Sebastian Ginzel

1.2k total citations
16 papers, 331 citations indexed

About

Sebastian Ginzel is a scholar working on Molecular Biology, Immunology and Public Health, Environmental and Occupational Health. According to data from OpenAlex, Sebastian Ginzel has authored 16 papers receiving a total of 331 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 6 papers in Immunology and 4 papers in Public Health, Environmental and Occupational Health. Recurrent topics in Sebastian Ginzel's work include Immune Cell Function and Interaction (5 papers), Acute Lymphoblastic Leukemia research (4 papers) and Immunodeficiency and Autoimmune Disorders (4 papers). Sebastian Ginzel is often cited by papers focused on Immune Cell Function and Interaction (5 papers), Acute Lymphoblastic Leukemia research (4 papers) and Immunodeficiency and Autoimmune Disorders (4 papers). Sebastian Ginzel collaborates with scholars based in Germany, Israel and China. Sebastian Ginzel's co-authors include Arndt Borkhardt, Michael Gombert, K. Bienemann, Svenja Daschkey, Elisabeth Salzer, Winfried F. Pickl, O. A. Haas, Ute Fischer, Kaan Boztuǧ and Elisabeth Förster‐Waldl and has published in prestigious journals such as Blood, Frontiers in Immunology and Haematologica.

In The Last Decade

Sebastian Ginzel

16 papers receiving 328 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sebastian Ginzel Germany 11 186 85 77 61 61 16 331
Michael Svatoň Czechia 11 139 0.7× 65 0.8× 84 1.1× 52 0.9× 58 1.0× 23 315
Ronald A van Soest Netherlands 8 206 1.1× 114 1.3× 106 1.4× 116 1.9× 70 1.1× 9 408
Shahrzad Bakhtiar Germany 14 191 1.0× 81 1.0× 82 1.1× 127 2.1× 61 1.0× 27 342
Veronika Kanderová Czechia 12 231 1.2× 46 0.5× 109 1.4× 62 1.0× 93 1.5× 24 372
K. Bienemann Germany 9 265 1.4× 125 1.5× 39 0.5× 70 1.1× 43 0.7× 12 367
Anja Troeger Germany 12 170 0.9× 106 1.2× 183 2.4× 91 1.5× 47 0.8× 35 493
K.‐P. Nera Finland 10 272 1.5× 55 0.6× 141 1.8× 41 0.7× 30 0.5× 16 422
Emily Y. Jen United States 7 82 0.4× 147 1.7× 119 1.5× 49 0.8× 46 0.8× 9 320
Emmanuel J. Volanakis United States 9 275 1.5× 71 0.8× 183 2.4× 77 1.3× 50 0.8× 15 499
Andrea Towlerton United States 10 216 1.2× 197 2.3× 82 1.1× 168 2.8× 24 0.4× 27 390

Countries citing papers authored by Sebastian Ginzel

Since Specialization
Citations

This map shows the geographic impact of Sebastian Ginzel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sebastian Ginzel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sebastian Ginzel more than expected).

Fields of papers citing papers by Sebastian Ginzel

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sebastian Ginzel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sebastian Ginzel. The network helps show where Sebastian Ginzel may publish in the future.

