Rosalba Satta

1.5k total citations
16 papers, 1.1k citations indexed

About

Rosalba Satta is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Genetics. According to data from OpenAlex, Rosalba Satta has authored 16 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 8 papers in Cellular and Molecular Neuroscience and 7 papers in Genetics. Recurrent topics in Rosalba Satta's work include Genetics and Neurodevelopmental Disorders (4 papers), Epigenetics and DNA Methylation (4 papers) and Receptor Mechanisms and Signaling (4 papers). Rosalba Satta is often cited by papers focused on Genetics and Neurodevelopmental Disorders (4 papers), Epigenetics and DNA Methylation (4 papers) and Receptor Mechanisms and Signaling (4 papers). Rosalba Satta collaborates with scholars based in United States, France and Italy. Rosalba Satta's co-authors include Alessandro Guidotti, Dennis R. Grayson, Amy W. Lasek, E. Costa, John M. Davis, Erbo Dong, Christine J. DiDonato, Cathleen Lutz, Christopher R. Heier and Marin Veldić and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and PLoS ONE.

In The Last Decade

Rosalba Satta

16 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Rosalba Satta United States	14	693	346	253	163	138	16	1.1k
Mira Jakovcevski Germany	22	1.2k 1.7×	442 1.3×	251 1.0×	114 0.7×	38 0.3×	35	1.8k
Dongho Geum South Korea	21	552 0.8×	105 0.3×	242 1.0×	85 0.5×	56 0.4×	51	1.4k
Zoya Marinova Switzerland	19	1.1k 1.5×	263 0.8×	612 2.4×	118 0.7×	61 0.4×	32	1.8k
Galit Shaltiel Israel	18	535 0.8×	240 0.7×	317 1.3×	142 0.9×	27 0.2×	29	1.2k
Gilles Maussion Canada	22	903 1.3×	430 1.2×	255 1.0×	289 1.8×	25 0.2×	36	1.8k
John D. H. Stead Canada	19	455 0.7×	244 0.7×	274 1.1×	187 1.1×	24 0.2×	25	1.3k
Luciana Romina Frick United States	25	379 0.5×	138 0.4×	379 1.5×	269 1.7×	23 0.2×	38	1.5k
Gursharan Kalsi United Kingdom	20	660 1.0×	637 1.8×	280 1.1×	68 0.4×	33 0.2×	56	1.4k
Tatiana M. Kazdoba United States	18	543 0.8×	426 1.2×	463 1.8×	67 0.4×	19 0.1×	24	1.3k
Astrid Vallès Netherlands	20	542 0.8×	189 0.5×	401 1.6×	199 1.2×	21 0.2×	33	1.5k

Countries citing papers authored by Rosalba Satta

Since Specialization

Citations

This map shows the geographic impact of Rosalba Satta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rosalba Satta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rosalba Satta more than expected).

Fields of papers citing papers by Rosalba Satta

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rosalba Satta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rosalba Satta. The network helps show where Rosalba Satta may publish in the future.

Co-authorship network of co-authors of Rosalba Satta

This figure shows the co-authorship network connecting the top 25 collaborators of Rosalba Satta. A scholar is included among the top collaborators of Rosalba Satta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rosalba Satta. Rosalba Satta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Satta, Rosalba, Donghong He, Chang You, et al.. (2020). Estrogen Receptor α Regulates Ethanol Excitation of Ventral Tegmental Area Neurons and Binge Drinking in Female Mice. Journal of Neuroscience. 40(27). 5196–5207. 41 indexed citations

Satta, Rosalba, Roderick G. Davis, Young Ah Goo, et al.. (2019). Multidimensional Top-Down Proteomics of Brain-Region-Specific Mouse Brain Proteoforms Responsive to Cocaine and Estradiol. Journal of Proteome Research. 18(11). 3999–4012. 13 indexed citations

You, Chang, et al.. (2017). Estradiol increases the sensitivity of ventral tegmental area dopamine neurons to dopamine and ethanol. PLoS ONE. 12(11). e0187698–e0187698. 73 indexed citations

Satta, Rosalba, et al.. (2017). Ovarian Hormones Contribute to High Levels of Binge‐Like Drinking by Female Mice. Alcoholism Clinical and Experimental Research. 42(2). 286–294. 73 indexed citations

Satta, Rosalba, et al.. (2017). Estrogen Receptor β in the Nucleus Accumbens Regulates the Rewarding Properties of Cocaine in Female Mice. The International Journal of Neuropsychopharmacology. 21(4). 382–392. 30 indexed citations

Maloku, Ekrem, Bashkim Kadriu, Adrian Zhubi, et al.. (2011). Selective α4β2 Nicotinic Acetylcholine Receptor Agonists Target Epigenetic Mechanisms in Cortical GABAergic Neurons. Neuropsychopharmacology. 36(7). 1366–1374. 32 indexed citations

Heier, Christopher R., Rosalba Satta, Cathleen Lutz, & Christine J. DiDonato. (2010). Arrhythmia and cardiac defects are a feature of spinal muscular atrophy model mice. Human Molecular Genetics. 19(20). 3906–3918. 141 indexed citations

Guidotti, A, James Auta, Y. Chen, et al.. (2010). Epigenetic GABAergic targets in schizophrenia and bipolar disorder. Neuropharmacology. 60(7-8). 1007–1016. 176 indexed citations

Guidotti, Alessandro, Erbo Dong, Marija Kundaković, et al.. (2008). Characterization of the action of antipsychotic subtypes on valproate-induced chromatin remodeling. Trends in Pharmacological Sciences. 30(2). 55–60. 90 indexed citations

10.

Costa, Erminio, Ying Chen, Erbo Dong, et al.. (2008). GABAergic promoter hypermethylation as a model to study the neurochemistry of schizophrenia vulnerability. Expert Review of Neurotherapeutics. 9(1). 87–98. 46 indexed citations

11.

Satta, Rosalba, Ekrem Maloku, Adrian Zhubi, et al.. (2008). Nicotine decreases DNA methyltransferase 1 expression and glutamic acid decarboxylase 67 promoter methylation in GABAergic interneurons. Proceedings of the National Academy of Sciences. 105(42). 16356–16361. 148 indexed citations

12.

Maloku, Ekrem, Rosalba Satta, Adrian Zhubi, et al.. (2007). Nicotinic cholinergic mechanisms in the regulation of brain DNA-methyltransferase 1 (DNMT1) expression. European Psychiatry. 22. S88–S88. 1 indexed citations

13.

Dimitrijević, Nikola, et al.. (2005). Drosophila GABAB receptors are involved in behavioral effects of γ-hydroxybutyric acid (GHB). European Journal of Pharmacology. 519(3). 246–252. 20 indexed citations

14.

Ruiu, Stefania, Giorgio Marchese, Rosalba Satta, et al.. (2003). In vitro evidence for the presence of [³H]‐haloperidol uptake in rat brain. British Journal of Pharmacology. 138(1). 188–192. 5 indexed citations

15.

Satta, Rosalba, Nikola Dimitrijević, & Hari Manev. (2003). Drosophila metabolize 1,4-butanediol into γ-hydroxybutyric acid in vivo. European Journal of Pharmacology. 473(2-3). 149–152. 17 indexed citations

16.

Veldić, Marin, Héctor J. Caruncho, John M. Davis, et al.. (2003). DNA-methyltransferase 1 mRNA is selectively overexpressed in telencephalic GABAergic interneurons of schizophrenia brains. Proceedings of the National Academy of Sciences. 101(1). 348–353. 226 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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