Roy C. Mootoosamy

849 total citations
10 papers, 728 citations indexed

About

Roy C. Mootoosamy is a scholar working on Molecular Biology, Genetics and Neurology. According to data from OpenAlex, Roy C. Mootoosamy has authored 10 papers receiving a total of 728 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 4 papers in Genetics and 2 papers in Neurology. Recurrent topics in Roy C. Mootoosamy's work include Developmental Biology and Gene Regulation (7 papers), Hedgehog Signaling Pathway Studies (3 papers) and Craniofacial Disorders and Treatments (3 papers). Roy C. Mootoosamy is often cited by papers focused on Developmental Biology and Gene Regulation (7 papers), Hedgehog Signaling Pathway Studies (3 papers) and Craniofacial Disorders and Treatments (3 papers). Roy C. Mootoosamy collaborates with scholars based in United Kingdom, United States and Denmark. Roy C. Mootoosamy's co-authors include Susanne Dietrich, Andrew Lumsden, F. Schubert, Lúcia Elvira Alvares, Andrew B. Lassar, Clifford J. Tabin, Hervé Kempf, Arhat Abzhanov, Eldad Tzahor and Colin Thorpe and has published in prestigious journals such as Genes & Development, Development and Journal of Cell Science.

In The Last Decade

Roy C. Mootoosamy

10 papers receiving 715 citations

Peers

Roy C. Mootoosamy

MB

GENET

NEURO

CB

GENET

Patricia Blanchet France

Magdalena Beneyto Spain

Shannon W. Davis United States

Stavros Malas Cyprus

David E. Bergstrom United States

David Baux France

Derek Silvius United States

Moira Crosier United Kingdom

Mohamed Drira Tunisia

Rachel E. Hardisty-Hughes United Kingdom

Patricia Blanchet France

Roy C. Mootoosamy

501 ×0.8

MB

245 ×1.5

GENET

84 ×1.2

NEURO

57 ×0.9

CB

34 ×0.6

GENET

Citations per year, relative to Roy C. Mootoosamy Roy C. Mootoosamy (= 1×) peers Patricia Blanchet

Countries citing papers authored by Roy C. Mootoosamy

Since Specialization

Citations

This map shows the geographic impact of Roy C. Mootoosamy's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Roy C. Mootoosamy with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Roy C. Mootoosamy more than expected).

Fields of papers citing papers by Roy C. Mootoosamy

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Roy C. Mootoosamy. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Roy C. Mootoosamy. The network helps show where Roy C. Mootoosamy may publish in the future.

Co-authorship network of co-authors of Roy C. Mootoosamy

This figure shows the co-authorship network connecting the top 25 collaborators of Roy C. Mootoosamy. A scholar is included among the top collaborators of Roy C. Mootoosamy based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Roy C. Mootoosamy. Roy C. Mootoosamy is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Jen, Angela, Roy C. Mootoosamy, Alice Warley, et al.. (2010). Neuronal low-density lipoprotein receptor-related protein 1 binds and endocytoses prion fibrils via receptor cluster 4. Journal of Cell Science. 123(2). 246–255. 49 indexed citations

2.

Mootoosamy, Roy C., Claire Sunyach, Christian Jacobsen, et al.. (2008). LRP1 controls biosynthetic and endocytic trafficking of neuronal prion protein. Journal of Cell Science. 121(6). 773–783. 91 indexed citations

3.

Scheven, Gudrun von, Lúcia Elvira Alvares, Roy C. Mootoosamy, & Susanne Dietrich. (2006). Neural tube derived signals and Fgf8 act antagonistically to specify eye versus mandibular arch muscles. Development. 133(14). 2731–2745. 51 indexed citations

4.

Alvares, Lúcia Elvira, F. Schubert, Colin Thorpe, et al.. (2003). Intrinsic, Hox-Dependent Cues Determine the Fate of Skeletal Muscle Precursors. Developmental Cell. 5(3). 379–390. 92 indexed citations

5.

Tzahor, Eldad, Hervé Kempf, Roy C. Mootoosamy, et al.. (2003). Antagonists of Wnt and BMP signaling promote the formation of vertebrate head muscle. Genes & Development. 17(24). 3087–3099. 178 indexed citations

6.

Schubert, F., Roy C. Mootoosamy, Anthony Graham, et al.. (2002). Wnt6 marks sites of epithelial transformations in the chick embryo. Mechanisms of Development. 114(1-2). 143–148. 82 indexed citations

7.

Mootoosamy, Roy C., et al.. (2002). Hypaxial Muscle Development. Results and problems in cell differentiation. 38. 127–141. 5 indexed citations

8.

Mootoosamy, Roy C. & Susanne Dietrich. (2002). Distinct regulatory cascades for head and trunk myogenesis. Development. 129(3). 573–583. 144 indexed citations

9.

Schubert, F., Susanne Dietrich, Roy C. Mootoosamy, Susan C. Chapman, & Andrew Lumsden. (2001). Lbx1 marks a subset of interneurons in chick hindbrain and spinal cord. Mechanisms of Development. 101(1-2). 181–185. 24 indexed citations

10.

Grierson, Andrew J., Roy C. Mootoosamy, & Christopher C.J. Miller. (1999). Polyglutamine repeat length influences human androgen receptor/c-Jun mediated transcription. Neuroscience Letters. 277(1). 9–12. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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