Rachel L. Self

623 total citations
19 papers, 492 citations indexed

About

Rachel L. Self is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Virology. According to data from OpenAlex, Rachel L. Self has authored 19 papers receiving a total of 492 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Cellular and Molecular Neuroscience, 9 papers in Molecular Biology and 5 papers in Virology. Recurrent topics in Rachel L. Self's work include Neuroscience and Neuropharmacology Research (12 papers), HIV Research and Treatment (5 papers) and Stress Responses and Cortisol (3 papers). Rachel L. Self is often cited by papers focused on Neuroscience and Neuropharmacology Research (12 papers), HIV Research and Treatment (5 papers) and Stress Responses and Cortisol (3 papers). Rachel L. Self collaborates with scholars based in United States and United Kingdom. Rachel L. Self's co-authors include Mark Prendergast, Patrick J. Mulholland, John M. Littleton, Barton R. Harris, Avindra Nath, Tracy R. Butler, Katherine J. Smith, Lincoln H. Wilkins, H.J. Little and Dennis T. Rogers and has published in prestigious journals such as Brain Research, Neuroscience and Alcoholism Clinical and Experimental Research.

In The Last Decade

Rachel L. Self

19 papers receiving 481 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Rachel L. Self United States 14 174 141 117 89 56 19 492
Yutaka Yamamuro Japan 12 88 0.5× 140 1.0× 37 0.3× 39 0.4× 24 0.4× 36 478
Julia Jecel Austria 7 91 0.5× 67 0.5× 18 0.2× 25 0.3× 88 1.6× 9 413
Brian M. Kelley United States 14 344 2.0× 227 1.6× 43 0.4× 35 0.4× 50 0.9× 22 631
Erinn S. Raborn United States 7 163 0.9× 76 0.5× 45 0.4× 57 0.6× 9 0.2× 8 453
J. Blackbeard United Kingdom 8 266 1.5× 67 0.5× 80 0.7× 51 0.6× 54 1.0× 8 652
Ester Marı́a López Argentina 14 160 0.9× 130 0.9× 6 0.1× 52 0.6× 16 0.3× 26 417
Itsuo Tokunaga Japan 15 142 0.8× 143 1.0× 5 0.0× 68 0.8× 13 0.2× 49 573
Bárbara Kremeyer United Kingdom 9 142 0.8× 122 0.9× 55 0.5× 13 0.1× 6 0.1× 10 535
Soraya Wilke Saliba Germany 13 128 0.7× 199 1.4× 5 0.0× 229 2.6× 36 0.6× 22 629
James E. Richards United Kingdom 11 214 1.2× 196 1.4× 20 0.2× 28 0.3× 24 0.4× 16 822

Countries citing papers authored by Rachel L. Self

Since Specialization
Citations

This map shows the geographic impact of Rachel L. Self's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rachel L. Self with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rachel L. Self more than expected).

Fields of papers citing papers by Rachel L. Self

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rachel L. Self. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rachel L. Self. The network helps show where Rachel L. Self may publish in the future.

Co-authorship network of co-authors of Rachel L. Self

This figure shows the co-authorship network connecting the top 25 collaborators of Rachel L. Self. A scholar is included among the top collaborators of Rachel L. Self based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rachel L. Self. Rachel L. Self is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
4.
Dunlop, Boadie W. & Rachel L. Self. (2008). Exercise for depression: efficacy, safety and clinical trial implications.. PubMed. 41(4). 65–75. 5 indexed citations
5.
Butler, Tracy R., et al.. (2008). Sex Differences in the Neurotoxic Effects of Adenosine A1 Receptor Antagonism During Ethanol Withdrawal: Reversal With an A1 Receptor Agonist or an NMDA Receptor Antagonist. Alcoholism Clinical and Experimental Research. 32(7). 1260–1270. 19 indexed citations
6.
Prendergast, Mark, Rachel L. Self, Katherine J. Smith, et al.. (2007). Microtubule-associated targets in chlorpyrifos oxon hippocampal neurotoxicity. Neuroscience. 146(1). 330–339. 62 indexed citations
8.
Smith, Katherine J., Rachel L. Self, Tracy R. Butler, et al.. (2007). Methamphetamine exposure antagonizes N-methyl-d-aspartate receptor-mediated neurotoxicity in organotypic hippocampal slice cultures. Brain Research. 1157. 74–80. 14 indexed citations
10.
Self, Rachel L., Katherine J. Smith, Patrick J. Mulholland, & Mark Prendergast. (2005). Ethanol Exposure and Withdrawal Sensitizes the Rat Hippocampal CA1 Pyramidal Cell Region to ??-Amyloid (25-35)-Induced Cytotoxicity: NMDA Receptor Involvement. Alcoholism Clinical and Experimental Research. 29(11). 2063–2069. 18 indexed citations
11.
Mulholland, Patrick J., et al.. (2005). Thiamine deficiency in the pathogenesis of chronic ethanol-associated cerebellar damage in vitro. Neuroscience. 135(4). 1129–1139. 28 indexed citations
12.
Mulholland, Patrick J., Rachel L. Self, Barton R. Harris, et al.. (2005). Corticosterone Increases Damage and Cytosolic Calcium Accumulation Associated With Ethanol Withdrawal in Rat Hippocampal Slice Cultures. Alcoholism Clinical and Experimental Research. 29(5). 871–881. 31 indexed citations
13.
Mulholland, Patrick J., et al.. (2005). Corticosterone and dexamethasone potentiate cytotoxicity associated with oxygen-glucose deprivation in organotypic cerebellar slice cultures. Neuroscience. 136(1). 259–267. 14 indexed citations
14.
Mulholland, Patrick J., Rachel L. Self, Barton R. Harris, John M. Littleton, & Mark Prendergast. (2004). Choline exposure reduces potentiation of N-methyl-d-aspartate toxicity by corticosterone in the developing hippocampus. Developmental Brain Research. 153(2). 203–211. 13 indexed citations
15.
Mulholland, Patrick J., Rachel L. Self, Barton R. Harris, John M. Littleton, & Mark Prendergast. (2004). (−)-nicotine ameliorates corticosterone's potentiation of N-methyl-d-aspartate receptor-mediated cornu ammonis 1 toxicity. Neuroscience. 125(3). 671–682. 23 indexed citations
16.
Self, Rachel L., Patrick J. Mulholland, Barton R. Harris, Avindra Nath, & Mark Prendergast. (2004). Cytotoxic Effects of Exposure to the Human Immunodeficiency Virus Type 1 Protein Tat in the Hippocampus Are Enhanced by Prior Ethanol Treatment. Alcoholism Clinical and Experimental Research. 28(12). 1916–1924. 23 indexed citations
17.
Self, Rachel L., Patrick J. Mulholland, Avindra Nath, Barton R. Harris, & Mark Prendergast. (2003). The human immunodeficiency virus type-1 transcription factor Tat produces elevations in intracellular Ca2+ that require function of an N-methyl-d-aspartate receptor polyamine-sensitive site. Brain Research. 995(1). 39–45. 58 indexed citations
18.
Mulholland, Patrick J., Barton R. Harris, Lincoln H. Wilkins, et al.. (2003). Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression. Alcohol. 31(1-2). 1–10. 21 indexed citations
19.
Prendergast, Mark, Dennis T. Rogers, Patrick J. Mulholland, et al.. (2002). Neurotoxic effects of the human immunodeficiency virus type-1 transcription factor Tat require function of a polyamine sensitive-site on the N-methyl-d-aspartate receptor. Brain Research. 954(2). 300–307. 67 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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