Rosa Sánchez‐Alvarez

994 total citations
19 papers, 797 citations indexed

About

Rosa Sánchez‐Alvarez is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Rosa Sánchez‐Alvarez has authored 19 papers receiving a total of 797 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 6 papers in Oncology and 6 papers in Cancer Research. Recurrent topics in Rosa Sánchez‐Alvarez's work include Connexins and lens biology (6 papers), Cancer, Hypoxia, and Metabolism (3 papers) and Cancer Cells and Metastasis (2 papers). Rosa Sánchez‐Alvarez is often cited by papers focused on Connexins and lens biology (6 papers), Cancer, Hypoxia, and Metabolism (3 papers) and Cancer Cells and Metastasis (2 papers). Rosa Sánchez‐Alvarez collaborates with scholars based in United Kingdom, Spain and United States. Rosa Sánchez‐Alvarez's co-authors include Federica Sotgia, Michael P. Lisanti, José M. Medina, Arantxa Tabernero, Marco Fiorillo, Ubaldo Martinez‐Outschoorn, Maria Peiris‐Pagès, Béla Ózsvári, Anna Rita Cappello and Arianna De Luca and has published in prestigious journals such as Nature Communications, PLoS ONE and Brain Research.

In The Last Decade

Rosa Sánchez‐Alvarez

19 papers receiving 788 citations

Peers

Rosa Sánchez‐Alvarez
Kaushik Datta United States
Jozef Hatok Slovakia
Soo‐Yeon Park South Korea
Hyung‐Chahn Lee South Korea
Namgyu Lee South Korea
Xinyan Li China
Kaushik Datta United States
Rosa Sánchez‐Alvarez
Citations per year, relative to Rosa Sánchez‐Alvarez Rosa Sánchez‐Alvarez (= 1×) peers Kaushik Datta

Countries citing papers authored by Rosa Sánchez‐Alvarez

Since Specialization
Citations

This map shows the geographic impact of Rosa Sánchez‐Alvarez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rosa Sánchez‐Alvarez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rosa Sánchez‐Alvarez more than expected).

Fields of papers citing papers by Rosa Sánchez‐Alvarez

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rosa Sánchez‐Alvarez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rosa Sánchez‐Alvarez. The network helps show where Rosa Sánchez‐Alvarez may publish in the future.

