Sharad Khare

2.0k total citations
52 papers, 1.6k citations indexed

About

Sharad Khare is a scholar working on Molecular Biology, Oncology and Pathology and Forensic Medicine. According to data from OpenAlex, Sharad Khare has authored 52 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 16 papers in Oncology and 15 papers in Pathology and Forensic Medicine. Recurrent topics in Sharad Khare's work include Vitamin D Research Studies (9 papers), Fibroblast Growth Factor Research (7 papers) and Inflammatory mediators and NSAID effects (7 papers). Sharad Khare is often cited by papers focused on Vitamin D Research Studies (9 papers), Fibroblast Growth Factor Research (7 papers) and Inflammatory mediators and NSAID effects (7 papers). Sharad Khare collaborates with scholars based in United States, Canada and Zambia. Sharad Khare's co-authors include Marc Bissonnette, Ramesh K. Wali, Tushar Khare, Thomas A. Brasitus, Michael D. Sitrin, Hemant K. Roy, B. Scaglione-Sewell, Brijesh Shah, Sushesh Srivatsa Palakurthi and Srinath Palakurthi and has published in prestigious journals such as Journal of Clinical Investigation, Gastroenterology and Cancer Research.

In The Last Decade

Sharad Khare

52 papers receiving 1.5k citations

Peers

Sharad Khare
C Paraskeva United Kingdom
Adel Kardosh United States
Xianyang Yio United States
Reba Mustafi United States
Ursula Ehmer Germany
Hua Xiong China
Fujun Li China
Sharad Khare
Citations per year, relative to Sharad Khare Sharad Khare (= 1×) peers Hesheng Luo

Countries citing papers authored by Sharad Khare

Since Specialization
Citations

This map shows the geographic impact of Sharad Khare's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sharad Khare with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sharad Khare more than expected).

Fields of papers citing papers by Sharad Khare

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sharad Khare. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sharad Khare. The network helps show where Sharad Khare may publish in the future.

Co-authorship network of co-authors of Sharad Khare

This figure shows the co-authorship network connecting the top 25 collaborators of Sharad Khare. A scholar is included among the top collaborators of Sharad Khare based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sharad Khare. Sharad Khare is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Khare, Tushar, et al.. (2025). NAFLD and NAFLD Related HCC: Emerging Treatments and Clinical Trials. International Journal of Molecular Sciences. 26(1). 306–306. 9 indexed citations
2.
Zeng, Shuai, Zhen Lyu, Wei Zhang, et al.. (2021). Epigenetic DNA Modifications Upregulate SPRY2 in Human Colorectal Cancers. Cells. 10(10). 2632–2632. 4 indexed citations
4.
Shah, Brijesh, Sushesh Srivatsa Palakurthi, Tushar Khare, Sharad Khare, & Srinath Palakurthi. (2020). Natural proteins and polysaccharides in the development of micro/nano delivery systems for the treatment of inflammatory bowel disease. International Journal of Biological Macromolecules. 165(Pt A). 722–737. 68 indexed citations
5.
Wei, Tao, Katherine Shim, Kevin Wright, et al.. (2015). Atypical role of sprouty in colorectal cancer: sprouty repression inhibits epithelial–mesenchymal transition. Oncogene. 35(24). 3151–3162. 32 indexed citations
6.
Dougherty, Urszula, Reba Mustafi, Devkumar Mustafi, et al.. (2014). The Renin–Angiotensin System Mediates EGF Receptor–Vitamin D Receptor Cross-Talk in Colitis-Associated Colon Cancer. Clinical Cancer Research. 20(22). 5848–5859. 36 indexed citations
7.
Vaish, Vivek, Tushar Khare, Mukesh Verma, & Sharad Khare. (2014). Epigenetic Therapy for Colorectal Cancer. Methods in molecular biology. 1238. 771–782. 12 indexed citations
8.
Khare, Sharad & Mukesh Verma. (2012). Epigenetics of Colon Cancer. Methods in molecular biology. 863. 177–185. 46 indexed citations
9.
Khare, Sharad, et al.. (2008). Aberrant Crypt Foci in Colon Cancer Epidemiology. Methods in molecular biology. 472. 373–386. 36 indexed citations
10.
Fichera, Alessandro, Sujatha Jagadeeswaran, Urszula Dougherty, et al.. (2007). Epidermal Growth Factor Receptor Signaling Is Required for Microadenoma Formation in the Mouse Azoxymethane Model of Colonic Carcinogenesis. Cancer Research. 67(2). 827–835. 47 indexed citations
11.
Cohen, Greg, Reba Mustafi, Sandra Cerda, et al.. (2006). Epidermal Growth Factor Receptor Signaling Is Up-regulated in Human Colonic Aberrant Crypt Foci. Cancer Research. 66(11). 5656–5664. 45 indexed citations
12.
Wali, Ramesh K., Sharad Khare, Maria Tretiakova, et al.. (2002). Ursodeoxycholic Acid and F6-D3 Inhibit Aberrant Crypt Proliferation in the Rat Azoxymethane Model of Colon Cancer. Cancer Epidemiology and Prevention Biomarkers. 11(12). 1653–1662. 2 indexed citations
13.
Cerda, Sandra, Marc Bissonnette, B. Scaglione-Sewell, et al.. (2001). PKC-δ inhibits anchorage-dependent and -independent growth, enhances differentiation, and increases apoptosis in CaCo-2 cells. Gastroenterology. 120(7). 1700–1712. 50 indexed citations
14.
Wali, Ramesh K., Marc Bissonnette, S. Skarosi, et al.. (1998). 1,25-Dihydroxyvitamin D3Targets PKC-βII but Not PKC-α to the Basolateral Plasma Membranes of Rat Colonocytes. Biochemical and Biophysical Research Communications. 250(1). 48–52. 4 indexed citations
15.
Khare, Sharad, Merry J.G. Bolt, Ramesh K. Wali, et al.. (1997). 1,25 dihydroxyvitamin D3 stimulates phospholipase C-gamma in rat colonocytes: role of c-Src in PLC-gamma activation.. Journal of Clinical Investigation. 99(8). 1831–1841. 58 indexed citations
16.
Bissonnette, Marc, et al.. (1996). Protein kinase C isoforms in the chemopreventive effects of a novel vitamin D3 analogue in rat colonic tumorigenesis. Gastroenterology. 111(1). 118–126. 20 indexed citations
17.
Wali, Ramesh K., Marc Bissonnette, Sharad Khare, et al.. (1995). 1,25‐dihydroxyvitamin D3 stimulates the phosphorylation of two acidic membrane proteins of 42,000 and 48,000 daltons in rat colonocytes: An effect modulated by vitamin D status. Journal of Cellular Physiology. 162(2). 172–180. 5 indexed citations
18.
Khare, Sharad, Xiao-Ying Tien, David M. Wilson, et al.. (1994). Vitamin D status modulates rat colonic M3 muscarinic receptor characteristics and coupling to guanylate cyclase. FEBS Letters. 347(1). 27–30. 1 indexed citations
19.
Khare, Sharad, David M. Wilson, Xiao-Ying Tien, et al.. (1994). Protein Kinase C Mediates the Calcium-Induced Activation of Rat Colonic Particulate Guanylate Cyclase. Archives of Biochemistry and Biophysics. 314(1). 200–204. 9 indexed citations
20.
Khare, Sharad, David M. Wilson, Xiao-Ying Tien, & Thomas A. Brasitus. (1993). M3 muscarinic receptors on rat colonocytes are coupled to particulate guanylate cyclase activation. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1179(2). 234–237. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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