Sebastian Raimundo

1.6k total citations · 1 hit paper
9 papers, 1.2k citations indexed

About

Sebastian Raimundo is a scholar working on Pharmacology, Oncology and Molecular Biology. According to data from OpenAlex, Sebastian Raimundo has authored 9 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Pharmacology, 6 papers in Oncology and 3 papers in Molecular Biology. Recurrent topics in Sebastian Raimundo's work include Pharmacogenetics and Drug Metabolism (8 papers), Drug Transport and Resistance Mechanisms (6 papers) and Statistical Methods in Clinical Trials (2 papers). Sebastian Raimundo is often cited by papers focused on Pharmacogenetics and Drug Metabolism (8 papers), Drug Transport and Resistance Mechanisms (6 papers) and Statistical Methods in Clinical Trials (2 papers). Sebastian Raimundo collaborates with scholars based in Germany and United States. Sebastian Raimundo's co-authors include Ulrich M. Zanger, Michel Eichelbaum, Matthias Schwab, Joachim E. Fischer, Bernd O. Evert, Ernst‐Ulrich Griese, Kathrin Klein, Tanja Saußele, Elke Schaeffeler and Julia Blievernicht and has published in prestigious journals such as Clinical Pharmacology & Therapeutics, Clinica Chimica Acta and Drug Metabolism and Disposition.

In The Last Decade

Sebastian Raimundo

9 papers receiving 1.2k citations

Hit Papers

Cytochrome P450 2D6: overview and update on pharmacology,... 2004 2026 2011 2018 2004 200 400 600

Peers

Sebastian Raimundo
Mats Hidestrand United States
H.K. Crewe United Kingdom
Hyung‐Keun Roh South Korea
Nadia Soukhova United States
Sebastian Raimundo
Citations per year, relative to Sebastian Raimundo Sebastian Raimundo (= 1×) peers Qun‐Ying Yue

Countries citing papers authored by Sebastian Raimundo

Since Specialization
Citations

This map shows the geographic impact of Sebastian Raimundo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sebastian Raimundo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sebastian Raimundo more than expected).

Fields of papers citing papers by Sebastian Raimundo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sebastian Raimundo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sebastian Raimundo. The network helps show where Sebastian Raimundo may publish in the future.

Co-authorship network of co-authors of Sebastian Raimundo

This figure shows the co-authorship network connecting the top 25 collaborators of Sebastian Raimundo. A scholar is included among the top collaborators of Sebastian Raimundo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sebastian Raimundo. Sebastian Raimundo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Klein, Kathrin, Sebastian Raimundo, Tanja Saußele, et al.. (2007). A Natural Variant of the Heme-Binding Signature (R441C) Resulting in Complete Loss of Function of CYP2D6. Drug Metabolism and Disposition. 35(8). 1247–1250. 17 indexed citations
2.
Raimundo, Sebastian, et al.. (2006). A silent mutation (2939G>A, exon 6; CYP2D6*59) leading to impaired expression and function of CYP2D6. Pharmacogenetics and Genomics. 16(10). 767–770. 17 indexed citations
3.
Klein, Kathrin, Julia Blievernicht, Elke Schaeffeler, et al.. (2006). Impaired expression of CYP2D6 in intermediate metabolizers carrying the *41 allele caused by the intronic SNP 2988G>A: evidence for modulation of splicing events. Pharmacogenetics and Genomics. 16(10). 755–766. 73 indexed citations
4.
Zanger, Ulrich M., et al.. (2005). Limited Association of the 2988g>a Single Nucleotide Polymorphism with CYP2D6*41 in Black Subjects: Reply*. Clinical Pharmacology & Therapeutics. 77(3). 230–231. 2 indexed citations
5.
Zanger, Ulrich M., Sebastian Raimundo, & Michel Eichelbaum. (2004). Cytochrome P450 2D6: overview and update on pharmacology, genetics, biochemistry. Naunyn-Schmiedeberg s Archives of Pharmacology. 369(1). 23–37. 623 indexed citations breakdown →
6.
Raimundo, Sebastian. (2004). A novel intronic mutation, 2988G>A, with high predictivity for impaired function of cytochrome P450 2D6 in white subjects*1. Clinical Pharmacology & Therapeutics. 76(2). 128–138. 138 indexed citations
7.
Zanger, Ulrich M., Joachim E. Fischer, Sebastian Raimundo, et al.. (2001). Comprehensive analysis of the genetic factors determining expression and function of hepatic CYP2D6. Pharmacogenetics. 11(7). 573–585. 217 indexed citations
8.
Raimundo, Sebastian, Joachim E. Fischer, Michel Eichelbaum, et al.. (2000). Elucidation of the genetic basis of the common ‘intermediate metabolizer’ phenotype for drug oxidation by CYP2D6. Pharmacogenetics. 10(7). 577–581. 149 indexed citations
9.
Raimundo, Sebastian, et al.. (1999). Cloning and sequence of partial cDNAs encoding the human type V and VI adenylyl cyclases and subsequent RNA-quantification in various tissues. Clinica Chimica Acta. 285(1-2). 155–161. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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