Ryuta Ishimura
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- Effects and risks of endocrine disrupting chemicals 6
- Toxic Organic Pollutants Impact 6
- Air Quality and Health Impacts 2
- Obstetrics and Gynecology top 5%
- Pregnancy and preeclampsia studies 4
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- Carcinogens and Genotoxicity Assessment 3
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- RNA modifications and cancer 4
- Reproductive Medicine top 10%
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- Birth, Development, and Health 3
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- Hormonal and reproductive studies 3
Ryuta Ishimura
20 papers receiving 1.2k citations
Peers
Comparison fields: 5 of 91
- Health, Toxicology and Mutagenesis 301
- Obstetrics and Gynecology 91
- Cancer Research 148
- Molecular Biology 669
- Reproductive Medicine 75
Countries citing papers authored by Ryuta Ishimura
This map shows the geographic impact of Ryuta Ishimura's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ryuta Ishimura with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ryuta Ishimura more than expected).
Fields of papers citing papers by Ryuta Ishimura
This network shows the impact of papers produced by Ryuta Ishimura. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ryuta Ishimura. The network helps show where Ryuta Ishimura may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Ryuta Ishimura, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2020 | 31 | |
| 2 | 2016 | 161 | |
| 3 | 2014 | 328 | |
| 4 | 2009 | 10 | |
| 5 | 2008 | 33 | |
| 6 | 2008 | 28 | |
| 7 | 2006 | 31 | |
| 8 | 2005 | 21 | |
| 9 | 2004 | 114 | |
| 10 | 2004 | 13 | |
| 11 | 2003 | 45 | |
| 12 | 2003 | 60 | |
| 13 | 2002 | 22 | |
| 14 | 2002 | 44 | |
| 15 | 2002 | 35 | |
| 16 | 2001 | 1 | |
| 17 | 2001 | 133 | |
| 18 | 1998 | 29 | |
| 19 | 1997 | 47 | |
| 20 | 1995 | 13 |
About Ryuta Ishimura
Ryuta Ishimura is a scholar working on Health, Toxicology and Mutagenesis, Obstetrics and Gynecology, Reproductive Medicine, Endocrinology, Diabetes and Metabolism and Physiology, having authored 20 papers that have together received 1.2k indexed citations. Recurring topics across this work include Effects and risks of endocrine disrupting chemicals (6 papers), Toxic Organic Pollutants Impact (6 papers), Pregnancy and preeclampsia studies (4 papers), RNA modifications and cancer (4 papers), Carcinogens and Genotoxicity Assessment (3 papers), Birth, Development, and Health (3 papers), Hormonal and reproductive studies (3 papers) and Air Quality and Health Impacts (2 papers). The work is most often cited by research in Health, Toxicology and Mutagenesis (301 citations), Obstetrics and Gynecology (91 citations), Cancer Research (148 citations), Molecular Biology (669 citations) and Reproductive Medicine (75 citations). Ryuta Ishimura has collaborated with scholars based in Japan, United States and France. Frequent co-authors include Chiharu Tohyama, Seiichiroh Ohsako, Susan L. Ackerman, Jeffrey H. Chuang, Gábor Nagy, Iván Dotú, Motoharu Sakaue, Takashige Kawakami, Yasuharu Nishimura and Paul Schimmel. Their work appears in journals such as Biology of Reproduction, Toxicology and Applied Pharmacology, eLife, Journal of Reproduction and Development and Molecular and Cellular Endocrinology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.