Ritsuko Iwanaga

1.8k total citations
37 papers, 1.4k citations indexed

About

Ritsuko Iwanaga is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Ritsuko Iwanaga has authored 37 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 23 papers in Oncology and 10 papers in Immunology. Recurrent topics in Ritsuko Iwanaga's work include Cancer-related Molecular Pathways (15 papers), Epigenetics and DNA Methylation (8 papers) and T-cell and Retrovirus Studies (7 papers). Ritsuko Iwanaga is often cited by papers focused on Cancer-related Molecular Pathways (15 papers), Epigenetics and DNA Methylation (8 papers) and T-cell and Retrovirus Studies (7 papers). Ritsuko Iwanaga collaborates with scholars based in United States, Japan and United Kingdom. Ritsuko Iwanaga's co-authors include Kiyoshi Ohtani, Heide L. Ford, Masataka Nakamura, Douglas S. Micalizzi, Hideyuki Komori, Rebecca L. Vartuli, David Drasin, Aik Choon Tan, Ashley Smith and Paul Jedlicka and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and The EMBO Journal.

In The Last Decade

Ritsuko Iwanaga

36 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ritsuko Iwanaga United States 17 907 528 405 278 183 37 1.4k
Alex C. Minella United States 20 929 1.0× 636 1.2× 244 0.6× 255 0.9× 68 0.4× 35 1.4k
Alexandra Bell United Kingdom 10 704 0.8× 689 1.3× 218 0.5× 100 0.4× 144 0.8× 13 1.3k
Massimo Zani Italy 17 731 0.8× 180 0.3× 182 0.4× 154 0.6× 153 0.8× 31 1.1k
Karen M. Watters Ireland 18 695 0.8× 189 0.4× 443 1.1× 124 0.4× 29 0.2× 22 1.0k
Susanne Saurer Switzerland 13 808 0.9× 791 1.5× 236 0.6× 201 0.7× 72 0.4× 17 1.5k
Ester Alvino Italy 17 500 0.6× 189 0.4× 280 0.7× 106 0.4× 98 0.5× 35 813
Ignacio Pérez‐Roger Spain 16 900 1.0× 458 0.9× 252 0.6× 126 0.5× 101 0.6× 31 1.3k
Malka Popliker Israel 12 485 0.5× 348 0.7× 139 0.3× 179 0.6× 36 0.2× 18 1.2k
A. Gutman France 8 899 1.0× 353 0.7× 429 1.1× 165 0.6× 30 0.2× 9 1.3k

Countries citing papers authored by Ritsuko Iwanaga

Since Specialization
Citations

This map shows the geographic impact of Ritsuko Iwanaga's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ritsuko Iwanaga with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ritsuko Iwanaga more than expected).

Fields of papers citing papers by Ritsuko Iwanaga

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ritsuko Iwanaga. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ritsuko Iwanaga. The network helps show where Ritsuko Iwanaga may publish in the future.

