Seiko Ikezu
Impact in
- Neurology top 0.5%
- Neuroinflammation and Neurodegeneration Mechanisms
- Biological Psychiatry top 2%
- Tryptophan and brain disorders
Papers in
-
- Extracellular vesicles in disease 18
- RNA Interference and Gene Delivery 3
- Neurology 20
- Neuroinflammation and Neurodegeneration Mechanisms 20
- Traumatic Brain Injury and Neurovascular Disturbances 2
- Co-authors
- Tsuneya Ikezu (40 shared papers)Jennifer I. Luebke (3 shared papers)Maria Medalla (4 shared papers)Hirohide Asai (3 shared papers)Oleg Butovsky (3 shared papers)Tarik F. Haydar (2 shared papers)Satoshi Tsunoda (1 shared paper)Benjamin Wolozin (2 shared papers)
- Journals
- Alzheimer s & Dementia (11 papers)Acta Neuropathologica Communications (3 papers)Molecular Neurodegeneration (3 papers)Cell Reports (2 papers)Aging and Disease (2 papers)
- Partner nations
- United StatesJapanGermany
In The Last Decade
Seiko Ikezu
37 papers receiving 2.6k citations
Seiko Ikezu's Hit Papers
Peers
Comparison fields: 5 of 88
- Neurology 1.0k
- Biological Psychiatry 149
- Physiology 885
- Cancer Research 430
- Developmental Neuroscience 105
Countries citing papers authored by Seiko Ikezu
This map shows the geographic impact of Seiko Ikezu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Seiko Ikezu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Seiko Ikezu more than expected).
Fields of papers citing papers by Seiko Ikezu
This network shows the impact of papers produced by Seiko Ikezu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Seiko Ikezu. The network helps show where Seiko Ikezu may publish in the future.
Co-authors
The 25 scholars most cited alongside Seiko Ikezu, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 41 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Depletion of microglia and inhibition of exosome synthesis halt tau propagation Hit paper breakdown → | 2015 | 1241 |
| 2 | 2020 | 204 | |
| 3 | 2021 | 164 | |
| 4 | 2020 | 155 | |
| 5 | 2020 | 106 | |
| 6 | 2020 | 101 | |
| 7 | 2015 | 76 | |
| 8 | 2015 | 57 | |
| 9 | 2014 | 52 | |
| 10 | 2023 | 52 | |
| 11 | 2021 | 49 | |
| 12 | 2023 | 45 | |
| 13 | 2019 | 43 | |
| 14 | 2014 | 36 | |
| 15 | 2021 | 27 | |
| 16 | 2022 | 21 | |
| 17 | 2020 | 19 | |
| 18 | 2021 | 18 | |
| 19 | 2017 | 17 | |
| 20 | 2022 | 13 |
About Seiko Ikezu
Seiko Ikezu is a scholar working on Molecular Biology, Neurology, Physiology, Immunology and Cellular and Molecular Neuroscience, having authored 41 papers that have together received 2.6k indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (20 papers), Extracellular vesicles in disease (18 papers), Alzheimer's disease research and treatments (10 papers), MicroRNA in disease regulation (5 papers), Neurogenesis and neuroplasticity mechanisms (4 papers), Immune cells in cancer (4 papers), RNA Interference and Gene Delivery (3 papers) and Traumatic Brain Injury and Neurovascular Disturbances (2 papers). The work is most often cited by research in Neurology (1.0k citations), Biological Psychiatry (149 citations), Physiology (885 citations), Cancer Research (430 citations) and Developmental Neuroscience (105 citations). Seiko Ikezu has collaborated with scholars based in United States, Japan and Germany. Frequent co-authors include Tsuneya Ikezu, Jennifer I. Luebke, Maria Medalla, Hirohide Asai, Oleg Butovsky, Tarik F. Haydar, Satoshi Tsunoda, Benjamin Wolozin, Sebastian Kügler and Satoshi Muraoka. Their work appears in journals such as Alzheimer s & Dementia, Acta Neuropathologica Communications, Molecular Neurodegeneration, Cell Reports and Aging and Disease.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.