Rolf J. Craven

11.2k total citations · 1 hit paper
58 papers, 4.0k citations indexed

About

Rolf J. Craven is a scholar working on Molecular Biology, Oncology and Immunology and Allergy. According to data from OpenAlex, Rolf J. Craven has authored 58 papers receiving a total of 4.0k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Molecular Biology, 19 papers in Oncology and 14 papers in Immunology and Allergy. Recurrent topics in Rolf J. Craven's work include Cell Adhesion Molecules Research (14 papers), HER2/EGFR in Cancer Research (9 papers) and DNA Repair Mechanisms (6 papers). Rolf J. Craven is often cited by papers focused on Cell Adhesion Molecules Research (14 papers), HER2/EGFR in Cancer Research (9 papers) and DNA Repair Mechanisms (6 papers). Rolf J. Craven collaborates with scholars based in United States, South Korea and Canada. Rolf J. Craven's co-authors include William G. Cance, Ikhlas Ahmed, Timothy M. Weiner, Edison T. Liu, Georgette A. Dent, Li Xu, Lewis V. Owens, Thomas D. Petes, Xihui Yang and Lori J. Kornberg and has published in prestigious journals such as The Lancet, Journal of Biological Chemistry and Molecular and Cellular Biology.

In The Last Decade

Rolf J. Craven

55 papers receiving 4.0k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Overexpression of the focal adhesion kinase (p125FAK) in invasive human tumors.

1995 541 citations Rolf J. Craven, Edison T. Liu et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Rolf J. Craven United States	35	2.4k	1.3k	955	930	656	58	4.0k
Peter Herrlich Germany	10	2.1k 0.9×	598 0.4×	1.0k 1.1×	875 0.9×	540 0.8×	12	3.7k
Kurt R. Auger United States	22	4.1k 1.7×	409 0.3×	1.0k 1.1×	1.3k 1.4×	405 0.6×	33	5.4k
Jean Paul ten Klooster Netherlands	23	2.7k 1.1×	783 0.6×	1.7k 1.7×	472 0.5×	294 0.4×	32	4.0k
Rosana Kapeller United States	26	4.1k 1.7×	378 0.3×	1.0k 1.1×	1.1k 1.2×	744 1.1×	41	5.9k
Yasuhisa Fukui Japan	35	3.4k 1.4×	366 0.3×	1.1k 1.2×	801 0.9×	310 0.5×	85	4.5k
Serge Roche France	37	3.1k 1.3×	373 0.3×	839 0.9×	1.0k 1.1×	371 0.6×	92	4.5k
Motoko Shibanuma Japan	31	2.2k 0.9×	536 0.4×	543 0.6×	367 0.4×	501 0.8×	67	3.3k
Elisa Bal de Kier Joffé Argentina	35	1.9k 0.8×	347 0.3×	389 0.4×	1.1k 1.2×	982 1.5×	123	3.5k
Jane McGlade Canada	23	5.0k 2.1×	552 0.4×	1.1k 1.1×	1.4k 1.5×	382 0.6×	26	6.6k
Christopher L. Carpenter United States	27	3.7k 1.5×	422 0.3×	1.5k 1.5×	583 0.6×	333 0.5×	55	5.3k

Countries citing papers authored by Rolf J. Craven

Since Specialization

Citations

This map shows the geographic impact of Rolf J. Craven's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rolf J. Craven with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rolf J. Craven more than expected).

Fields of papers citing papers by Rolf J. Craven

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rolf J. Craven. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rolf J. Craven. The network helps show where Rolf J. Craven may publish in the future.

Co-authorship network of co-authors of Rolf J. Craven

This figure shows the co-authorship network connecting the top 25 collaborators of Rolf J. Craven. A scholar is included among the top collaborators of Rolf J. Craven based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rolf J. Craven. Rolf J. Craven is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wei, Ying, Junfeng Shi, Kai Cheng, et al.. (2023). Targeting receptor tyrosine kinases in ovarian cancer: Genomic dysregulation, clinical evaluation of inhibitors, and potential for combinatorial therapies. Molecular Therapy — Oncolytics. 28. 293–306. 13 indexed citations

Craven, Rolf J., et al.. (2022). GAPDH in neuroblastoma: Functions in metabolism and survival. Frontiers in Oncology. 12. 979683–979683. 10 indexed citations

Craven, Rolf J., et al.. (2018). Insulin Receptor Levels Regulated by the Receptor- Associated Protein Progesterone Receptor Membrane Component 1 (PGRMC1). 4(2). 189–195. 1 indexed citations

Anderson, Katie L., et al.. (2018). Insulin Receptor Plasma Membrane Levels Increased by the Progesterone Receptor Membrane Component 1. Molecular Pharmacology. 94(1). 665–673. 32 indexed citations

