Robin van Schendel

2.0k total citations
29 papers, 1.4k citations indexed

About

Robin van Schendel is a scholar working on Molecular Biology, Plant Science and Aging. According to data from OpenAlex, Robin van Schendel has authored 29 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Molecular Biology, 9 papers in Plant Science and 5 papers in Aging. Recurrent topics in Robin van Schendel's work include CRISPR and Genetic Engineering (22 papers), DNA Repair Mechanisms (14 papers) and Chromosomal and Genetic Variations (6 papers). Robin van Schendel is often cited by papers focused on CRISPR and Genetic Engineering (22 papers), DNA Repair Mechanisms (14 papers) and Chromosomal and Genetic Variations (6 papers). Robin van Schendel collaborates with scholars based in Netherlands, United States and Sweden. Robin van Schendel's co-authors include Marcel Tijsterman, Joost Schimmel, Sophie Roerink, Bennie Lemmens, Hanneke Kool, Ron Romeijn, Wouter Koole, Kristy L. Okihara, Johan T. den Dunnen and Peter Zeller and has published in prestigious journals such as Nucleic Acids Research, Nature Communications and Nature Genetics.

In The Last Decade

Robin van Schendel

27 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robin van Schendel Netherlands 17 1.3k 309 190 167 143 29 1.4k
Madelaine Gogol United States 26 2.4k 1.9× 340 1.1× 214 1.1× 103 0.6× 57 0.4× 42 2.6k
С. Г. Георгиева Russia 21 1.7k 1.3× 359 1.2× 181 1.0× 129 0.8× 27 0.2× 128 1.8k
Michael J. Guertin United States 19 1.1k 0.9× 111 0.4× 141 0.7× 107 0.6× 83 0.6× 33 1.3k
Kirk T. Ehmsen United States 10 1.1k 0.9× 151 0.5× 183 1.0× 213 1.3× 32 0.2× 13 1.2k
Eric F. Joyce United States 26 1.9k 1.5× 689 2.2× 342 1.8× 62 0.4× 61 0.4× 52 2.2k
Yurii Sedkov United States 20 1.7k 1.3× 286 0.9× 285 1.5× 46 0.3× 55 0.4× 24 1.9k
Beth Elliott United States 9 962 0.8× 235 0.8× 190 1.0× 152 0.9× 35 0.2× 10 1.1k
Xin D. Gao United States 12 1.3k 1.0× 127 0.4× 275 1.4× 141 0.8× 39 0.3× 18 1.4k
Son C. Nguyen United States 20 1.2k 0.9× 336 1.1× 205 1.1× 70 0.4× 28 0.2× 36 1.3k

Countries citing papers authored by Robin van Schendel

Since Specialization
Citations

This map shows the geographic impact of Robin van Schendel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robin van Schendel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robin van Schendel more than expected).

Fields of papers citing papers by Robin van Schendel

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robin van Schendel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robin van Schendel. The network helps show where Robin van Schendel may publish in the future.

