Rosário Santos

2.5k citations
79 papers · 821 indexed · h-index 19

Rosário Santos

73 papers receiving 796 citations

Peers

Rosário Santos
Comparison fields: 5 of 85
  • Genetics 115
  • Genetics 224
  • Pediatrics, Perinatology and Child Health 153
  • Molecular Biology 491
  • Cell Biology 90
Replace Alison J. Coffey with:
Alison J. Coffey United Kingdom
Faiqa Imtiaz Saudi Arabia
Reeval Segel Israel
Eckhard Korsch Germany
Gözde Yeşil Türkiye
Natalia Martín United States
Rita Genesio Italy
Wanda E. Filipiak United States
Oscar Suzuki United States
Emma L. Baple United Kingdom
Rosário Santos relative to Alison J. Coffey United Kingdom Alison J. Coffey's profile →
Citations per field
00.5×3.6×
Alison J. Coffey · 1×
Citations per year

Countries citing papers authored by Rosário Santos

Since Specialization
Citations

This map shows the geographic impact of Rosário Santos's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rosário Santos with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rosário Santos more than expected).

Fields of papers citing papers by Rosário Santos

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rosário Santos. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rosário Santos. The network helps show where Rosário Santos may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Rosário Santos, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Rosário Santos Line = papers co-authored together Rosário Santos links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20251
2 20242
3 20221
4 20211
5 20211
6 20209
7 202039
8 20201
9 20195
10 20186
11 201619
12 20143
13 201429
14 20134
15 201127
16
Gene symbol: UGT1A1. Disease: Crigler-Najjar syndrome 1.
20082
17 20078
18 200411
19
[Glucose-6-phosphate dehydrogenase deficiency, neonatal hyperbilirubinemia and Gilbert syndrome].
20033
20 19985

About Rosário Santos

Rosário Santos is a scholar working on Genetics, Genetics and Immunology and Allergy, having authored 79 papers that have together received 821 indexed citations. Recurring topics across this work include Muscle Physiology and Disorders (16 papers), Genetics and Neurodevelopmental Disorders (12 papers), Neonatal Health and Biochemistry (10 papers), RNA Research and Splicing (10 papers), RNA modifications and cancer (7 papers), Congenital heart defects research (6 papers), Genetic Neurodegenerative Diseases (6 papers) and Cardiomyopathy and Myosin Studies (6 papers). The work is most often cited by research in Genetics (115 citations), Genetics (224 citations) and Pediatrics, Perinatology and Child Health (153 citations). Rosário Santos has collaborated with scholars based in Portugal, Netherlands and Brazil. Frequent co-authors include Jorge Oliveira, Elı́sio Costa, Mário Sousa, Ana Gonçalves, Elsa Bronze-da-Rocha, Rute Pereira, Manuel Melo Pires, Paula Jorge, Ricardo Taipa and Teresinha Evangelista. Their work appears in journals such as SHILAP Revista de lepidopterología, Blood and PLoS ONE.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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