Co-authorship network of co-authors of Rosa Sánchez‐Alvarez

This figure shows the co-authorship network connecting the top 25 collaborators of Rosa Sánchez‐Alvarez. A scholar is included among the top collaborators of Rosa Sánchez‐Alvarez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rosa Sánchez‐Alvarez. Rosa Sánchez‐Alvarez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Sánchez‐Alvarez, Rosa, François Halloy, Jonathan Hall, et al.. (2025). miRNA ‐29 regulates epidermal and mesenchymal functions in skin repair. FEBS Letters. 599(12). 1795–1817. 2 indexed citations
2.
Sánchez‐Alvarez, Rosa, Tomasz M. Witkos, Ioannis Bantounas, et al.. (2023). Vimentin intermediate filaments provide structural stability to the mammalian Golgi complex. Journal of Cell Science. 136(20). 5 indexed citations
3.
Bruce, Jason I.E., Rosa Sánchez‐Alvarez, Maria Dolors Sans, et al.. (2021). Insulin protects acinar cells during pancreatitis by preserving glycolytic ATP supply to calcium pumps. Nature Communications. 12(1). 4386–4386. 34 indexed citations
4.
Sánchez‐Alvarez, Rosa, Ernestina Marianna De Francesco, Marco Fiorillo, Federica Sotgia, & Michael P. Lisanti. (2020). Mitochondrial Fission Factor (MFF) Inhibits Mitochondrial Metabolism and Reduces Breast Cancer Stem Cell (CSC) Activity. Frontiers in Oncology. 10. 1776–1776. 34 indexed citations
5.
Fiorillo, Marco, Maria Peiris‐Pagès, Rosa Sánchez‐Alvarez, et al.. (2018). Bergamot natural products eradicate cancer stem cells (CSCs) by targeting mevalonate, Rho-GDI-signalling and mitochondrial metabolism. Biochimica et Biophysica Acta (BBA) - Bioenergetics. 1859(9). 984–996. 53 indexed citations
6.
Fiorillo, Marco, Rosa Sánchez‐Alvarez, Federica Sotgia, & Michael P. Lisanti. (2018). The ER-alpha mutation Y537S confers Tamoxifen-resistance via enhanced mitochondrial metabolism, glycolysis and Rho-GDI/PTEN signaling: Implicating TIGAR in somatic resistance to endocrine therapy. Aging. 10(12). 4000–4023. 23 indexed citations
7.
Ózsvári, Béla, Gloria Bonuccelli, Rosa Sánchez‐Alvarez, et al.. (2017). Targeting flavin-containing enzymes eliminates cancer stem cells (CSCs), by inhibiting mitochondrial respiration: Vitamin B2 (Riboflavin) in cancer therapy. Aging. 9(12). 2610–2628. 46 indexed citations
8.
Luca, Arianna De, Marco Fiorillo, Maria Peiris‐Pagès, et al.. (2015). Mitochondrial biogenesis is required for the anchorage-independent survival and propagation of stem-like cancer cells. Oncotarget. 6(17). 14777–14795. 208 indexed citations
9.
Lisanti, Michael P., Maria Peiris‐Pagès, Amy L. Chadwick, et al.. (2013). JNK1 stress signaling is hyper-activated in high breast density and the tumor stroma: Connecting fibrosis, inflammation, and stemness for cancer prevention. Cell Cycle. 13(4). 580–599. 40 indexed citations
10.
Sánchez‐Alvarez, Rosa, Ubaldo Martinez‐Outschoorn, Lin Zhao, et al.. (2013). Ethanol exposure induces the cancer-associated fibroblast phenotype and lethal tumor metabolism. Cell Cycle. 12(2). 289–301. 45 indexed citations
11.
Sánchez‐Alvarez, Rosa, et al.. (2013). Ethanol Diverts Early Neuronal Differentiation Trajectory of Embryonic Stem Cells by Disrupting the Balance of Lineage Specifiers. PLoS ONE. 8(5). e63794–e63794. 30 indexed citations
12.
Sánchez‐Alvarez, Rosa, Ubaldo Martinez‐Outschoorn, Rebecca Lamb, et al.. (2012). Mitochondrial dysfunction in breast cancer cells prevents tumor growth. Cell Cycle. 12(1). 172–182. 74 indexed citations
13.
Herrero‐González, Sandra, José Carlos Valle‐Casuso, Rosa Sánchez‐Alvarez, et al.. (2008). Connexin43 is involved in the effect of endothelin‐1 on astrocyte proliferation and glucose uptake. Glia. 57(2). 222–233. 39 indexed citations
14.
Sánchez‐Alvarez, Rosa, Teresa Paíno, Sandra Herrero‐González, José M. Medina, & Arantxa Tabernero. (2006). Tolbutamide reduces glioma cell proliferation by increasing connexin43, which promotes the up‐regulation of p21 and p27 and subsequent changes in retinoblastoma phosphorylation. Glia. 54(2). 125–134. 32 indexed citations
15.
Tabernero, Arantxa, Rosa Sánchez‐Alvarez, & José M. Medina. (2006). Increased levels of cyclins D1 and D3 after inhibition of gap junctional communication in astrocytes. Journal of Neurochemistry. 96(4). 973–982. 26 indexed citations
16.
Sánchez‐Alvarez, Rosa, Arantxa Tabernero, & José M. Medina. (2005). The increase in gap junctional communication decreases the rate of glucose uptake in C6 glioma cells by releasing hexokinase from mitochondria. Brain Research. 1039(1-2). 189–198. 10 indexed citations
17.
Sánchez‐Alvarez, Rosa, Arantxa Tabernero, & José M. Medina. (2004). Endothelin‐1 stimulates the translocation and upregulation of both glucose transporter and hexokinase in astrocytes: relationship with gap junctional communication. Journal of Neurochemistry. 89(3). 703–714. 63 indexed citations
18.
Sánchez‐Alvarez, Rosa, Ángeles Almeida, & José M. Medina. (2002). Oxidative Stress in Preterm Rat Brain Is Due to Mitochondrial Dysfunction. Pediatric Research. 51(1). 34–39. 17 indexed citations
19.
Sánchez‐Alvarez, Rosa, et al.. (2001). Proliferation of C6 glioma cells is blunted by the increase in gap junction communication caused by tolbutamide. FEBS Letters. 509(2). 202–206. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026