Co-authorship network of co-authors of Robin van Schendel

This figure shows the co-authorship network connecting the top 25 collaborators of Robin van Schendel. A scholar is included among the top collaborators of Robin van Schendel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robin van Schendel. Robin van Schendel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Schendel, Robin van, et al.. (2024). Double-strand breaks in facultative heterochromatin require specific movements and chromatin changes for efficient repair. Nature Communications. 15(1). 8984–8984. 2 indexed citations
2.
Schendel, Robin van, et al.. (2024). Genetic dissection of mutagenic repair and T-DNA capture at CRISPR-induced DNA breaks in Arabidopsis thaliana. PNAS Nexus. 3(3). pgae094–pgae094. 5 indexed citations
3.
Wang, Jing, Lars Nilsson, Anjali Pandey, et al.. (2023). Dissecting the genetic landscape of GPCR signaling through phenotypic profiling in C. elegans. Nature Communications. 14(1). 8410–8410. 13 indexed citations
4.
Schimmel, Joost, Hanneke Kool, Robin van Schendel, et al.. (2023). Modulating mutational outcomes and improving precise gene editing at CRISPR-Cas9-induced breaks by chemical inhibition of end-joining pathways. Cell Reports. 42(2). 112019–112019. 30 indexed citations
5.
Schendel, Robin van, Joost Schimmel, & Marcel Tijsterman. (2022). SIQ: easy quantitative measurement of mutation profiles in sequencing data. NAR Genomics and Bioinformatics. 4(3). lqac063–lqac063. 11 indexed citations
6.
Höijer, Ida, Anastasia Emmanouilidou, Robin van Schendel, et al.. (2022). CRISPR-Cas9 induces large structural variants at on-target and off-target sites in vivo that segregate across generations. Nature Communications. 13(1). 627–627. 113 indexed citations
7.
Pater, Sylvia de, et al.. (2022). Distinct mechanisms for genomic attachment of the 5′ and 3′ ends of Agrobacterium T-DNA in plants. Nature Plants. 8(5). 526–534. 18 indexed citations
8.
Lemmens, Bennie, et al.. (2021). Helicase Q promotes homology-driven DNA double-strand break repair and prevents tandem duplications. Nature Communications. 12(1). 7126–7126. 22 indexed citations
9.
Schimmel, Joost, et al.. (2021). Small tandem DNA duplications result from CST-guided Pol α-primase action at DNA break termini. Nature Communications. 12(1). 4843–4843. 26 indexed citations
10.
Schendel, Robin van, et al.. (2020). Translesion synthesis polymerases are dispensable for C. elegans reproduction but suppress genome scarring by polymerase theta-mediated end joining. PLoS Genetics. 16(4). e1008759–e1008759. 13 indexed citations
11.
Schendel, Robin van, et al.. (2020). BRCA1-associated structural variations are a consequence of polymerase theta-mediated end-joining. Nature Communications. 11(1). 3615–3615. 44 indexed citations
12.
Schimmel, Joost, Robin van Schendel, Johan T. den Dunnen, & Marcel Tijsterman. (2019). Templated Insertions: A Smoking Gun for Polymerase Theta-Mediated End Joining. Trends in Genetics. 35(9). 632–644. 98 indexed citations
13.
Pater, Sylvia de, et al.. (2016). T-DNA integration in plants results from polymerase-θ-mediated DNA repair. Nature Plants. 2(11). 16164–16164. 114 indexed citations
14.
Zeller, Peter, Jan Padeken, Robin van Schendel, et al.. (2016). Histone H3K9 methylation is dispensable for Caenorhabditis elegans development but suppresses RNA:DNA hybrid-associated repeat instability. Nature Genetics. 48(11). 1385–1395. 148 indexed citations
15.
Schendel, Robin van, et al.. (2016). Genomic Scars Generated by Polymerase Theta Reveal the Versatile Mechanism of Alternative End-Joining. PLoS Genetics. 12(10). e1006368–e1006368. 50 indexed citations
16.
Agostinho, Ana, Otto Manneberg, Robin van Schendel, et al.. (2016). High density of REC 8 constrains sister chromatid axes and prevents illegitimate synaptonemal complex formation. EMBO Reports. 17(6). 901–913. 28 indexed citations
17.
Lemmens, Bennie, Robin van Schendel, & Marcel Tijsterman. (2015). Mutagenic consequences of a single G-quadruplex demonstrate mitotic inheritance of DNA replication fork barriers. Nature Communications. 6(1). 8909–8909. 98 indexed citations
18.
Roerink, Sophie, Robin van Schendel, & Marcel Tijsterman. (2014). Polymerase theta-mediated end joining of replication-associated DNA breaks in C. elegans. Genome Research. 24(6). 954–962. 132 indexed citations
19.
Koole, Wouter, et al.. (2014). A Polymerase Theta-dependent repair pathway suppresses extensive genomic instability at endogenous G4 DNA sites. Nature Communications. 5(1). 3216–3216. 175 indexed citations
20.
Schendel, Robin van & Marcel Tijsterman. (2013). Microhomology-Mediated Intron Loss during Metazoan Evolution. Genome Biology and Evolution. 5(6). 1212–1219. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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