Richard M. Schultz

1.5k total citations
11 papers, 1.3k citations indexed

About

Richard M. Schultz is a scholar working on Molecular Biology, Public Health, Environmental and Occupational Health and Reproductive Medicine. According to data from OpenAlex, Richard M. Schultz has authored 11 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 7 papers in Public Health, Environmental and Occupational Health and 3 papers in Reproductive Medicine. Recurrent topics in Richard M. Schultz's work include Reproductive Biology and Fertility (7 papers), Pluripotent Stem Cells Research (3 papers) and Ovarian function and disorders (2 papers). Richard M. Schultz is often cited by papers focused on Reproductive Biology and Fertility (7 papers), Pluripotent Stem Cells Research (3 papers) and Ovarian function and disorders (2 papers). Richard M. Schultz collaborates with scholars based in United States and Japan. Richard M. Schultz's co-authors include Diane M. Worrad, John J. Eppig, Franck Chesnel, Marilyn J. O’Brien, Fugaku Aoki, Paula Stein, George L. Gerton, Laura Dı́az-Cueto, Chwen-Ming Shih and Jo Ann Dempsey and has published in prestigious journals such as Developmental Biology, Biochemical Pharmacology and Journal of Experimental Zoology.

In The Last Decade

Richard M. Schultz

11 papers receiving 1.2k citations

Peers

Richard M. Schultz
M Antczak United States
Mark B. Harvey Australia
Akiko Spindle United States
J. A. G. Davids Netherlands
Hana Mlčochová United Kingdom
M Antczak United States
Richard M. Schultz
Citations per year, relative to Richard M. Schultz Richard M. Schultz (= 1×) peers M Antczak

Countries citing papers authored by Richard M. Schultz

Since Specialization
Citations

This map shows the geographic impact of Richard M. Schultz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Richard M. Schultz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Richard M. Schultz more than expected).

Fields of papers citing papers by Richard M. Schultz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Richard M. Schultz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Richard M. Schultz. The network helps show where Richard M. Schultz may publish in the future.

Co-authorship network of co-authors of Richard M. Schultz

This figure shows the co-authorship network connecting the top 25 collaborators of Richard M. Schultz. A scholar is included among the top collaborators of Richard M. Schultz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Richard M. Schultz. Richard M. Schultz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Schultz, Richard M. & Jerome A. Dempsey. (2002). Sequence dependence of Alimta (LY231514, MTA) combined with doxorubicin in ZR-75-1 human breast carcinoma cells.. PubMed. 21(5). 3209–14. 5 indexed citations
2.
Dı́az-Cueto, Laura, et al.. (2000). Modulation of Mouse Preimplantation Embryo Development by Acrogranin (Epithelin/Granulin Precursor). Developmental Biology. 217(2). 406–418. 95 indexed citations
3.
Aoki, Fugaku & Richard M. Schultz. (1999). DNA replication in the 1-cell mouse embryo: stimulatory effect of histone acetylation. Zygote. 7(2). 165–172. 71 indexed citations
4.
Schultz, Richard M., et al.. (1999). Biological activity of the multitargeted antifolate, MTA (LY231514), in human cell lines with different resistance mechanisms to antifolate drugs.. PubMed. 26(2 Suppl 6). 68–73. 79 indexed citations
5.
Worrad, Diane M., et al.. (1997). Regulation of Transcriptional Activity during the First and Second Cell Cycles in the Preimplantation Mouse Embryo. Developmental Biology. 181(2). 296–307. 483 indexed citations
6.
Xu, Zhe, et al.. (1996). Effects of Calcium-BAPTA Buffers and the Calmodulin Antagonist W-7 on Mouse Egg Activation. Developmental Biology. 180(2). 594–604. 42 indexed citations
7.
Schultz, Richard M.. (1995). Newer antifolates in cancer therapy. Birkhäuser Basel eBooks. 44. 129–157. 8 indexed citations
8.
Schmitz, John C., et al.. (1994). Impact of dietary folic acid on reduced folates in mouse plasma and tissues. Biochemical Pharmacology. 48(2). 319–325. 43 indexed citations
9.
Eppig, John J., Richard M. Schultz, Marilyn J. O’Brien, & Franck Chesnel. (1994). Relationship between the Developmental Programs Controlling Nuclear and Cytoplasmic Maturation of Mouse Oocytes. Developmental Biology. 164(1). 1–9. 270 indexed citations
10.
Kaji, Eugene, Elayne A. Bornslaeger, & Richard M. Schultz. (1987). Inhibition of mouse oocyte cyclic AMP phosphodiesterase by steroid hormones: A possible mechanism for steroid hormone inhibition of oocyte maturation. Journal of Experimental Zoology. 243(3). 489–493. 20 indexed citations
11.
Schultz, Richard M., et al.. (1981). Biochemical studies of mammalian oogenesis: Metabolic cooperativity between granulosa cells and growing mouse oocytes. Developmental Biology. 84(2). 455–464. 141 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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