Shi Eun Kang
Impact in
- Cancer Research top 5%
- MicroRNA in disease regulation
- Cancer-related molecular mechanisms research
- Oncology top 10%
- Cancer Cells and Metastasis
- Cancer-related Molecular Pathways
Papers in
-
- Wnt/β-catenin signaling in development and cancer 3
- RNA Research and Splicing 1
- Oncology 3
- Cancer Cells and Metastasis 2
- Cytokine Signaling Pathways and Interactions 1
- Co-authors
- Joo Kyung Ryu (4 shared papers)Nam Hee Kim (4 shared papers)So Young (4 shared papers)Inhan Lee (4 shared papers)Jong In Yook (4 shared papers)Hyun Sil Kim (4 shared papers)Stephen J. Weiss (3 shared papers)Xiaoyan Li (2 shared papers)
- Journals
- The Journal of Experimental Medicine (1 paper)The Journal of Cell Biology (1 paper)Cell Cycle (1 paper)International Journal of Oncology (1 paper)Science Signaling (1 paper)
- Partner nations
- South KoreaUnited StatesPoland
In The Last Decade
Shi Eun Kang
5 papers receiving 747 citations
Peers
Comparison fields: 5 of 70
- Cancer Research 434
- Oncology 246
- Molecular Biology 587
- Cell Biology 45
- Pathology and Forensic Medicine 34
Countries citing papers authored by Shi Eun Kang
This map shows the geographic impact of Shi Eun Kang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shi Eun Kang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shi Eun Kang more than expected).
Fields of papers citing papers by Shi Eun Kang
This network shows the impact of papers produced by Shi Eun Kang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shi Eun Kang. The network helps show where Shi Eun Kang may publish in the future.
Co-authors
The 25 scholars most cited alongside Shi Eun Kang, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2011 | 363 | |
| 2 | 2011 | 256 | |
| 3 | 2013 | 94 | |
| 4 | 2016 | 30 | |
| 5 | 2011 | 11 |
About Shi Eun Kang
Shi Eun Kang is a scholar working on Molecular Biology, Oncology, Surgery, Cancer Research and Immunology, having authored 5 papers that have together received 754 indexed citations. Recurring topics across this work include Wnt/β-catenin signaling in development and cancer (3 papers), MicroRNA in disease regulation (2 papers), Tissue Engineering and Regenerative Medicine (2 papers), Cancer Cells and Metastasis (2 papers), interferon and immune responses (1 paper), Cytokine Signaling Pathways and Interactions (1 paper), RNA Research and Splicing (1 paper) and Cancer-related molecular mechanisms research (1 paper). The work is most often cited by research in Cancer Research (434 citations), Oncology (246 citations), Molecular Biology (587 citations), Cell Biology (45 citations) and Pathology and Forensic Medicine (34 citations). Shi Eun Kang has collaborated with scholars based in South Korea, United States and Poland. Frequent co-authors include Joo Kyung Ryu, Nam Hee Kim, So Young, Inhan Lee, Jong In Yook, Hyun Sil Kim, Stephen J. Weiss, Xiaoyan Li, Christopher A. Maher and Dojun Yoon. Their work appears in journals such as The Journal of Experimental Medicine, The Journal of Cell Biology, Cell Cycle, International Journal of Oncology and Science Signaling.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.