Samuel G. Jacobson
About
In The Last Decade
Samuel G. Jacobson
359 papers receiving 25.0k citations
Hit Papers
Peers
Comparison fields: 5 of 154
- Molecular Biology 22.1k
- Ophthalmology 12.2k
- Cellular and Molecular Neuroscience 6.4k
- Genetics 4.6k
- Radiology, Nuclear Medicine and Imaging 3.4k
Countries citing papers authored by Samuel G. Jacobson
This map shows the geographic impact of Samuel G. Jacobson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Samuel G. Jacobson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Samuel G. Jacobson more than expected).
Fields of papers citing papers by Samuel G. Jacobson
This network shows the impact of papers produced by Samuel G. Jacobson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Samuel G. Jacobson. The network helps show where Samuel G. Jacobson may publish in the future.
Co-authorship network of co-authors of Samuel G. Jacobson
This figure shows the co-authorship network connecting the top 25 collaborators of Samuel G. Jacobson. A scholar is included among the top collaborators of Samuel G. Jacobson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Samuel G. Jacobson. Samuel G. Jacobson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 21 | |
| 3 | 28 | |
| 4 | 129 | |
| 5 | Long-term preservation of photoreceptor function and structure following early-stage treatment by AAV-mediated gene augmentation in canine model of NPHP5 Leber congenital amaurosis | 1 |
| 6 | 12 | |
| 7 | 26 | |
| 8 | In Vivo Imaging of BEST1-Related Retinal Changes in the Canine Model | 1 |
| 9 | 51 | |
| 10 | Electroretinographic analyses of Rpe65-mutant rd12 mice: developing an in vivo bioassay for human gene therapy trials of Leber congenital amaurosis. | 30 |
| 11 | CRB1– Associated Retinal Degeneration: Macular Pigment and Lutein Supplementation | 0 |
| 12 | 27 | |
| 13 | Prevalence of Mutations in the RPE65, CRX, AIPL1, TULP1, GUCY2D and CRB1 Genes in Leber Congenital Amaurosis | 4 |
| 14 | Natural History of Retinal Function and Structure in the Rpe65-/- Murine Model of Leber Congenital Amaurosis | 2 |
| 15 | Ciliary Neurotrophic Factor (CNTF) Delivered by Adeno-associated Viruses (AAV) Leads to Photoreceptor Loss and Impairs Retinal Function in Wild-type Rats | 1 |
| 16 | In Vivo Micropathology of Best Macular Dystrophy | 1 |
| 17 | Genetics and phenotypes of RPE65 mutations in inherited retinal degeneration. | 214 |
| 18 | Allelic variation in the VMD2 gene in best disease and age-related macular degeneration. | 126 |
| 19 | A homozygous deletion in RPE65 in a small Sardinian family with autosomal recessive retinal dystrophy. | 23 |
| 20 | Photoreceptor function in patients with heterozygous peripherin/RDS gene mutations | 3 |
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.