Guido David

511 total citations
10 papers, 429 citations indexed

About

Guido David is a scholar working on Cell Biology, Molecular Biology and Immunology and Allergy. According to data from OpenAlex, Guido David has authored 10 papers receiving a total of 429 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Cell Biology, 8 papers in Molecular Biology and 4 papers in Immunology and Allergy. Recurrent topics in Guido David's work include Proteoglycans and glycosaminoglycans research (8 papers), Glycosylation and Glycoproteins Research (4 papers) and Cell Adhesion Molecules Research (4 papers). Guido David is often cited by papers focused on Proteoglycans and glycosaminoglycans research (8 papers), Glycosylation and Glycoproteins Research (4 papers) and Cell Adhesion Molecules Research (4 papers). Guido David collaborates with scholars based in Belgium, United States and Netherlands. Guido David's co-authors include Robbert van der Voort, Lia Smit, Steven T. Pals, Remko Prevo, Marcel Spaargaren, Guido R. Hartmann, Taher E.I. Taher, Ermanno Gherardi, K. Nackaerts and Georges Deneffe and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Molecular Biology and Biochemical Journal.

In The Last Decade

Guido David

9 papers receiving 424 citations

Peers

Guido David

CB

MB

IA

HEPAT

ONCOL

Evgeny Edovitsky Israel

Feodor Berdichevsky United Kingdom

Tomomi Hirosaki Japan

Julien Deheuninck France

Ilanit Boyango Israel

Yongsheng Ren United States

R. Arch Germany

Péter Hollósi Hungary

Helicia Paz United States

Karin G. Wirth Austria

Evgeny Edovitsky Israel

Guido David

591 ×2.2

CB

569 ×2.2

MB

55 ×0.5

IA

22 ×0.3

HEPAT

80 ×1.2

ONCOL

Citations per year, relative to Guido David Guido David (= 1×) peers Evgeny Edovitsky

Countries citing papers authored by Guido David

Since Specialization

Citations

This map shows the geographic impact of Guido David's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Guido David with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Guido David more than expected).

Fields of papers citing papers by Guido David

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Guido David. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Guido David. The network helps show where Guido David may publish in the future.

Co-authorship network of co-authors of Guido David

This figure shows the co-authorship network connecting the top 25 collaborators of Guido David. A scholar is included among the top collaborators of Guido David based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Guido David. Guido David is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Leblanc, Raphaël, Stéphane Audebert, Kim De Veirman, et al.. (2025). Syntenin inhibition impairs stroma‐tumor communication in multiple myeloma and improves bortezomib treatment efficiency. HemaSphere. 9(8). e70197–e70197.

2.

Leblanc, Raphaël, Rania Ghossoub, Armelle Goubard, et al.. (2023). Downregulation of stromal syntenin sustains AML development. EMBO Molecular Medicine. 15(11). e17570–e17570. 6 indexed citations

3.

Zimmermann, Pascale, Zhe Zhang, Gisèle Degeest, et al.. (2005). Syndecan Recycling Is Controlled by Syntenin-PIP2 Interaction and Arf6. Developmental Cell. 9(5). 721–721. 5 indexed citations

4.

Voort, Robbert van der, Taher E.I. Taher, Marcel Spaargaren, et al.. (1999). Heparan Sulfate-modified CD44 Promotes Hepatocyte Growth Factor/Scatter Factor-induced Signal Transduction through the Receptor Tyrosine Kinase c-Met. Journal of Biological Chemistry. 274(10). 6499–6506. 192 indexed citations

5.

Göhring, Walter, et al.. (1999). Structural basis of glycosaminoglycan modification and of heterotypic interactions of perlecan domain V 1 1Edited by I. B. Holland. Journal of Molecular Biology. 294(1). 259–270. 66 indexed citations

6.

Schofield, K. P., John T. Gallagher, & Guido David. (1999). Expression of proteoglycan core proteins in human bone marrow stroma. Biochemical Journal. 343(3). 663–663. 12 indexed citations

7.

Nackaerts, K., et al.. (1997). Heparan sulfate proteoglycan expression in human lung-cancer cells. International Journal of Cancer. 74(3). 335–345. 102 indexed citations

8.

Nackaerts, K., et al.. (1997). Heparan sulfate proteoglycan expression in human lung‐cancer cells. International Journal of Cancer. 74(3). 335–345. 6 indexed citations

9.

Oguri, Kayoko, et al.. (1996). Phosphorylation of a membrane-intercalated proteoglycan, syndecan-2, expressed in a stroma-inducing clone from a mouse Lewis lung carcinoma. Biochemical Journal. 315(3). 925–930. 27 indexed citations

10.

David, Guido & Herman Van den Berghe. (1989). Cell‐surface heparan sulfate and heparan‐sulfate/chondroitin‐sulfate hybrid proteoglycans of mouse mammary epithelial cells. European Journal of Biochemistry. 178(3). 609–617. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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