Sarah Mouche

562 total citations
11 papers, 258 citations indexed

About

Sarah Mouche is a scholar working on Molecular Biology, Epidemiology and Hematology. According to data from OpenAlex, Sarah Mouche has authored 11 papers receiving a total of 258 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Epidemiology and 5 papers in Hematology. Recurrent topics in Sarah Mouche's work include Acute Myeloid Leukemia Research (5 papers), Autophagy in Disease and Therapy (3 papers) and Protein Degradation and Inhibitors (3 papers). Sarah Mouche is often cited by papers focused on Acute Myeloid Leukemia Research (5 papers), Autophagy in Disease and Therapy (3 papers) and Protein Degradation and Inhibitors (3 papers). Sarah Mouche collaborates with scholars based in Switzerland, France and United States. Sarah Mouche's co-authors include Ildikó Szántó, Michelangelo Foti, Stéphanie Carnésecchi, Patrick Muzzin, Éric Ogier‐Denis, Wei Wang, Yu‐Hua Tseng, Christoph A. Meier, Maša Katić and Klaus Steger and has published in prestigious journals such as Blood, Free Radical Biology and Medicine and Science Translational Medicine.

In The Last Decade

Sarah Mouche

11 papers receiving 255 citations

Peers

Sarah Mouche
Yaluan Ma China
Annie J. Kruger United States
Lkhagvasuren Munkhtulga Japan
Hamid Suhail United States
Michelle Melino Australia
Emma Koppe United Kingdom
Rebecca Pask United Kingdom
A. R. Mariani Italy
Li Fan China
Atsuko Asao Japan
Yaluan Ma China
Sarah Mouche
Citations per year, relative to Sarah Mouche Sarah Mouche (= 1×) peers Yaluan Ma

Countries citing papers authored by Sarah Mouche

Since Specialization
Citations

This map shows the geographic impact of Sarah Mouche's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sarah Mouche with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sarah Mouche more than expected).

Fields of papers citing papers by Sarah Mouche

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sarah Mouche. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sarah Mouche. The network helps show where Sarah Mouche may publish in the future.

Co-authorship network of co-authors of Sarah Mouche

This figure shows the co-authorship network connecting the top 25 collaborators of Sarah Mouche. A scholar is included among the top collaborators of Sarah Mouche based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sarah Mouche. Sarah Mouche is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Larrue, Clément, Sarah Mouche, & Jérôme Tamburini. (2024). The E3 ubiquitin ligase MARCH5 promotes mitochondrial fusion and cell-cycle progression in acute myeloid leukemia. Blood Advances. 9(2). 337–342. 1 indexed citations
2.
Larrue, Clément, Sarah Mouche, Paolo Angelino, et al.. (2024). Targeting ferritinophagy impairs quiescent cancer stem cells in acute myeloid leukemia in vitro and in vivo models. Science Translational Medicine. 16(757). eadk1731–eadk1731. 11 indexed citations
3.
Tamburini, Jérôme, Sarah Mouche, Clément Larrue, et al.. (2023). Very short insertions in the FLT3 gene are of therapeutic significance in acute myeloid leukemia. Blood Advances. 7(24). 7576–7580. 3 indexed citations
4.
Larrue, Clément, Sarah Mouche, Shan Lin, et al.. (2023). Mitochondrial fusion is a therapeutic vulnerability of acute myeloid leukemia. Leukemia. 37(4). 765–775. 31 indexed citations
5.
Pradier, Amandine, Laury Poulain, Sarah Mouche, et al.. (2021). AMP-Activated Protein Kinase Contributes to Apoptosis Induced by the Bcl-2 Inhibitor Venetoclax in Acute Myeloid Leukemia. Cancers. 13(23). 5966–5966. 2 indexed citations
6.
Bouzakri, Karim, Christelle Veyrat‐Durebex, Chet E. Holterman, et al.. (2020). Beta-Cell-Specific Expression of Nicotinamide Adenine Dinucleotide Phosphate Oxidase 5 Aggravates High-Fat Diet-Induced Impairment of Islet Insulin Secretion in Mice. Antioxidants and Redox Signaling. 32(9). 618–635. 10 indexed citations
7.
Poulain, Laury, Johanna Mondésir, Arnaud Jacquel, et al.. (2019). PKR-like Endoplasmic Reticulum Kinase Mediates Apoptosis Induced By Pharmacological AMP-Activated Protein Kinase Activation in Acute Myeloid Leukemia. Blood. 134(Supplement_1). 2552–2552. 1 indexed citations
8.
Carnésecchi, Stéphanie, Anne‐Laure Rougemont, James H. Doroshow, et al.. (2015). The NADPH oxidase NOX5 protects against apoptosis in ALK-positive anaplastic large-cell lymphoma cell lines. Free Radical Biology and Medicine. 84. 22–29. 15 indexed citations
9.
Li, Yun, Sarah Mouche, Tatjana Sajic, et al.. (2012). Deficiency in the NADPH oxidase 4 predisposes towards diet-induced obesity. International Journal of Obesity. 36(12). 1503–1513. 72 indexed citations
10.
Mouche, Sarah, Sanae Ben Mkaddem, Wei Wang, et al.. (2007). Reduced expression of the NADPH oxidase NOX4 is a hallmark of adipocyte differentiation. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1773(7). 1015–1027. 72 indexed citations
11.
Hezareh, Marjan, Ildikó Szántó, Sarah Mouche, et al.. (2004). Mechanisms of HIV Receptor and Co-Receptor Down-Regulation by Prostratin: Role of Conventional and Novel PKC Isoforms. Antiviral chemistry & chemotherapy. 15(4). 207–222. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026