Revital Levy

582 total citations
13 papers, 447 citations indexed

About

Revital Levy is a scholar working on Immunology, Molecular Biology and Cancer Research. According to data from OpenAlex, Revital Levy has authored 13 papers receiving a total of 447 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Immunology, 1 paper in Molecular Biology and 1 paper in Cancer Research. Recurrent topics in Revital Levy's work include Immune Response and Inflammation (12 papers), Immunotherapy and Immune Responses (10 papers) and T-cell and B-cell Immunology (9 papers). Revital Levy is often cited by papers focused on Immune Response and Inflammation (12 papers), Immunotherapy and Immune Responses (10 papers) and T-cell and B-cell Immunology (9 papers). Revital Levy collaborates with scholars based in Israel and Italy. Revital Levy's co-authors include Raymond Kaempfer, Gila Arad, Dalia Hillman, Uri Barash, Tomer Shpilka, Adi Minis, Mark Katzenellenbogen, Shimon Slavin, Israel Pecht and Maria Teresa Fiorillo and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Medicine and PLoS Biology.

In The Last Decade

Revital Levy

13 papers receiving 426 citations

Peers

Revital Levy
Saskia Boisot United States
Shibali Das United States
Angeline Tilly Dang United States
Melissa A. Gessner United States
E. M. Ayoub United States
U. Shu Canada
Revital Levy
Citations per year, relative to Revital Levy Revital Levy (= 1×) peers Dalia Hillman

Countries citing papers authored by Revital Levy

Since Specialization
Citations

This map shows the geographic impact of Revital Levy's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Revital Levy with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Revital Levy more than expected).

Fields of papers citing papers by Revital Levy

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Revital Levy. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Revital Levy. The network helps show where Revital Levy may publish in the future.

Co-authorship network of co-authors of Revital Levy

This figure shows the co-authorship network connecting the top 25 collaborators of Revital Levy. A scholar is included among the top collaborators of Revital Levy based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Revital Levy. Revital Levy is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Levy, Michal, Dalia Hillman, Revital Levy, et al.. (2023). The homodimer interfaces of costimulatory receptors B7 and CD28 control their engagement and pro-inflammatory signaling. Journal of Biomedical Science. 30(1). 49–49. 3 indexed citations
2.
Kunkl, Martina, Silvana Caristi, Valentina Tedeschi, et al.. (2021). Binding of Staphylococcal Enterotoxin B (SEB) to B7 Receptors Triggers TCR- and CD28-Mediated Inflammatory Signals in the Absence of MHC Class II Molecules. Frontiers in Immunology. 12. 723689–723689. 15 indexed citations
4.
Levy, Revital, et al.. (2016). Superantigens hyperinduce inflammatory cytokines by enhancing the B7-2/CD28 costimulatory receptor interaction. Proceedings of the National Academy of Sciences. 113(42). E6437–E6446. 45 indexed citations
5.
Kaempfer, Raymond, et al.. (2013). CD28: Direct and Critical Receptor for Superantigen Toxins. Toxins. 5(9). 1531–1542. 29 indexed citations
6.
Arad, Gila, Revital Levy, Dalia Hillman, et al.. (2011). Binding of Superantigen Toxins into the CD28 Homodimer Interface Is Essential for Induction of Cytokine Genes That Mediate Lethal Shock. PLoS Biology. 9(9). e1001149–e1001149. 104 indexed citations
7.
Arad, Gila, Revital Levy, Dalia Hillman, et al.. (2011). PS1-048 Binding of superantigen toxins into the CD28 homodimer interface is essential for induction of Th1 cytokine genes that mediate lethal shock. Cytokine. 56(1). 29–29. 1 indexed citations
8.
Arad, Gila, Dalia Hillman, Revital Levy, & Raymond Kaempfer. (2003). Broad-spectrum immunity against superantigens is elicited in mice protected from lethal shock by a superantigen antagonist peptide. Immunology Letters. 91(2-3). 141–145. 26 indexed citations
9.
Arad, Gila, Revital Levy, & Raymond Kaempfer. (2002). Superantigen concomitantly induces Th1 cytokine genes and the ability to shut off their expression on re-exposure to superantigen. Immunology Letters. 82(1-2). 75–78. 16 indexed citations
10.
Kaempfer, Raymond, Gila Arad, Revital Levy, & Dalia Hillman. (2002). Defense against biologic warfare with superantigen toxins.. PubMed. 4(7). 520–3. 18 indexed citations
11.
Arad, Gila, Dalia Hillman, Revital Levy, & Raymond Kaempfer. (2001). Superantigen antagonist blocks Th1 cytokine gene induction and lethal shock. Journal of Leukocyte Biology. 69(6). 921–927. 36 indexed citations
12.
Arad, Gila, Revital Levy, Dalia Hillman, & Raymond Kaempfer. (2000). Superantigen antagonist protects against lethal shock and defines a new domain for T-cell activation. Nature Medicine. 6(4). 414–421. 126 indexed citations
13.
Arad, Gila, Mark Katzenellenbogen, Revital Levy, et al.. (1996). Linomide, an immunomodulator that inhibits Th1 cytokine gene expression. International Immunology. 8(10). 1603–1607. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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