Satoru Suzuki
- Endocrinology, Diabetes and Metabolism top 1%
- Molecular Biology top 10%
- Surgery top 5%
- Cardiology and Cardiovascular Medicine top 5%
- Physiology top 5%
- Co-authors
- Takahide MiyamotoKiyoshi HashizumeTeiji TakedaFumitake GejyoHideki KimuraLeslie J. DeGrootKazuo IchikawaHiroki Shimura
- Topics
- Thyroid Disorders and Treatments (22 papers)Renal Diseases and Glomerulopathies (21 papers)Radiation Dose and Imaging (19 papers)
- Partner nations
- JapanUnited StatesCanada
In The Last Decade
Satoru Suzuki
225 papers receiving 3.5k citations
Peers
Comparison fields: 5 of 146
- Endocrinology, Diabetes and Metabolism 1.0k
- Molecular Biology 809
- Surgery 534
- Cardiology and Cardiovascular Medicine 424
- Physiology 409
Countries citing papers authored by Satoru Suzuki
This map shows the geographic impact of Satoru Suzuki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Satoru Suzuki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Satoru Suzuki more than expected).
Fields of papers citing papers by Satoru Suzuki
This network shows the impact of papers produced by Satoru Suzuki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Satoru Suzuki. The network helps show where Satoru Suzuki may publish in the future.
Co-authorship network of co-authors of Satoru Suzuki
This figure shows the co-authorship network connecting the top 25 collaborators of Satoru Suzuki. A scholar is included among the top collaborators of Satoru Suzuki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Satoru Suzuki. Satoru Suzuki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 10 | |
| 3 | 3 | |
| 4 | 7 | |
| 5 | 6 | |
| 6 | 37 | |
| 7 | 13 | |
| 8 | 19 | |
| 9 | 14 | |
| 10 | 3 | |
| 11 | 15 | |
| 12 | 28 | |
| 13 | 2 | |
| 14 | 2 | |
| 15 | 2 | |
| 16 | 15 | |
| 17 | 10 | |
| 18 | 3 | |
| 19 | Dipyridamole reduces urinary albumin excretion in diabetic patients with normo- or microalbuminuria. | 18 |
| 20 | 2 |
About Satoru Suzuki
Satoru Suzuki is a scholar working on Nephrology, Endocrinology, Diabetes and Metabolism and Cardiology and Cardiovascular Medicine, having authored 240 papers that have together received 3.7k indexed citations. Recurring topics across this work include Thyroid Disorders and Treatments (22 papers), Renal Diseases and Glomerulopathies (21 papers) and Radiation Dose and Imaging (19 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (1.0k citations), Nephrology (298 citations) and Pharmaceutical Science (134 citations). Satoru Suzuki has collaborated with scholars based in Japan, United States and Canada. Frequent co-authors include Takahide Miyamoto, Kiyoshi Hashizume, Teiji Takeda, Fumitake Gejyo, Hideki Kimura, Leslie J. DeGroot, Kazuo Ichikawa, Hiroki Shimura, Mutsuhiro Kobayashi and Shinichi Suzuki. Their work appears in journals such as New England Journal of Medicine, Circulation and Blood.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.