Sara Ladu

4.2k total citations · 1 hit paper
30 papers, 3.0k citations indexed

About

Sara Ladu is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Sara Ladu has authored 30 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 12 papers in Oncology and 5 papers in Cancer Research. Recurrent topics in Sara Ladu's work include Cancer-related Molecular Pathways (10 papers), Cancer-related gene regulation (6 papers) and Cancer, Hypoxia, and Metabolism (5 papers). Sara Ladu is often cited by papers focused on Cancer-related Molecular Pathways (10 papers), Cancer-related gene regulation (6 papers) and Cancer, Hypoxia, and Metabolism (5 papers). Sara Ladu collaborates with scholars based in Italy, United States and Germany. Sara Ladu's co-authors include Diego F. Calvisi, Snorri S. Thorgeirsson, Valentina M. Factor, Elizabeth A. Conner, Miriam Farina, Alexis Gorden, Ju‐Seog Lee, Matthias Evert, Frank Dombrowski and Susie A. Lee and has published in prestigious journals such as Journal of Clinical Investigation, Gastroenterology and Hepatology.

In The Last Decade

Sara Ladu

30 papers receiving 3.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Ubiquitous Activation of Ras and Jak/Stat Pathways in Human HCC

2006 558 citations Diego F. Calvisi, Sara Ladu et al. Gastroenterology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Sara Ladu Italy	25	2.2k	880	664	460	449	30	3.0k
Haiquan Qiao China	29	1.2k 0.6×	710 0.8×	504 0.8×	275 0.6×	277 0.6×	60	2.3k
Antonio Cigliano Germany	29	1.4k 0.7×	851 1.0×	330 0.5×	271 0.6×	259 0.6×	70	2.3k
Jeonghoon Heo South Korea	16	1.4k 0.6×	763 0.9×	517 0.8×	389 0.8×	949 2.1×	33	2.6k
Maria M. Simile Italy	27	1.5k 0.7×	626 0.7×	442 0.7×	298 0.6×	175 0.4×	59	2.2k
Akiko Kokubu Japan	25	2.2k 1.0×	594 0.7×	649 1.0×	230 0.5×	125 0.3×	29	3.0k
Jin Ding China	40	3.0k 1.4×	2.0k 2.3×	1.0k 1.5×	630 1.4×	821 1.8×	97	4.8k
Prasanna K. Santhekadur India	23	994 0.5×	369 0.4×	411 0.6×	784 1.7×	222 0.5×	57	2.1k
Chunrong Yu United States	33	2.2k 1.0×	348 0.4×	669 1.0×	305 0.7×	126 0.3×	49	3.0k
Liankang Sun China	31	1.4k 0.7×	996 1.1×	606 0.9×	201 0.4×	126 0.3×	55	2.2k

Countries citing papers authored by Sara Ladu

Since Specialization

Citations

This map shows the geographic impact of Sara Ladu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sara Ladu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sara Ladu more than expected).

Fields of papers citing papers by Sara Ladu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sara Ladu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sara Ladu. The network helps show where Sara Ladu may publish in the future.

Co-authorship network of co-authors of Sara Ladu

This figure shows the co-authorship network connecting the top 25 collaborators of Sara Ladu. A scholar is included among the top collaborators of Sara Ladu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sara Ladu. Sara Ladu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Pesce, Mirko, Alessio Ferrone, Sara Ladu, et al.. (2014). The SHP-1 expression is associated with cytokines and psychopathological status in unmedicated first episode Schizophrenia patients. Brain Behavior and Immunity. 41. 251–260. 30 indexed citations

Evert, Matthias, Maddalena Frau, Maria Lauda Tomasi, et al.. (2013). Deregulation of DNA-dependent protein kinase catalytic subunit contributes to human hepatocarcinogenesis development and has a putative prognostic value. British Journal of Cancer. 109(10). 2654–2664. 42 indexed citations

Evert, Matthias, Diego F. Calvisi, Katja Evert, et al.. (2012). V-AKT murine thymoma viral oncogene homolog/mammalian target of rapamycin activation induces a module of metabolic changes contributing to growth in insulin-induced hepatocarcinogenesis. Hepatology. 55(5). 1473–1484. 50 indexed citations

Calvisi, Diego F., Maria M. Simile, Sara Ladu, et al.. (2011). Activation of v-Myb avian myeloblastosis viral oncogene homolog-like2 (MYBL2)-LIN9 complex contributes to human hepatocarcinogenesis and identifies a subset of hepatocellular carcinoma with mutant p53. Hepatology. 53(4). 1226–1236. 50 indexed citations

Ho, Coral, Chunmei Wang, Sandra Mattu, et al.. (2011). AKT (v-akt murine thymoma viral oncogene homolog 1) and N-Ras (neuroblastoma ras viral oncogene homolog) coactivation in the mouse liver promotes rapid carcinogenesis by way of mTOR (mammalian target of rapamycin complex 1), FOXM1 (forkhead box M1)/SKP2, and c-Myc pathways. Hepatology. 55(3). 833–845. 185 indexed citations

