Ronald C. Orlowski
- Molecular Biology top 10%
- Cellular and Molecular Neuroscience top 2%
- Oncology top 10%
- Physiology
- Endocrinology, Diabetes and Metabolism top 10%
- Co-authors
- Richard M. EpandRoderich WalterTadashi YoshimotoHiromichi NakamutaRaquel F. EpandMasao KoidaJay K. SeylerRobert J. Schlueter
- Topics
- Neuropeptides and Animal Physiology (29 papers)Receptor Mechanisms and Signaling (15 papers)Stress Responses and Cortisol (8 papers)
- Partner nations
- CanadaJapanUnited States
In The Last Decade
Ronald C. Orlowski
49 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 81
- Molecular Biology 761
- Cellular and Molecular Neuroscience 649
- Oncology 269
- Physiology 161
- Endocrinology, Diabetes and Metabolism 139
Countries citing papers authored by Ronald C. Orlowski
This map shows the geographic impact of Ronald C. Orlowski's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ronald C. Orlowski with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ronald C. Orlowski more than expected).
Fields of papers citing papers by Ronald C. Orlowski
This network shows the impact of papers produced by Ronald C. Orlowski. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ronald C. Orlowski. The network helps show where Ronald C. Orlowski may publish in the future.
Co-authorship network of co-authors of Ronald C. Orlowski
This figure shows the co-authorship network connecting the top 25 collaborators of Ronald C. Orlowski. A scholar is included among the top collaborators of Ronald C. Orlowski based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ronald C. Orlowski. Ronald C. Orlowski is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 6 | |
| 2 | 6 | |
| 3 | 37 | |
| 4 | 7 | |
| 5 | 6 | |
| 6 | 19 | |
| 7 | 6 | |
| 8 | 11 | |
| 9 | 30 | |
| 10 | 19 | |
| 11 | 10 | |
| 12 | 8 | |
| 13 | 39 | |
| 14 | 7 | |
| 15 | 28 | |
| 16 | 113 | |
| 17 | 4 | |
| 18 | 5 | |
| 19 | 2 | |
| 20 | 148 |
About Ronald C. Orlowski
Ronald C. Orlowski is a scholar working on Behavioral Neuroscience, Cellular and Molecular Neuroscience and Endocrine and Autonomic Systems, having authored 49 papers that have together received 1.2k indexed citations. Recurring topics across this work include Neuropeptides and Animal Physiology (29 papers), Receptor Mechanisms and Signaling (15 papers) and Stress Responses and Cortisol (8 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (649 citations), Behavioral Neuroscience (90 citations) and Endocrine and Autonomic Systems (79 citations). Ronald C. Orlowski has collaborated with scholars based in Canada, Japan and United States. Frequent co-authors include Richard M. Epand, Roderich Walter, Tadashi Yoshimoto, Hiromichi Nakamuta, Raquel F. Epand, Masao Koida, Jay K. Seyler, Robert J. Schlueter, Lawrence T. Boni and S. W. Hui. Their work appears in journals such as Journal of Biological Chemistry, Biochemistry and Brain Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.