Shaoli Lu

704 total citations
7 papers, 607 citations indexed

About

Shaoli Lu is a scholar working on Molecular Biology, Oncology and Pharmacology. According to data from OpenAlex, Shaoli Lu has authored 7 papers receiving a total of 607 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 3 papers in Oncology and 1 paper in Pharmacology. Recurrent topics in Shaoli Lu's work include Epigenetics and DNA Methylation (4 papers), Cancer-related Molecular Pathways (3 papers) and Cancer-related gene regulation (3 papers). Shaoli Lu is often cited by papers focused on Epigenetics and DNA Methylation (4 papers), Cancer-related Molecular Pathways (3 papers) and Cancer-related gene regulation (3 papers). Shaoli Lu collaborates with scholars based in China, United States and Italy. Shaoli Lu's co-authors include Wei‐Guo Zhu, Wen Zhou, Haiying Wang, Lipeng Wu, Gregory A. Otterson, Yu Yu, Ying Zhao, Guolin Chai, Yingqi Chen and Jia Sun and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Molecular and Cellular Biology.

In The Last Decade

Shaoli Lu

7 papers receiving 596 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Shaoli Lu China 7 522 151 50 46 45 7 607
Rachel S. Salamon United States 9 317 0.6× 69 0.5× 42 0.8× 38 0.8× 34 0.8× 10 436
Christine von Klitzing Germany 6 244 0.5× 116 0.8× 36 0.7× 33 0.7× 40 0.9× 7 340
Małgorzata Krześniak Poland 11 237 0.5× 108 0.7× 89 1.8× 18 0.4× 28 0.6× 23 325
Steven S. Foster United Kingdom 8 503 1.0× 132 0.9× 54 1.1× 28 0.6× 22 0.5× 8 554
Maike Schmitz Germany 5 249 0.5× 92 0.6× 35 0.7× 116 2.5× 34 0.8× 7 400
Alexander C. Jordan United States 7 307 0.6× 134 0.9× 87 1.7× 33 0.7× 38 0.8× 10 453
Lupin Jiang China 8 195 0.4× 80 0.5× 83 1.7× 45 1.0× 95 2.1× 12 352
Daphné Dupéré-Richer United States 9 347 0.7× 62 0.4× 51 1.0× 51 1.1× 22 0.5× 18 409
Agnieszka Chęcińska Netherlands 9 314 0.6× 182 1.2× 83 1.7× 108 2.3× 15 0.3× 10 477
Victoria Luchenko United States 10 372 0.7× 153 1.0× 29 0.6× 38 0.8× 29 0.6× 13 483

Countries citing papers authored by Shaoli Lu

Since Specialization
Citations

This map shows the geographic impact of Shaoli Lu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shaoli Lu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shaoli Lu more than expected).

Fields of papers citing papers by Shaoli Lu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shaoli Lu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shaoli Lu. The network helps show where Shaoli Lu may publish in the future.

Co-authorship network of co-authors of Shaoli Lu

This figure shows the co-authorship network connecting the top 25 collaborators of Shaoli Lu. A scholar is included among the top collaborators of Shaoli Lu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shaoli Lu. Shaoli Lu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Lu, Shaoli, Yang Yang, Yipeng Du, et al.. (2015). The transcription factor c-Fos coordinates with histone lysine-specific demethylase 2A to activate the expression of cyclooxygenase-2. Oncotarget. 6(33). 34704–34717. 7 indexed citations
2.
Chai, Guolin, Lian Li, Wen Zhou, et al.. (2008). HDAC Inhibitors Act with 5-aza-2′-Deoxycytidine to Inhibit Cell Proliferation by Suppressing Removal of Incorporated Abases in Lung Cancer Cells. PLoS ONE. 3(6). e2445–e2445. 59 indexed citations
3.
Wu, Lipeng, Xi Wang, Lian Li, et al.. (2008). Histone Deacetylase Inhibitor Depsipeptide Activates Silenced Genes through Decreasing both CpG and H3K9 Methylation on the Promoter. Molecular and Cellular Biology. 28(10). 3219–3235. 109 indexed citations
4.
Wang, Haiying, Ying Zhao, Lian Li, et al.. (2007). An ATM- and Rad3-related (ATR) Signaling Pathway and a Phosphorylation-Acetylation Cascade Are Involved in Activation of p53/p21Waf1/Cip1 in Response to 5-Aza-2′-deoxycytidine Treatment. Journal of Biological Chemistry. 283(5). 2564–2574. 50 indexed citations
5.
Zhao, Ying, Shaoli Lu, Lipeng Wu, et al.. (2006). Acetylation of p53 at Lysine 373/382 by the Histone Deacetylase Inhibitor Depsipeptide Induces Expression of p21Waf1/Cip1. Molecular and Cellular Biology. 26(7). 2782–2790. 244 indexed citations
6.
Lu, Shaoli, Jerrold Hill, & Howard Fillit. (2005). Impact of donepezil use in routine clinical practice on health care costs in patients with Alzheimer's disease and related dementias enrolled in a large medicare managed care plan: A case-control study. ˜The œAmerican journal of geriatric pharmacotherapy. 3(2). 92–102. 16 indexed citations
7.
Zhu, Wei‐Guo, Yang Ke, Peichang Wang, et al.. (2004). 5-Aza-2′-deoxycytidine Activates the p53/p21Waf1/Cip1 Pathway to Inhibit Cell Proliferation. Journal of Biological Chemistry. 279(15). 15161–15166. 122 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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