S. W. Knight

2.6k citations
20 papers · 1.9k indexed · 1 hit paper · h-index 13
Topics
RNA regulation and disease (9 papers)Telomeres, Telomerase, and Senescence (8 papers)RNA modifications and cancer (5 papers)

In The Last Decade

S. W. Knight

20 papers receiving 1.9k citations

Hit Papers

X-linked dyskeratosis congenita is caused by mutations in...19982026200720161998200400600

Peers

S. W. Knight
Comparison fields: 5 of 72
  • Molecular Biology 1.3k
  • Physiology 1.2k
  • Genetics 254
  • Immunology 194
  • Genetics 150
Replace Natalie Erdmann with:
Natalie Erdmann Canada
Robert R. Adams United States
William T. Yewdell United States
S Kyo Japan
Gary de Jong Canada
Haiying Liu China
Veronika Butin‐Israeli United States
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S. W. Knight relative to Natalie Erdmann Canada Natalie Erdmann's profile →
Citations per field
00.5×2.8×
Natalie Erdmann · 1×
Citations per year

Countries citing papers authored by S. W. Knight

Since Specialization
Citations

This map shows the geographic impact of S. W. Knight's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S. W. Knight with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S. W. Knight more than expected).

Fields of papers citing papers by S. W. Knight

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by S. W. Knight. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S. W. Knight. The network helps show where S. W. Knight may publish in the future.

Co-authorship network of co-authors of S. W. Knight

This figure shows the co-authorship network connecting the top 25 collaborators of S. W. Knight. A scholar is included among the top collaborators of S. W. Knight based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with S. W. Knight. S. W. Knight is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 236
2 66
3 174
4 45
5 46
6 187
7 163
8 3
9 43
10
X-linked dyskeratosis congenita is caused by mutations in a highly conserved gene with putative nucleolar functionsbreakdown →
708
11 133
12 30
13 7
14 52
15 5
16 4
17 24
18 8
19 6
20 3

About S. W. Knight

S. W. Knight is a scholar working on Physiology, Molecular Biology and Cell Biology, having authored 20 papers that have together received 1.9k indexed citations. Recurring topics across this work include RNA regulation and disease (9 papers), Telomeres, Telomerase, and Senescence (8 papers) and RNA modifications and cancer (5 papers). The work is most often cited by research in Physiology (1.2k citations), Aging (79 citations) and Molecular Biology (1.3k citations). S. W. Knight has collaborated with scholars based in United Kingdom, Germany and United States. Frequent co-authors include Philip J. Mason, Inderjeet Dokal, Tom Vulliamy, Annemarie Poustka, Nina S. Heiss, Stefan Wiemann, Sabine M. Klauck, Amanda J. Walne, Anna Marrone and T. Vulliamy. Their work appears in journals such as Nature Genetics, The EMBO Journal and Blood.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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