial Virus-induced Airway Mucus. Annals of the American Thoracic Society. 15(Supplement_3). S205–S209. 10 indexed citations

Toki, Shinji, Kasia Goleniewska, Sara Reiss, et al.. (2018). Glucagon-like peptide 1 signaling inhibits allergen-induced lung IL-33 release and reduces group 2 innate lymphoid cell cytokine production in vivo. Journal of Allergy and Clinical Immunology. 142(5). 1515–1528.e8. 70 indexed citations

Stier, Matthew T., Hubaida Fuseini, Jeffrey A. Yung, et al.. (2017). Testosterone Attenuates Group 2 Innate Lymphoid Cell-Mediated Airway Inflammation. Cell Reports. 21(9). 2487–2499. 197 indexed citations

Stier, Matthew T., Kasia Goleniewska, Jacqueline Cephus, et al.. (2017). STAT1 Represses Cytokine-Producing Group 2 and Group 3 Innate Lymphoid Cells during Viral Infection. The Journal of Immunology. 199(2). 510–519. 54 indexed citations

10.

Juttukonda, Lillian J., Evelien T.M. Berends, Joseph P. Zackular, et al.. (2017). Dietary Manganese Promotes Staphylococcal Infection of the Heart. Cell Host & Microbe. 22(4). 531–542.e8. 54 indexed citations

11.

Stier, Matthew T. & R. Stokes Peebles. (2017). Innate lymphoid cells and allergic disease. Annals of Allergy Asthma & Immunology. 119(6). 480–488. 24 indexed citations

12.

Ashley, Shanna L., Carla D. Pretto, Matthew T. Stier, et al.. (2017). Matrix Metalloproteinase Activity in Infections by an Encephalitic Virus, Mouse Adenovirus Type 1. Journal of Virology. 91(6). 22 indexed citations

13.

Stier, Matthew T., et al.. (2017). IL-33 Promotes Egress of Group 2 Innate Lymphoids Cells (ILC2) from the Bone Marrow. Journal of Allergy and Clinical Immunology. 139(2). AB80–AB80. 1 indexed citations

14.

Toki, Shinji, Kasia Goleniewska, Sara Reiss, et al.. (2016). The histone deacetylase inhibitor trichostatin A suppresses murine innate allergic inflammation by blocking group 2 innate lymphoid cell (ILC2) activation. Thorax. 71(7). 633–645. 46 indexed citations

15.

Stier, Matthew T., Melissa H. Bloodworth, Shinji Toki, et al.. (2016). Respiratory syncytial virus infection activates IL-13–producing group 2 innate lymphoid cells through thymic stromal lymphopoietin. Journal of Allergy and Clinical Immunology. 138(3). 814–824.e11. 164 indexed citations

16.

Zhou, Weisong, Shinji Toki, Jian Zhang, et al.. (2015). Prostaglandin I2 Signaling and Inhibition of Group 2 Innate Lymphoid Cell Responses. American Journal of Respiratory and Critical Care Medicine. 193(1). 31–42. 109 indexed citations

17.

Stier, Matthew T. & Katherine R. Spindler. (2011). Polymorphisms in Ly6 genes in Msq1 encoding susceptibility to mouse adenovirus type 1. Mammalian Genome. 23(3-4). 250–258. 14 indexed citations

18.

Sivaprasad, Umasundari, David J. Askew, Mark B. Ericksen, et al.. (2010). A non-redundant role for Serpinb3a in the induction of mucus production in asthma (141.17). The Journal of Immunology. 184(Supplement_1). 141.17–141.17. 1 indexed citations

19.

Sivaprasad, Umasundari, David J. Askew, Mark B. Ericksen, et al.. (2010). A nonredundant role for mouse Serpinb3a in the induction of mucus production in asthma. Journal of Allergy and Clinical Immunology. 127(1). 254–261.e6. 33 indexed citations

20.

Chen, Weiguo, Umasundari Sivaprasad, Yasuhiro Tabata, et al.. (2009). IL-13Rα2 Membrane and Soluble Isoforms Differ in Humans and Mice. The Journal of Immunology. 183(12). 7870–7876. 52 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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