Rufina Schuligoi
- Physiology top 1%
- Asthma and respiratory diseases 24
- Pain Mechanisms and Treatments 23
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- Neuropeptides and Animal Physiology 39
- Nerve injury and regeneration 10
- Pharmacology top 1%
- Inflammatory mediators and NSAID effects 30
- Sensory Systems top 2%
- Biochemistry top 1%
- Eicosanoids and Hypertension Pharmacology 13
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- Allergic Rhinitis and Sensitization 7
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- Antiplatelet Therapy and Cardiovascular Diseases 7
- Co-authors
- Josef DonnererÁkos HeinemannRainer AmannChristoph SteinBernhard A. PeskarGunther MarscheViktória KónyaPeter Holzer
- Partner nations
- AustriaGermanyUnited Kingdom
In The Last Decade
Rufina Schuligoi
111 papers receiving 4.0k citations
Hit Papers
Peers
Comparison fields: 5 of 126
- Physiology 1.6k
- Cellular and Molecular Neuroscience 1.1k
- Pharmacology 890
- Sensory Systems 253
- Biochemistry 322
Countries citing papers authored by Rufina Schuligoi
This map shows the geographic impact of Rufina Schuligoi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rufina Schuligoi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rufina Schuligoi more than expected).
Fields of papers citing papers by Rufina Schuligoi
This network shows the impact of papers produced by Rufina Schuligoi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rufina Schuligoi. The network helps show where Rufina Schuligoi may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Rufina Schuligoi, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 44 | |
| 2 | 2012 | 48 | |
| 3 | 2011 | 51 | |
| 4 | 2010 | 28 | |
| 5 | 2009 | 18 | |
| 6 | 2008 | 101 | |
| 7 | 2008 | 8 | |
| 8 | 2007 | 61 | |
| 9 | 2007 | 13 | |
| 10 | 2004 | 10 | |
| 11 | 2003 | 93 | |
| 12 | 2003 | 6 | |
| 13 | 2001 | 56 | |
| 14 | 2000 | 5 | |
| 15 | 1999 | 15 | |
| 16 | 1999 | 11 | |
| 17 | 1998 | 13 | |
| 18 | 1996 | 69 | |
| 19 | 1996 | 13 | |
| 20 | 1994 | 12 |
About Rufina Schuligoi
Rufina Schuligoi is a scholar working on Cellular and Molecular Neuroscience, Physiology and Pharmacology, having authored 111 papers that have together received 4.1k indexed citations. Recurring topics across this work include Neuropeptides and Animal Physiology (39 papers), Inflammatory mediators and NSAID effects (30 papers), Asthma and respiratory diseases (24 papers), Pain Mechanisms and Treatments (23 papers), Eicosanoids and Hypertension Pharmacology (13 papers), Nerve injury and regeneration (10 papers), Allergic Rhinitis and Sensitization (7 papers) and Antiplatelet Therapy and Cardiovascular Diseases (7 papers). The work is most often cited by research in Physiology (1.6k citations), Cellular and Molecular Neuroscience (1.1k citations) and Pharmacology (890 citations). Rufina Schuligoi has collaborated with scholars based in Austria, Germany and United Kingdom. Frequent co-authors include Josef Donnerer, Ákos Heinemann, Rainer Amann, Christoph Stein, Bernhard A. Peskar, Gunther Marsche, Viktória Kónya, Peter Holzer, Eva M. Sturm and B. M. Peskar. Their work appears in journals such as Neuron, The Journal of Immunology and Gastroenterology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.