Seshagiri Duvvuri

1.1k citations
15 papers · 875 indexed · 1 hit paper · h-index 9

Seshagiri Duvvuri

15 papers receiving 865 citations

Hit Papers

Pharmacologic inhibition of fatty acid oxidation sensitiz...5522009202620142020100200300400500

Peers

Seshagiri Duvvuri
Comparison fields: 5 of 71
  • Cancer Research 380
  • Hematology 211
  • Molecular Biology 657
  • Biochemistry 60
  • Genetics 58
Replace Nabilah Khan with:
Nabilah Khan United States
Kumar Kaluarachchi United States
Longzhen Cui China
Kimie Kondo United States
Anagha Inguva Sheth United States
Andreas Janzer Germany
Pablo Baquero Spain
Angelo B. A. Laranjeira United States
Chengyin Min United States
Yanling Sun China
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Citations per field
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Nabilah Khan · 1×
Citations per year

Countries citing papers authored by Seshagiri Duvvuri

Since Specialization
Citations

This map shows the geographic impact of Seshagiri Duvvuri's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Seshagiri Duvvuri with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Seshagiri Duvvuri more than expected).

Fields of papers citing papers by Seshagiri Duvvuri

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Seshagiri Duvvuri. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Seshagiri Duvvuri. The network helps show where Seshagiri Duvvuri may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Seshagiri Duvvuri, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Seshagiri Duvvuri Line = papers co-authored together Seshagiri Duvvuri links everyone, so they are left out of the graph.

All Works

15 of 15 papers shown
#Work
1 201528
2 201430
3 20142
4 2013161
5 201210
6 20121
7 201216
8 201119
9 20113
10 20111
11 201010
12 20102
13
Pharmacologic inhibition of fatty acid oxidation sensitizes human leukemia cells to apoptosis inductionbreakdown →
2009552
14 20091
15 200239

About Seshagiri Duvvuri

Seshagiri Duvvuri is a scholar working on Hematology, Genetics and Oncology, having authored 15 papers that have together received 875 indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (8 papers), Cancer-related Molecular Pathways (7 papers), Chronic Lymphocytic Leukemia Research (3 papers), Autophagy in Disease and Therapy (2 papers), RNA modifications and cancer (2 papers), Lymphoma Diagnosis and Treatment (2 papers), Protein Degradation and Inhibitors (2 papers) and Amino Acid Enzymes and Metabolism (1 paper). The work is most often cited by research in Cancer Research (380 citations), Hematology (211 citations) and Molecular Biology (657 citations). Seshagiri Duvvuri has collaborated with scholars based in United States. Frequent co-authors include Michael Andreeff, Hagop M. Kantarjian, Marina Konopleva, Ismael Samudio, William G. Bornmann, Heinrich Taegtmeyer, Romain Harmancey, Michael Fiegl, Borys Korchin and Kumar Kaluarachchi. Their work appears in journals such as Blood, Cancer, Journal of Clinical Oncology, Journal of Biological Chemistry and Journal of Clinical Investigation.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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