Robert G. Pergolizzi
- Molecular Biology top 10%
- Immunology top 5%
- Genetics top 5%
- Epidemiology top 10%
- Genetics top 5%
- Co-authors
- Ravi HingoraniPeter K. GregersenBeena Gulwani-AkolkarPradip N. AkolkarSusan ErsterRonald G. CrystalIn‐Hong ChoiMing-der Y. Chang
- Topics
- T-cell and B-cell Immunology (10 papers)Virus-based gene therapy research (6 papers)RNA and protein synthesis mechanisms (6 papers)
- Cited by
- ImmunologyGenetics
- Partner nations
- United StatesSwedenHungary
In The Last Decade
Robert G. Pergolizzi
60 papers receiving 2.3k citations
Peers
Comparison fields: 5 of 112
- Molecular Biology 761
- Immunology 717
- Genetics 613
- Epidemiology 316
- Genetics 223
Countries citing papers authored by Robert G. Pergolizzi
This map shows the geographic impact of Robert G. Pergolizzi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert G. Pergolizzi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert G. Pergolizzi more than expected).
Fields of papers citing papers by Robert G. Pergolizzi
This network shows the impact of papers produced by Robert G. Pergolizzi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert G. Pergolizzi. The network helps show where Robert G. Pergolizzi may publish in the future.
Co-authorship network of co-authors of Robert G. Pergolizzi
This figure shows the co-authorship network connecting the top 25 collaborators of Robert G. Pergolizzi. A scholar is included among the top collaborators of Robert G. Pergolizzi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert G. Pergolizzi. Robert G. Pergolizzi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 27 | |
| 3 | 40 | |
| 4 | 30 | |
| 5 | 148 | |
| 6 | 3 | |
| 7 | 12 | |
| 8 | 93 | |
| 9 | 6 | |
| 10 | 237 | |
| 11 | 11 | |
| 12 | 52 | |
| 13 | 2 | |
| 14 | 11 | |
| 15 | 2 | |
| 16 | 6 | |
| 17 | Analysis of a CGG sequence at the FMR-1 locus in fragile X families and in the general population. | 190 |
| 18 | 115 | |
| 19 | 74 | |
| 20 | 10 |
About Robert G. Pergolizzi
Robert G. Pergolizzi is a scholar working on Virology, Immunology and Genetics, having authored 61 papers that have together received 2.4k indexed citations. Recurring topics across this work include T-cell and B-cell Immunology (10 papers), Virus-based gene therapy research (6 papers) and RNA and protein synthesis mechanisms (6 papers). The work is most often cited by research in Immunology (717 citations), Genetics (613 citations) and Genetics (223 citations). Robert G. Pergolizzi has collaborated with scholars based in United States, Sweden and Hungary. Frequent co-authors include Ravi Hingorani, Peter K. Gregersen, Beena Gulwani-Akolkar, Pradip N. Akolkar, Susan Erster, Ronald G. Crystal, In‐Hong Choi, Ming-der Y. Chang, Houman Khalili and Rohini Deshpande. Their work appears in journals such as Proceedings of the National Academy of Sciences, The Lancet and Nucleic Acids Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.