Mir, Shakeel U.R., Steven R. Schwarze, Ling Jin, et al.. (2013). Progesterone receptor membrane component 1/Sigma-2 receptor associates with MAP1LC3B and promotes autophagy. Autophagy. 9(10). 1566–1578. 70 indexed citations

Mir, Shakeel U.R., Ling Jin, & Rolf J. Craven. (2012). Neutrophil Gelatinase-associated Lipocalin (NGAL) Expression Is Dependent on the Tumor-associated Sigma-2 Receptor S2RPgrmc1. Journal of Biological Chemistry. 287(18). 14494–14501. 34 indexed citations

Mir, Shakeel U.R., Ling Jin, & Rolf J. Craven. (2012). Abstract 3604: Elevated sigma-2 receptor/Pgrmc1 (progesterone receptor membrane component 1) levels as a potential cancer biomarker in tumors and plasma. Cancer Research. 72(8_Supplement). 3604–3604. 2 indexed citations

Ahmed, Ikhlas, et al.. (2012). S2R^Pgrmc1: the cytochrome-related sigma-2 receptor that regulates lipid and drug metabolism and hormone signaling. Expert Opinion on Drug Metabolism & Toxicology. 8(3). 361–370. 58 indexed citations

Mir, Shakeel U.R., Ikhlas Ahmed, Susanne M. Arnold, & Rolf J. Craven. (2011). Elevated progesterone receptor membrane component 1/sigma‐2 receptor levels in lung tumors and plasma from lung cancer patients. International Journal of Cancer. 131(2). E1–9. 85 indexed citations

10.

Ahmed, Ikhlas, et al.. (2010). Pgrmc1 (Progesterone Receptor Membrane Component 1) Associates with Epidermal Growth Factor Receptor and Regulates Erlotinib Sensitivity. Journal of Biological Chemistry. 285(32). 24775–24782. 115 indexed citations

11.

Yim, Eun-Kyoung, Guang Peng, Hui Dai, et al.. (2009). Rak Functions as a Tumor Suppressor by Regulating PTEN Protein Stability and Function. Cancer Cell. 15(4). 304–314. 152 indexed citations

12.

Dietrich, Charles S., Christopher P. DeSimone, Susan C. Modesitt, et al.. (2009). Suberoylanilide hydroxamic acid (SAHA) potentiates paclitaxel-induced apoptosis in ovarian cancer cell lines. Gynecologic Oncology. 116(1). 126–130. 35 indexed citations

13.

Ahmed, Ikhlas, et al.. (2008). PGRMC1 (progesterone receptor membrane component 1): A targetable protein with multiple functions in steroid signaling, P450 activation and drug binding. Pharmacology & Therapeutics. 121(1). 14–19. 165 indexed citations

14.

Ahmed, Ikhlas, et al.. (2007). Multiple pathways regulating the anti-apoptotic protein clusterin in breast cancer. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1772(9). 1103–1111. 21 indexed citations

15.

Ahmed, Ikhlas, et al.. (2006). Clusterin is induced by cancer chemotherapy and suppresses histone deacetylase inhibitor-induced apoptosis in breast cancer. Clinical Cancer Research. 12. 1 indexed citations

16.

Mallory, Julia C., et al.. (2005). A Novel Group of Genes Regulates Susceptibility to Antineoplastic Drugs in Highly Tumorigenic Breast Cancer Cells. Molecular Pharmacology. 68(6). 1747–1756. 51 indexed citations

17.

Lark, Amy L., Chad Livasy, Lynn G. Dressler, et al.. (2005). High focal adhesion kinase expression in invasive breast carcinomas is associated with an aggressive phenotype. Modern Pathology. 18(10). 1289–1294. 142 indexed citations

18.

Kurenova, Elena, Xihui Yang, Albert S. Baldwin, et al.. (2004). Focal Adhesion Kinase Suppresses Apoptosis by Binding to the Death Domain of Receptor-Interacting Protein. Molecular and Cellular Biology. 24(10). 4361–4371. 147 indexed citations

19.

Beviglia, Lucia, Vita Golubovskaya, Lihui Xu, et al.. (2003). Focal adhesion kinase N-terminus in breast carcinoma cells induces rounding, detachment and apoptosis. Biochemical Journal. 373(1). 201–210. 49 indexed citations

20.

Weiner, Timothy M., Edison T. Liu, Rolf J. Craven, & William G. Cance. (1994). Expression of growth factor receptors, the focal adhesion kinase, and other tyrosine kinases in human soft tissue tumors. Annals of Surgical Oncology. 1(1). 18–27. 79 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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