Vivek Vaish

784 total citations
30 papers, 639 citations indexed

About

Vivek Vaish is a scholar working on Pharmacology, Molecular Biology and Genetics. According to data from OpenAlex, Vivek Vaish has authored 30 papers receiving a total of 639 indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Pharmacology, 12 papers in Molecular Biology and 12 papers in Genetics. Recurrent topics in Vivek Vaish's work include Inflammatory mediators and NSAID effects (20 papers), Estrogen and related hormone effects (11 papers) and Cancer, Stress, Anesthesia, and Immune Response (10 papers). Vivek Vaish is often cited by papers focused on Inflammatory mediators and NSAID effects (20 papers), Estrogen and related hormone effects (11 papers) and Cancer, Stress, Anesthesia, and Immune Response (10 papers). Vivek Vaish collaborates with scholars based in India and United States. Vivek Vaish's co-authors include Sankar Nath Sanyal, Honit Piplani, Maria Marjorette O. Peña, Bimla Nehru, Lalita Tanwar, Minsub Shim, Kim Vaiphei, Manpreet Kaur Saini, Jasmeet Kaur and Mingxiao Feng and has published in prestigious journals such as Gastroenterology, International Journal of Molecular Sciences and Biochimica et Biophysica Acta (BBA) - General Subjects.

In The Last Decade

Vivek Vaish

30 papers receiving 631 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Vivek Vaish India 17 278 170 117 116 80 30 639
Naveena B. Janakiram United States 14 320 1.2× 141 0.8× 180 1.5× 82 0.7× 168 2.1× 28 800
Amy Pavone United States 15 313 1.1× 153 0.9× 141 1.2× 97 0.8× 135 1.7× 30 726
Haiming Ding United States 15 514 1.8× 100 0.6× 162 1.4× 45 0.4× 79 1.0× 30 898
Karen Holloway United Kingdom 11 472 1.7× 134 0.8× 167 1.4× 136 1.2× 169 2.1× 18 989
Honit Piplani United States 16 276 1.0× 72 0.4× 74 0.6× 44 0.4× 69 0.9× 25 580
Hsiao‐Mei Kuo Taiwan 20 456 1.6× 84 0.5× 88 0.8× 94 0.8× 147 1.8× 43 855
Asma Kheirollahi Iran 14 295 1.1× 57 0.3× 98 0.8× 75 0.6× 54 0.7× 27 707
Sarmistha Banerjee United States 12 627 2.3× 44 0.3× 206 1.8× 92 0.8× 198 2.5× 27 1.1k
Dinesh Thapa South Korea 14 404 1.5× 99 0.6× 83 0.7× 75 0.6× 150 1.9× 20 705
Jin Won Yang South Korea 19 592 2.1× 78 0.5× 140 1.2× 81 0.7× 123 1.5× 37 1.0k

Countries citing papers authored by Vivek Vaish

Since Specialization
Citations

This map shows the geographic impact of Vivek Vaish's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Vivek Vaish with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Vivek Vaish more than expected).

Fields of papers citing papers by Vivek Vaish

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Vivek Vaish. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Vivek Vaish. The network helps show where Vivek Vaish may publish in the future.

