Sergio Padilla‐Lopez

925 total citations
11 papers, 296 citations indexed

About

Sergio Padilla‐Lopez is a scholar working on Molecular Biology, Cell Biology and Physiology. According to data from OpenAlex, Sergio Padilla‐Lopez has authored 11 papers receiving a total of 296 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 5 papers in Cell Biology and 3 papers in Physiology. Recurrent topics in Sergio Padilla‐Lopez's work include Cellular transport and secretion (4 papers), Mitochondrial Function and Pathology (3 papers) and Lysosomal Storage Disorders Research (3 papers). Sergio Padilla‐Lopez is often cited by papers focused on Cellular transport and secretion (4 papers), Mitochondrial Function and Pathology (3 papers) and Lysosomal Storage Disorders Research (3 papers). Sergio Padilla‐Lopez collaborates with scholars based in United States, United Kingdom and Spain. Sergio Padilla‐Lopez's co-authors include David A. Pearce, Plácido Navas, Alejandro Martín‐Montalvo, Jared W. Benedict, Carlos Santos‐Ocaña, Cecı́lia Leão, Fernando Rodrigues, Paula Ludovico, João Laranjinha and Nuno S. Osório and has published in prestigious journals such as Journal of Biological Chemistry, Human Molecular Genetics and Biochimica et Biophysica Acta (BBA) - Biomembranes.

In The Last Decade

Sergio Padilla‐Lopez

11 papers receiving 287 citations

Peers

Sergio Padilla‐Lopez

PHYSI

GENET

PHYSI

Yuichi Yagita Japan

Arati Tripathi United States

Silvia A. Belmonte Argentina

Björn D. M. Bean Canada

Beichen Xie China

Sabine Weys Austria

Inés Noher de Halac Argentina

Jason Burgess Canada

Shady Saad Switzerland

Yuichi Yagita Japan

Sergio Padilla‐Lopez

345 ×1.8

52 ×0.5

PHYSI

68 ×0.8

27 ×0.8

GENET

10 ×0.4

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Citations per year, relative to Sergio Padilla‐Lopez Sergio Padilla‐Lopez (= 1×) peers Yuichi Yagita

Countries citing papers authored by Sergio Padilla‐Lopez

Since Specialization

Citations

This map shows the geographic impact of Sergio Padilla‐Lopez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sergio Padilla‐Lopez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sergio Padilla‐Lopez more than expected).

Fields of papers citing papers by Sergio Padilla‐Lopez

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sergio Padilla‐Lopez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sergio Padilla‐Lopez. The network helps show where Sergio Padilla‐Lopez may publish in the future.

Co-authorship network of co-authors of Sergio Padilla‐Lopez

This figure shows the co-authorship network connecting the top 25 collaborators of Sergio Padilla‐Lopez. A scholar is included among the top collaborators of Sergio Padilla‐Lopez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sergio Padilla‐Lopez. Sergio Padilla‐Lopez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

Lewis, Sara A., Somayeh Bakhtiari, Allan Bayat, et al.. (2023). AGAP1-associated endolysosomal trafficking abnormalities link gene–environment interactions in neurodevelopmental disorders. Disease Models & Mechanisms. 16(9). 5 indexed citations

Lewis, Sara A., Somayeh Bakhtiari, Jennifer Heim, et al.. (2021). Mutation in ZDHHC15 Leads to Hypotonic Cerebral Palsy, Autism, Epilepsy, and Intellectual Disability. Neurology Genetics. 7(4). e602–e602. 9 indexed citations

Cao, Siqi, Laura L. Smith, Sergio Padilla‐Lopez, et al.. (2017). Homozygous EEF1A2 mutation causes dilated cardiomyopathy, failure to thrive, global developmental delay, epilepsy and early death. Human Molecular Genetics. 26(18). 3545–3552. 25 indexed citations

Masuho, Ikuo, Mingyan Fang, Chunyu Geng, et al.. (2016). Homozygous GNAL mutation associated with familial childhood-onset generalized dystonia. Neurology Genetics. 2(3). e78–e78. 23 indexed citations

Martín‐Montalvo, Alejandro, Isabel González‐Mariscal, Sergio Padilla‐Lopez, et al.. (2013). The Phosphatase Ptc7 Induces Coenzyme Q Biosynthesis by Activating the Hydroxylase Coq7 in Yeast. Journal of Biological Chemistry. 288(39). 28126–28137. 38 indexed citations

Padilla‐Lopez, Sergio, et al.. (2011). BTN1, the Saccharomyces cerevisiae homolog to the human Batten disease gene, is involved in phospholipid distribution. Disease Models & Mechanisms. 5(2). 191–199. 15 indexed citations

Wolfe, Devin M., et al.. (2010). pH-dependent localization of Btn1p in the yeast model for Batten disease. Disease Models & Mechanisms. 4(1). 120–125. 11 indexed citations

Benedict, Jared W., et al.. (2009). Interaction between Sdo1p and Btn1p in the Saccharomyces cerevisiae model for Batten disease. Human Molecular Genetics. 19(5). 931–942. 27 indexed citations

Padilla‐Lopez, Sergio, et al.. (2009). Genetic evidence for the requirement of the endocytic pathway in the uptake of coenzyme Q6 in Saccharomyces cerevisiae. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1788(6). 1238–1248. 20 indexed citations

10.

Osório, Nuno S., Agostinho Carvalho, Agostinho J. Almeida, et al.. (2007). Nitric Oxide Signaling Is Disrupted in the Yeast Model for Batten Disease. Molecular Biology of the Cell. 18(7). 2755–2767. 52 indexed citations

11.

Padilla‐Lopez, Sergio & David A. Pearce. (2006). Saccharomyces cerevisiae Lacking Btn1p Modulate Vacuolar ATPase Activity to Regulate pH Imbalance in the Vacuole. Journal of Biological Chemistry. 281(15). 10273–10280. 71 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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