Robert P. A. Wallin

2.8k total citations
30 papers, 2.2k citations indexed

About

Robert P. A. Wallin is a scholar working on Immunology, Molecular Biology and Epidemiology. According to data from OpenAlex, Robert P. A. Wallin has authored 30 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Immunology, 8 papers in Molecular Biology and 7 papers in Epidemiology. Recurrent topics in Robert P. A. Wallin's work include Immunotherapy and Immune Responses (10 papers), Immune Cell Function and Interaction (9 papers) and T-cell and B-cell Immunology (6 papers). Robert P. A. Wallin is often cited by papers focused on Immunotherapy and Immune Responses (10 papers), Immune Cell Function and Interaction (9 papers) and T-cell and B-cell Immunology (6 papers). Robert P. A. Wallin collaborates with scholars based in Sweden, United States and Germany. Robert P. A. Wallin's co-authors include Hans‐Gustaf Ljunggren, Alf Grandien, Valentina Screpanti, Rolf Kiessling, Arne von Bonin, Andreas Lundqvist, Alan R. Prescott, Rossana Zaru, Michele West and Colin Watts and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Robert P. A. Wallin

29 papers receiving 2.2k citations

Peers

Robert P. A. Wallin
Robert P. A. Wallin
Citations per year, relative to Robert P. A. Wallin Robert P. A. Wallin (= 1×) peers Bénédicte Manoury

Countries citing papers authored by Robert P. A. Wallin

Since Specialization
Citations

This map shows the geographic impact of Robert P. A. Wallin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert P. A. Wallin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert P. A. Wallin more than expected).

Fields of papers citing papers by Robert P. A. Wallin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert P. A. Wallin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert P. A. Wallin. The network helps show where Robert P. A. Wallin may publish in the future.

Co-authorship network of co-authors of Robert P. A. Wallin

This figure shows the co-authorship network connecting the top 25 collaborators of Robert P. A. Wallin. A scholar is included among the top collaborators of Robert P. A. Wallin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert P. A. Wallin. Robert P. A. Wallin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sjöholm, Johan, et al.. (2025). Immunomodulation by extracorporeal ozone-based bactericide system in porcine Pseudomonas aeruginosa septic shock. Scientific Reports. 15(1). 20295–20295.
2.
Elder, Elizabeth, Catharina Johansson, Robert P. A. Wallin, et al.. (2023). Protective immunity induced by an inhaled SARS-CoV-2 subunit vaccine. Vaccine. 41(32). 4743–4751. 7 indexed citations
3.
Wallin, Robert P. A., et al.. (2022). Long-lasting T-cell response to SARS-CoV-2 antigens after vaccination—a prospective cohort study of HCWs working with COVID-19 patients. Infectious Diseases. 55(2). 142–148. 3 indexed citations
4.
Zirakzadeh, A. Ali, et al.. (2018). Detection of micro-metastases by flow cytometry in lymph nodes from patients with penile cancer. BMC Urology. 18(1). 86–86. 2 indexed citations
5.
Baptista, Marisa A. P., Márton Keszei, Mariana M.S. Oliveira, et al.. (2016). Deletion of Wiskott–Aldrich syndrome protein triggers Rac2 activity and increased cross-presentation by dendritic cells. Nature Communications. 7(1). 12175–12175. 27 indexed citations
6.
Weidner, Jessica M., Sachie Kanatani, Jonas M. Fuks, et al.. (2013). Rapid cytoskeleton remodelling in dendritic cells following invasion byToxoplasma gondiicoincides with the onset of a hypermigratory phenotype. Cellular Microbiology. 15(10). n/a–n/a. 62 indexed citations
7.
Fuks, Jonas M., Romanico B. G. Arrighi, Jessica M. Weidner, et al.. (2012). GABAergic Signaling Is Linked to a Hypermigratory Phenotype in Dendritic Cells Infected by Toxoplasma gondii. PLoS Pathogens. 8(12). e1003051–e1003051. 135 indexed citations
8.
Falet, Hervé, et al.. (2011). Gelsolin-Independent Podosome Formation in Dendritic Cells. PLoS ONE. 6(7). e21615–e21615. 8 indexed citations
9.
Johansson, Linda, et al.. (2010). M1 Protein-Dependent Intracellular Trafficking Promotes Persistence and Replication of <i>Streptococcus pyogenes</i> in Macrophages. Journal of Innate Immunity. 2(6). 534–545. 47 indexed citations
10.
Wallin, Robert P. A., et al.. (2010). Dendritic cell function at low physiological temperature. Journal of Leukocyte Biology. 88(4). 747–756. 7 indexed citations
11.
Watts, Colin, Rossana Zaru, Alan R. Prescott, Robert P. A. Wallin, & Michele West. (2006). Proximal effects of Toll-like receptor activation in dendritic cells. Current Opinion in Immunology. 19(1). 73–78. 51 indexed citations
12.
Screpanti, Valentina, Robert P. A. Wallin, Alf Grandien, & Hans‐Gustaf Ljunggren. (2004). Impact of FASL-induced apoptosis in the elimination of tumor cells by NK cells. Molecular Immunology. 42(4). 495–499. 131 indexed citations
13.
Westerberg, Lisa S., Robert P. A. Wallin, Gediminas Greicius, Hans‐Gustaf Ljunggren, & Eva Severinson. (2003). Efficient antigen presentation of soluble, but not particulate, antigen in the absence of Wiskott–Aldrich syndrome protein. Immunology. 109(3). 384–391. 33 indexed citations
14.
Wallin, Robert P. A., Valentina Screpanti, Jakob Michaëlsson, Alf Grandien, & Hans‐Gustaf Ljunggren. (2003). Regulation of perforin‐independent NK cell‐mediated cytotoxicity. European Journal of Immunology. 33(10). 2727–2735. 79 indexed citations
15.
Buentke, Eva, Annika Scheynius, Julia L. Wilson, et al.. (2002). Natural Killer and Dendritic Cell Contact in Lesional Atopic Dermatitis Skin –Malassezia-Influenced Cell Interaction. Journal of Investigative Dermatology. 119(4). 850–857. 105 indexed citations
16.
Wallin, Robert P. A., et al.. (2002). Heat-shock proteins as activators of the innate immune system. Trends in Immunology. 23(3). 130–135. 482 indexed citations
17.
Screpanti, Valentina, Robert P. A. Wallin, Hans‐Gustaf Ljunggren, & Alf Grandien. (2001). A Central Role for Death Receptor-Mediated Apoptosis in the Rejection of Tumors by NK Cells. The Journal of Immunology. 167(4). 2068–2073. 147 indexed citations
18.
Buentke, Eva, et al.. (2001). The allergenic yeast Malassezia furfur induces maturation of human dendritic cells. Clinical & Experimental Allergy. 31(10). 1583–1593. 60 indexed citations
19.
Sandberg, Johan K., Lars Franksson, Jonas Sundbäck, et al.. (2000). T Cell Tolerance Based on Avidity Thresholds Rather Than Complete Deletion Allows Maintenance of Maximal Repertoire Diversity. The Journal of Immunology. 165(1). 25–33. 63 indexed citations
20.
Johansson, Maria H., Robert P. A. Wallin, Johan K. Sandberg, et al.. (1998). MHC Class I Mosaic Mice Reveal Insights into Control of Ly49C Inhibitory Receptor Expression in NK Cells. The Journal of Immunology. 161(12). 6475–6479. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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