Co-authorship network of co-authors of Vivek Vaish

This figure shows the co-authorship network connecting the top 25 collaborators of Vivek Vaish. A scholar is included among the top collaborators of Vivek Vaish based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Vivek Vaish. Vivek Vaish is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sanyal, Sankar Nath, et al.. (2020). Imatinib exhibit synergistic pleiotropy in the prevention of colorectal cancer by suppressing proinflammatory, cell survival and angiogenic signaling. Cellular Signalling. 76. 109803–109803. 6 indexed citations
2.
Kaur, Gagandeep, et al.. (2019). Imatinib modulates pro-inflammatory microenvironment with angiostatic effects in experimental lung carcinogenesis. Inflammopharmacology. 28(1). 231–252. 7 indexed citations
3.
Piplani, Honit, et al.. (2015). Downregulation of PI3-K/Akt/PTEN pathway and activation of mitochondrial intrinsic apoptosis by Diclofenac and Curcumin in colon cancer. Molecular and Cellular Biochemistry. 402(1-2). 225–241. 68 indexed citations
4.
Vaish, Vivek, Tushar Khare, Mukesh Verma, & Sharad Khare. (2014). Epigenetic Therapy for Colorectal Cancer. Methods in molecular biology. 1238. 771–782. 12 indexed citations
5.
Vaish, Vivek, Qiong Zhang, & Sharad Khare. (2014). Tu1248 Ursodeoxycholic Acid Modulates Cell-Cell Adhesion Molecules to Reduce the Intestinal Permeability in Inflammatory Bowel Disease. Gastroenterology. 146(5). S–794. 1 indexed citations
7.
Vaish, Vivek, et al.. (2013). Sulindac and Celecoxib Regulate Cell Cycle Progression by p53/p21 Up Regulation to Induce Apoptosis During Initial Stages of Experimental Colorectal Cancer. Cell Biochemistry and Biophysics. 68(2). 301–319. 17 indexed citations
8.
Piplani, Honit, et al.. (2013). Dolastatin, along with Celecoxib, stimulates apoptosis by a mechanism involving oxidative stress, membrane potential change and PI3-K/AKT pathway down regulation. Biochimica et Biophysica Acta (BBA) - General Subjects. 1830(11). 5142–5156. 20 indexed citations
10.
Saini, Manpreet Kaur, Vivek Vaish, & Sankar Nath Sanyal. (2012). Role of cytokines and Jak3/Stat3 signaling in the 1,2-dimethylhydrazine dihydrochloride-induced rat model of colon carcinogenesis. European Journal of Cancer Prevention. 22(3). 215–228. 23 indexed citations
11.
Vaish, Vivek, et al.. (2012). Angiostatic Properties of Sulindac and Celecoxib in the Experimentally Induced Inflammatory Colorectal Cancer. Cell Biochemistry and Biophysics. 66(2). 205–227. 17 indexed citations
13.
Vaish, Vivek, et al.. (2012). Chemopreventive effects of NSAIDs as inhibitors of cyclooxygenase-2 and inducers of apoptosis in experimental lung carcinogenesis. Molecular and Cellular Biochemistry. 366(1-2). 89–99. 15 indexed citations
14.
Vaish, Vivek, et al.. (2012). Role of cytokines in experimentally induced lung cancer and chemoprevention by COX-2 selective inhibitor, etoricoxib. Molecular and Cellular Biochemistry. 372(1-2). 101–112. 16 indexed citations
15.
Kaur, Jasmeet, Vivek Vaish, & Sankar Nath Sanyal. (2012). COX-2 as a molecular target of colon cancer chemoprevention: Promise and reality. 2(3). 67–72. 5 indexed citations
16.
Vaish, Vivek & Sankar Nath Sanyal. (2011). Non steroidal anti-inflammatory drugs modulate the physicochemical properties of plasma membrane in experimental colorectal cancer: a fluorescence spectroscopic study. Molecular and Cellular Biochemistry. 358(1-2). 161–171. 13 indexed citations
17.
Vaish, Vivek & Sankar Nath Sanyal. (2011). Chemopreventive effects of NSAIDs on cytokines and transcription factors during the early stages of colorectal cancer. Pharmacological Reports. 63(5). 1210–1221. 35 indexed citations
18.
Vaish, Vivek, Lalita Tanwar, & Sankar Nath Sanyal. (2010). The role of NF-κB and PPARγ in experimentally induced colorectal cancer and chemoprevention by cyclooxygenase-2 inhibitors. Tumor Biology. 31(5). 427–436. 32 indexed citations
19.
Vaish, Vivek, Lalita Tanwar, Jasmeet Kaur, & Sankar Nath Sanyal. (2010). Chemopreventive Effects of Non-steroidal Anti-inflammatory Drugs in Early Neoplasm of Experimental Colorectal Cancer: an Apoptosome Study. Journal of Gastrointestinal Cancer. 42(4). 195–203. 18 indexed citations
20.
Tanwar, Lalita, Vivek Vaish, & Sankar Nath Sanyal. (2010). Chemopreventive role of etoricoxib (MK-0663) in experimental colon cancer: induction of mitochondrial proapoptotic factors. European Journal of Cancer Prevention. 19(4). 280–287. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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