Robert D. Cardiff

462 total citations
7 papers, 389 citations indexed

About

Robert D. Cardiff is a scholar working on Oncology, Genetics and Molecular Biology. According to data from OpenAlex, Robert D. Cardiff has authored 7 papers receiving a total of 389 indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Oncology, 3 papers in Genetics and 2 papers in Molecular Biology. Recurrent topics in Robert D. Cardiff's work include Protease and Inhibitor Mechanisms (2 papers), Animal Genetics and Reproduction (2 papers) and Proteoglycans and glycosaminoglycans research (2 papers). Robert D. Cardiff is often cited by papers focused on Protease and Inhibitor Mechanisms (2 papers), Animal Genetics and Reproduction (2 papers) and Proteoglycans and glycosaminoglycans research (2 papers). Robert D. Cardiff collaborates with scholars based in United States and Canada. Robert D. Cardiff's co-authors include William J. Muller, Lilly Bourguignon, Hongbo Zhu, Zeenat Gunja‐Smith, Naoko Iida, Eric Sinn, Philip Leder, Ursula K. Ehmann, Raphael C. Guzman and S. Nandi and has published in prestigious journals such as JNCI Journal of the National Cancer Institute, Journal of Cellular Physiology and Seminars in Cancer Biology.

In The Last Decade

Robert D. Cardiff

7 papers receiving 387 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Robert D. Cardiff United States	7	218	183	116	112	80	7	389
Douglas Boyd United States	8	198 0.9×	191 1.0×	209 1.8×	42 0.4×	80 1.0×	11	416
Mikael Herlevsen United States	8	301 1.4×	111 0.6×	67 0.6×	99 0.9×	68 0.8×	9	406
Jinli Chang United States	6	276 1.3×	156 0.9×	100 0.9×	127 1.1×	249 3.1×	8	504
Sylvie Maubant France	8	261 1.2×	103 0.6×	69 0.6×	78 0.7×	186 2.3×	20	425
R J Tressler United States	4	221 1.0×	146 0.8×	182 1.6×	25 0.2×	86 1.1×	7	350
Guido David Belgium	7	257 1.2×	67 0.4×	64 0.6×	267 2.4×	101 1.3×	10	429
Helena Antoniadis Switzerland	7	235 1.1×	128 0.7×	64 0.6×	44 0.4×	51 0.6×	8	350
Renard C. Walker United States	12	554 2.5×	157 0.9×	190 1.6×	57 0.5×	43 0.5×	14	694
Mahdhia Soula-Rothhut France	8	262 1.2×	98 0.5×	121 1.0×	35 0.3×	58 0.7×	9	406
Helicia Paz United States	10	308 1.4×	117 0.6×	79 0.7×	84 0.8×	40 0.5×	12	463

Countries citing papers authored by Robert D. Cardiff

Since Specialization

Citations

This map shows the geographic impact of Robert D. Cardiff's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert D. Cardiff with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert D. Cardiff more than expected).

Fields of papers citing papers by Robert D. Cardiff

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert D. Cardiff. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert D. Cardiff. The network helps show where Robert D. Cardiff may publish in the future.

Co-authorship network of co-authors of Robert D. Cardiff

This figure shows the co-authorship network connecting the top 25 collaborators of Robert D. Cardiff. A scholar is included among the top collaborators of Robert D. Cardiff based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert D. Cardiff. Robert D. Cardiff is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

7 of 7 papers shown

Hosokawa, Yoshitaka, Αλέξανδρος Παπανικολάου, Robert D. Cardiff, et al.. (2001). In vivo analysis of mammary and non-mammary tumorigenesis in MMTV-cyclin D1 transgenic mice deficient in p53. Transgenic Research. 10(5). 471–478. 12 indexed citations

Bourguignon, Lilly, Zeenat Gunja‐Smith, Naoko Iida, et al.. (1998). CD44v3,8-10 is involved in cytoskeleton-mediated tumor cell migration and matrix metalloproteinase (MMP-9) association in metastatic breast cancer cells. Journal of Cellular Physiology. 176(1). 206–215. 229 indexed citations

Bourguignon, Lilly, Zeenat Gunja‐Smith, Naoko Iida, et al.. (1998). CD44v3,810 is involved in cytoskeleton‐mediated tumor cell migration and matrix metalloproteinase (MMP‐9) association in metastatic breast cancer cells. Journal of Cellular Physiology. 176(1). 206–215. 6 indexed citations

Munn, Robert J., Marc Webster, William J. Muller, & Robert D. Cardiff. (1995). Histopathology of transgenic mouse mammary tumors (a short atlas). Seminars in Cancer Biology. 6(3). 153–158. 12 indexed citations

Cardiff, Robert D., Eric Sinn, William J. Muller, & Philip Leder. (1991). Transgenic oncogene mice. Tumor phenotype predicts genotype.. PubMed. 139(3). 495–501. 83 indexed citations

Cardiff, Robert D.. (1988). Cellular and molecular aspects of neoplastic progression in the mammary gland. European Journal of Cancer and Clinical Oncology. 24(1). 15–20. 23 indexed citations

Ehmann, Ursula K., et al.. (1987). Cultured Mouse Mammary Epithelial Cells: Normal Phenotype After Implantation<xref ref-type="fn" rid="FN2">2</xref>. JNCI Journal of the National Cancer Institute. 78(4). 751–7. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact