Ranganath Mamidi

896 total citations
29 papers, 749 citations indexed

About

Ranganath Mamidi is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cell Biology. According to data from OpenAlex, Ranganath Mamidi has authored 29 papers receiving a total of 749 indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Cardiology and Cardiovascular Medicine, 8 papers in Molecular Biology and 3 papers in Cell Biology. Recurrent topics in Ranganath Mamidi's work include Cardiomyopathy and Myosin Studies (29 papers), Cardiovascular Effects of Exercise (18 papers) and Muscle Physiology and Disorders (8 papers). Ranganath Mamidi is often cited by papers focused on Cardiomyopathy and Myosin Studies (29 papers), Cardiovascular Effects of Exercise (18 papers) and Muscle Physiology and Disorders (8 papers). Ranganath Mamidi collaborates with scholars based in United States, Australia and Germany. Ranganath Mamidi's co-authors include Julian E. Stelzer, Murali Chandra, Kenneth S. Gresham, Jiayang Li, Steven J. Ford, Sujeet Verma, Cristobal G. dos Remedios, Amy Li, Siegfried Labeit and Sampath K. Gollapudi and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Molecular Biology and Circulation Research.

In The Last Decade

Ranganath Mamidi

29 papers receiving 744 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ranganath Mamidi United States 16 680 386 72 36 30 29 749
Chandra Saripalli United States 12 596 0.9× 337 0.9× 61 0.8× 45 1.3× 14 0.5× 15 704
Paul J.M. Wijnker Netherlands 16 548 0.8× 398 1.0× 26 0.4× 44 1.2× 21 0.7× 22 706
Kimberly A. Palmiter United States 11 670 1.0× 438 1.1× 69 1.0× 18 0.5× 17 0.6× 13 722
Marion von Frieling-Salewsky Germany 10 442 0.7× 271 0.7× 51 0.7× 57 1.6× 8 0.3× 11 567
Adam Jacques United Kingdom 10 671 1.0× 412 1.1× 33 0.5× 27 0.8× 7 0.2× 26 732
Anna Gaertner Germany 14 443 0.7× 244 0.6× 131 1.8× 21 0.6× 7 0.2× 21 595
Faris Albayya United States 15 635 0.9× 466 1.2× 33 0.5× 20 0.6× 5 0.2× 25 801
Pierre Coutu United States 13 640 0.9× 431 1.1× 49 0.7× 19 0.5× 9 0.3× 15 782
Cheavar A. Blair United States 10 236 0.3× 141 0.4× 22 0.3× 43 1.2× 13 0.4× 17 322
Yuping Xie China 8 367 0.5× 294 0.8× 43 0.6× 38 1.1× 12 0.4× 14 517

Countries citing papers authored by Ranganath Mamidi

Since Specialization
Citations

This map shows the geographic impact of Ranganath Mamidi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ranganath Mamidi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ranganath Mamidi more than expected).

Fields of papers citing papers by Ranganath Mamidi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ranganath Mamidi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ranganath Mamidi. The network helps show where Ranganath Mamidi may publish in the future.

Co-authorship network of co-authors of Ranganath Mamidi

This figure shows the co-authorship network connecting the top 25 collaborators of Ranganath Mamidi. A scholar is included among the top collaborators of Ranganath Mamidi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ranganath Mamidi. Ranganath Mamidi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Mamidi, Ranganath, et al.. (2022). Molecular characterization of linker and loop-mediated structural modulation and hinge motion in the C4-C5 domains of cMyBPC. Journal of Structural Biology. 214(2). 107856–107856. 7 indexed citations
2.
Li, Jiayang, et al.. (2020). AAV9 gene transfer of cMyBPC N-terminal domains ameliorates cardiomyopathy in cMyBPC-deficient mice. JCI Insight. 5(17). 19 indexed citations
3.
Li, Jiayang, et al.. (2019). The HCM-causing Y235S cMyBPC mutation accelerates contractile function by altering C1 domain structure. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1865(3). 661–677. 15 indexed citations
4.
Li, Jiayang, et al.. (2019). The Hcm-Causing Y235S Cmybpc Mutation Accelerates Contractile Function by Altering C1 Domain Structure. Biophysical Journal. 116(3). 266a–266a. 3 indexed citations
5.
Li, Jiayang, Kenneth S. Gresham, Ranganath Mamidi, et al.. (2018). Sarcomere-based genetic enhancement of systolic cardiac function in a murine model of dilated cardiomyopathy. International Journal of Cardiology. 273. 168–176. 14 indexed citations
6.
Mamidi, Ranganath, et al.. (2018). Lost in translation: Interpreting cardiac muscle mechanics data in clinical practice. Archives of Biochemistry and Biophysics. 662. 213–218. 10 indexed citations
7.
Mamidi, Ranganath, et al.. (2018). Impact of the Myosin Modulator Mavacamten on Force Generation and Cross‐Bridge Behavior in a Murine Model of Hypercontractility. Journal of the American Heart Association. 7(17). e009627–e009627. 46 indexed citations
8.
Mamidi, Ranganath, Jiayang Li, Kenneth S. Gresham, et al.. (2017). Dose-Dependent Effects of the Myosin Activator Omecamtiv Mecarbil on Cross-Bridge Behavior and Force Generation in Failing Human Myocardium. Circulation Heart Failure. 10(10). 41 indexed citations
9.
Mamidi, Ranganath, Kenneth S. Gresham, Sujeet Verma, & Julian E. Stelzer. (2016). Cardiac Myosin Binding Protein-C Phosphorylation Modulates Myofilament Length-Dependent Activation. Frontiers in Physiology. 7. 38–38. 48 indexed citations
10.
Gresham, Kenneth S., et al.. (2016). Sarcomeric protein modification during adrenergic stress enhances cross-bridge kinetics and cardiac output. Journal of Applied Physiology. 122(3). 520–530. 14 indexed citations
11.
Mamidi, Ranganath, Kenneth S. Gresham, Amy Li, Cristobal G. dos Remedios, & Julian E. Stelzer. (2015). Molecular effects of the myosin activator omecamtiv mecarbil on contractile properties of skinned myocardium lacking cardiac myosin binding protein-C. Journal of Molecular and Cellular Cardiology. 85. 262–272. 42 indexed citations
12.
Mamidi, Ranganath, Kenneth S. Gresham, & Julian E. Stelzer. (2014). Length-dependent changes in contractile dynamics are blunted due to cardiac myosin binding protein-C ablation. Frontiers in Physiology. 5. 461–461. 36 indexed citations
13.
Mamidi, Ranganath, Jiayang Li, Kenneth S. Gresham, & Julian E. Stelzer. (2013). Cardiac myosin binding protein-C: a novel sarcomeric target for gene therapy. Pflügers Archiv - European Journal of Physiology. 466(2). 225–230. 13 indexed citations
14.
Mamidi, Ranganath, Mariappan Muthuchamy, & Murali Chandra. (2013). Instability in the Central Region of Tropomyosin Modulates the Function of Its Overlapping Ends. Biophysical Journal. 105(9). 2104–2113. 6 indexed citations
15.
Mamidi, Ranganath & Murali Chandra. (2013). Divergent effects of α- and β-myosin heavy chain isoforms on the N terminus of rat cardiac troponin T. The Journal of General Physiology. 142(4). 413–423. 15 indexed citations
16.
Ford, Steven J., Ranganath Mamidi, Jesús Jiménez, Jil C. Tardiff, & Murali Chandra. (2012). Effects of R92 mutations in mouse cardiac troponin T are influenced by changes in myosin heavy chain isoform. Journal of Molecular and Cellular Cardiology. 53(4). 542–551. 38 indexed citations
17.
Mamidi, Ranganath, et al.. (2012). Identification of two new regions in the N‐terminus of cardiac troponin T that have divergent effects on cardiac contractile function. The Journal of Physiology. 591(5). 1217–1234. 21 indexed citations
18.
Gollapudi, Sampath K., et al.. (2012). The N-Terminal Extension of Cardiac Troponin T Stabilizes the Blocked State of Cardiac Thin Filament. Biophysical Journal. 103(5). 940–948. 33 indexed citations
19.
Mamidi, Ranganath, et al.. (2010). Structural and Kinetic Effects of PAK3 Phosphorylation Mimic of cTnI(S151E) on the cTnC-cTnI Interaction in the Cardiac Thin Filament. Journal of Molecular Biology. 400(5). 1036–1045. 9 indexed citations
20.
Rodgers, Buel D., Dilip K. Garikipati, Ranganath Mamidi, et al.. (2009). Myostatin represses physiological hypertrophy of the heart and excitation–contraction coupling. The Journal of Physiology. 587(20). 4873–4886. 52 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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