Co-authorship network of co-authors of Sebastian Ginzel

This figure shows the co-authorship network connecting the top 25 collaborators of Sebastian Ginzel. A scholar is included among the top collaborators of Sebastian Ginzel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sebastian Ginzel. Sebastian Ginzel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Ginzel, Sebastian, et al.. (2021). Towards the Detection and Visual Analysis of COVID-19 Infection Clusters. Fraunhofer-Publica (Fraunhofer-Gesellschaft). 3 indexed citations
2.
Kuhlen, Michaela, Sebastian Ginzel, Jörg Schaper, et al.. (2019). Mutated SON putatively causes a cancer syndrome comprising high-risk medulloblastoma combined with café-au-lait spots. Familial Cancer. 18(3). 353–358. 3 indexed citations
3.
Wimmer, Katharina, Martine Muleris, Olivier Lascols, et al.. (2018). Diagnostic challenges in a child with early onset desmoplastic medulloblastoma and homozygous variants in MSH2 and MSH6. European Journal of Human Genetics. 26(3). 440–444. 17 indexed citations
4.
Schlütermann, David, Andrea Hönscheid, Schafiq Nabhani, et al.. (2018). EBV Negative Lymphoma and Autoimmune Lymphoproliferative Syndrome Like Phenotype Extend the Clinical Spectrum of Primary Immunodeficiency Caused by STK4 Deficiency. Frontiers in Immunology. 9. 2400–2400. 32 indexed citations
5.
Brozou, Triantafyllia, Nan Qin, Jasmin Bartl, et al.. (2017). Congenital embryonal rhabdomyosarcoma caused by heterozygous concomitant PTCH1 and PTCH2 germline mutations. European Journal of Human Genetics. 26(1). 137–142. 16 indexed citations
6.
Nabhani, Schafiq, Hagit Miskin, Carina Levin, et al.. (2017). STAT3 gain-of-function mutations associated with autoimmune lymphoproliferative syndrome like disease deregulate lymphocyte apoptosis and can be targeted by BH3 mimetic compounds. Clinical Immunology. 181. 32–42. 33 indexed citations
7.
Daschkey, Svenja, K. Bienemann, Volker Schuster, et al.. (2016). Fatal Lymphoproliferative Disease in Two Siblings Lacking Functional FAAP24. Journal of Clinical Immunology. 36(7). 684–692. 12 indexed citations
8.
Nabhani, Schafiq, Sebastian Ginzel, Hagit Miskin, et al.. (2015). Deregulation of Fas ligand expression as a novel cause of autoimmune lymphoproliferative syndrome-like disease. Haematologica. 100(9). 1189–1198. 11 indexed citations
9.
Chen, Cai, Christoph Bartenhagen, Michael Gombert, et al.. (2015). Next-generation-sequencing of recurrent childhood high hyperdiploid acute lymphoblastic leukemia reveals mutations typically associated with high risk patients. Leukemia Research. 39(9). 990–1001. 28 indexed citations
10.
Linka, Yvonne, Sebastian Ginzel, Arndt Borkhardt, & Pablo Landgraf. (2014). Identification of TEL-AML1 (ETV6-RUNX1) associated DNA and its impact on mRNA and protein output using ChIP, mRNA expression arrays and SILAC. Genomics Data. 2. 85–88. 2 indexed citations
11.
Hoell, Jessica I., Michael Gombert, Sebastian Ginzel, et al.. (2014). Constitutional Mismatch Repair-deficiency and Whole-exome Sequencing as the Means of the Rapid Detection of the Causative MSH6 Defect. Klinische Pädiatrie. 226(06/07). 357–361. 13 indexed citations
12.
Linka, Yvonne, Sebastian Ginzel, Marcus Krüger, et al.. (2013). The impact of TEL-AML1 (ETV6-RUNX1) expression in precursor B cells and implications for leukaemia using three different genome-wide screening methods. Blood Cancer Journal. 3(10). e151–e151. 26 indexed citations
13.
Chen, Cai, Christoph Bartenhagen, Michael Gombert, et al.. (2013). Next‐generation‐sequencing‐based risk stratification and identification of new genes involved in structural and sequence variations in near haploid lymphoblastic leukemia. Genes Chromosomes and Cancer. 52(6). 564–579. 16 indexed citations
14.
Chen, Cai, Christoph Bartenhagen, Michael Gombert, et al.. (2013). Chromothripsis-Mediated Structural Variations and Clonal Evolution In Recurrent Childhood High Hyperdiploid Acute Lymphoblastic Leukemia. Blood. 122(21). 233–233. 2 indexed citations
15.
Salzer, Elisabeth, Svenja Daschkey, Sharon Choo, et al.. (2012). Combined immunodeficiency with life-threatening EBV-associated lymphoproliferative disorder in patients lacking functional CD27. Haematologica. 98(3). 473–478. 115 indexed citations
16.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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