Lee, Susie A., Sara Ladu, Matthias Evert, et al.. (2010). Synergistic role of sprouty2 inactivation and c‐Met up‐regulation in mouse and human hepatocarcinogenesis†. Hepatology. 52(2). 506–517. 48 indexed citations

Calvisi, Diego F., Chunmei Wang, Coral Ho, et al.. (2010). Increased Lipogenesis, Induced by AKT-mTORC1-RPS6 Signaling, Promotes Development of Human Hepatocellular Carcinoma. Gastroenterology. 140(3). 1071–1083.e5. 468 indexed citations

Frau, Maddalena, Sara Ladu, Diego F. Calvisi, et al.. (2010). Mybl2 expression is under genetic control and contributes to determine a hepatocellular carcinoma susceptible phenotype. Journal of Hepatology. 55(1). 111–119. 41 indexed citations

Pellegrino, Rossella, Diego F. Calvisi, Sara Ladu, et al.. (2009). Oncogenic and Tumor Suppressive Roles of Polo-Like Kinases in Human Hepatocellular Carcinoma. Hepatology. 51(3). 857–868. 129 indexed citations

10.

Calvisi, Diego F., Federico Pinna, Sara Ladu, et al.. (2009). Forkhead box M1B is a determinant of rat susceptibility to hepatocarcinogenesis and sustains ERK activity in human HCC. Gut. 58(5). 679–687. 75 indexed citations

11.

Calvisi, Diego F., Federico Pinna, Sara Ladu, et al.. (2009). The degradation of cell cycle regulators by SKP2/CKS1 ubiquitin ligase is genetically controlled in rodent liver cancer and contributes to determine the susceptibility to the disease. International Journal of Cancer. 126(5). 1275–1281. 22 indexed citations

12.

Calvisi, Diego F., Federico Pinna, Sara Ladu, et al.. (2008). Dual-Specificity Phosphatase 1 Ubiquitination in Extracellular Signal-Regulated Kinase–Mediated Control of Growth in Human Hepatocellular Carcinoma. Cancer Research. 68(11). 4192–4200. 107 indexed citations

13.

Calvisi, Diego F., Sara Ladu, Alexis Gorden, et al.. (2007). Mechanistic and prognostic significance of aberrant methylation in the molecular pathogenesis of human hepatocellular carcinoma. Journal of Clinical Investigation. 117(9). 2713–2722. 313 indexed citations

14.

Calvisi, Diego F., Maria M. Simile, Sara Ladu, et al.. (2007). Altered methionine metabolism and global DNA methylation in liver cancer: Relationship with genomic instability and prognosis. International Journal of Cancer. 121(11). 2410–2420. 69 indexed citations

15.

Calvisi, Diego F., Sara Ladu, Alexis Gorden, et al.. (2006). Molecular pathogenesis of human hepatocellular carcinoma: Mechanistic and prognostic significance of aberrant methylation. Cancer Research. 66. 763–763. 2 indexed citations

16.

Ladu, Sara, Diego F. Calvisi, Elizabeth A. Conner, Valentina M. Factor, & Snorri S. Thorgeirsson. (2005). Co-expression of c-Myc and E2F1 in a mouse model of liver cancer suppresses apoptosis through activation of Akt/mTOR/p70S6K and COX-2 pathways: relevance for human hepatocellular carcinoma. Cancer Research. 65. 258–258. 1 indexed citations

17.

Calvisi, Diego F., Elizabeth A. Conner, Sara Ladu, et al.. (2005). Activation of the canonical Wnt/β-catenin pathway confers growth advantages in c-Myc/E2F1 transgenic mouse model of liver cancer. Journal of Hepatology. 42(6). 842–849. 83 indexed citations

18.

Calvisi, Diego F., Sara Ladu, Valentina M. Factor, & Snorri S. Thorgeirsson. (2004). Activation of β-catenin provides proliferative and invasive advantages in c-myc /TGF-α hepatocarcinogenesis promoted by phenobarbital. Carcinogenesis. 25(6). 901–908. 47 indexed citations

19.

Calvisi, Diego F., Sara Ladu, Koji Hironaka, Valentina M. Factor, & Snorri S. Thorgeirsson. (2004). Vitamin E down-modulates iNOS and NADPH oxidase in c-Myc/TGF-α transgenic mouse model of liver cancer. Journal of Hepatology. 41(5). 815–822. 70 indexed citations

20.

Calvisi, Diego F., Sara Ladu, Elizabeth A. Conner, Valentina M. Factor, & Snorri S. Thorgeirsson. (2004). Disregulation of E-cadherin in transgenic mouse models of liver cancer. Laboratory Investigation. 84(9). 1137–1147. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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