Richard C. Witt
- Co-authors
- Thomas E. AdrianWeigang TongGeorge J. RozanskiXianzhong DingRené HennigJens StandopParviz M. PourMatthew J. Strouch
- Topics
- Ion channel regulation and function (4 papers)Cardiac electrophysiology and arrhythmias (4 papers)Inflammatory mediators and NSAID effects (3 papers)
- Cited by
- PharmacologyBiochemistryToxicology
- Partner nations
- United StatesUnited Arab EmiratesRussia
In The Last Decade
Richard C. Witt
15 papers receiving 493 citations
Peers
Comparison fields: 5 of 75
- Molecular Biology 221
- Pharmacology 185
- Cancer Research 81
- Oncology 77
- Immunology 65
Countries citing papers authored by Richard C. Witt
This map shows the geographic impact of Richard C. Witt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Richard C. Witt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Richard C. Witt more than expected).
Fields of papers citing papers by Richard C. Witt
This network shows the impact of papers produced by Richard C. Witt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Richard C. Witt. The network helps show where Richard C. Witt may publish in the future.
Co-authorship network of co-authors of Richard C. Witt
This figure shows the co-authorship network connecting the top 25 collaborators of Richard C. Witt. A scholar is included among the top collaborators of Richard C. Witt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Richard C. Witt. Richard C. Witt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 126 | |
| 2 | 30 | |
| 3 | 6 | |
| 4 | 6 | |
| 5 | Leukotriene B4 receptor antagonist LY293111 inhibits proliferation and induces apoptosis in human pancreatic cancer cells. | 96 |
| 6 | Lipoxygenase inhibitors attenuate growth of human pancreatic cancer xenografts and induce apoptosis through the mitochondrial pathway. | 131 |
| 7 | A Preliminary Catalog of the Lichen-Forming Fungi of Nebraska | 1 |
| 8 | 9 | |
| 9 | 40 | |
| 10 | 4 | |
| 11 | 12 | |
| 12 | 1 | |
| 13 | 7 | |
| 14 | 29 | |
| 15 | 6 | |
| 16 | [Electrophysiological aspects of the effect of creatine phosphate on myocardial cellular activity in the normal state and in ischemia]. | 2 |
About Richard C. Witt
Richard C. Witt is a scholar working on Toxicology, Cardiology and Cardiovascular Medicine and Virology, having authored 16 papers that have together received 506 indexed citations. Recurring topics across this work include Ion channel regulation and function (4 papers), Cardiac electrophysiology and arrhythmias (4 papers) and Inflammatory mediators and NSAID effects (3 papers). The work is most often cited by research in Pharmacology (185 citations), Biochemistry (57 citations) and Toxicology (24 citations). Richard C. Witt has collaborated with scholars based in United States, United Arab Emirates and Russia. Frequent co-authors include Thomas E. Adrian, Weigang Tong, George J. Rozanski, Xianzhong Ding, René Hennig, Jens Standop, Parviz M. Pour, Xianzhong Ding, Matthew J. Strouch and Laleh G. Melstrom. Their work appears in journals such as Circulation, Cancer and Clinical Cancer Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.