Co-authorship network of co-authors of Ritsuko Iwanaga

This figure shows the co-authorship network connecting the top 25 collaborators of Ritsuko Iwanaga. A scholar is included among the top collaborators of Ritsuko Iwanaga based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ritsuko Iwanaga. Ritsuko Iwanaga is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Iwanaga, Ritsuko, et al.. (2025). Transcriptional Activation Mechanisms and Target Genes of the Oncogene Product Tax of Human T-Cell Leukemia Virus Type 1. Genes. 16(10). 1221–1221. 1 indexed citations
2.
Iwanaga, Ritsuko, Tomomi M. Yamamoto, Lily L. Nguyen, et al.. (2024). Tumor-Intrinsic Activity of Chromobox 2 Remodels the Tumor Microenvironment in High-grade Serous Carcinoma. Cancer Research Communications. 4(8). 1919–1932. 1 indexed citations
3.
Nguyen, Lily L., Zachary L. Watson, Raquel Ortega, et al.. (2024). Combining EHMT and PARP Inhibition: A Strategy to Diminish Therapy-Resistant Ovarian Cancer Tumor Growth while Stimulating Immune Activation. Molecular Cancer Therapeutics. 23(9). 1332–1347. 3 indexed citations
4.
Nguyen, Lily L., Zachary L. Watson, Raquel Ortega, et al.. (2024). EHMT1/2 Inhibition Promotes Regression of Therapy-Resistant Ovarian Cancer Tumors in a CD8 T-cell–Dependent Manner. Molecular Cancer Research. 22(12). 1117–1127.
5.
Kamiya, Yuki, Ritsuko Iwanaga, Andrew P. Bradford, et al.. (2024). DEAD/H Box 5 (DDX5) Augments E2F1-Induced Cell Death Independent of the Tumor Suppressor p53. International Journal of Molecular Sciences. 25(24). 13251–13251. 1 indexed citations
8.
Iwanaga, Ritsuko, et al.. (2023). Deregulated E2F Activity as a Cancer-Cell Specific Therapeutic Tool. Genes. 14(2). 393–393. 12 indexed citations
9.
Iwanaga, Ritsuko, et al.. (2023). Expanding Roles of the E2F-RB-p53 Pathway in Tumor Suppression. Biology. 12(12). 1511–1511. 17 indexed citations
10.
Komori, Hideyuki, et al.. (2023). The TFDP1 gene coding for DP1, the heterodimeric partner of the transcription factor E2F, is a target of deregulated E2F. Biochemical and Biophysical Research Communications. 663. 154–162. 9 indexed citations
11.
Komori, Hideyuki, Eiko Ozono, Ritsuko Iwanaga, et al.. (2018). Differential requirement for dimerization partner DP between E2F-dependent activation of tumor suppressor and growth-related genes. Scientific Reports. 8(1). 8438–8438. 12 indexed citations
12.
Wang, Chu-An, J. Chuck Harrell, Ritsuko Iwanaga, Paul Jedlicka, & Heide L. Ford. (2014). Vascular endothelial growth factor C promotes breast cancer progression via a novel antioxidant mechanism that involves regulation of superoxide dismutase 3. Breast Cancer Research. 16(5). 462–462. 44 indexed citations
13.
Smith, Ashley, Ritsuko Iwanaga, David Drasin, et al.. (2012). The miR-106b-25 cluster targets Smad7, activates TGF-β signaling, and induces EMT and tumor initiating cell characteristics downstream of Six1 in human breast cancer. Oncogene. 31(50). 5162–5171. 255 indexed citations
14.
McCoy, Erica, Ritsuko Iwanaga, Paul Jedlicka, et al.. (2009). Six1 expands the mouse mammary epithelial stem/progenitor cell pool and induces mammary tumors that undergo epithelial-mesenchymal transition. Journal of Clinical Investigation. 119(9). 2663–2677. 137 indexed citations
15.
Iwanaga, Ritsuko, Eiko Ozono, Jun–ichi Fujisawa, et al.. (2008). Activation of the cyclin D2 and cdk6 genes through NF-κB is critical for cell-cycle progression induced by HTLV-I Tax. Oncogene. 27(42). 5635–5642. 56 indexed citations
16.
Iwanaga, Ritsuko, Hideyuki Komori, Susumu Ishida, et al.. (2005). Identification of novel E2F1 target genes regulated in cell cycle-dependent and independent manners. Oncogene. 25(12). 1786–1798. 55 indexed citations
17.
Ohtani, Kiyoshi, et al.. (2000). Cell Type-specific E2F Activation and Cell Cycle Progression Induced by the Oncogene Product Tax of Human T-cell Leukemia Virus Type I. Journal of Biological Chemistry. 275(15). 11154–11163. 66 indexed citations
18.
Ohtani, Kiyoshi, Ritsuko Iwanaga, Masataka Nakamura, et al.. (1999). Cell growth-regulated expression of mammalian MCM5 and MCM6 genes mediated by the transcription factor E2F. Oncogene. 18(14). 2299–2309. 115 indexed citations
19.
Akiyama, Yoshimitsu, Ritsuko Iwanaga, Tomoko Ishikawa, et al.. (1996). Mutations of the transforming growth factor-β type II receptor gene are strongly related to sporadic proximal colon carcinomas with microsatellite instability. Cancer. 78(12). 2478–2484. 47 indexed citations
20.
Sasaki, Atsushi, Makoto Nagashima, Masayuki Shiseki, et al.. (1996). Microsatellite instability in gastric cancer prone families. Cancer Letters. 99(2). 169–175. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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