Rachel E. Jenkins
- Pathology and Forensic Medicine top 5%
- Rheumatology top 5%
- Genetics top 10%
- Pharmacology
- Dermatology top 10%
- Co-authors
- Martin M. BlackStefanie A JonesJeff K. ShornickDavid BriggsSusan KellyS.E. HANDFIELD-JONESD. McGibbonCéline Besse
- Topics
- Autoimmune Bullous Skin Diseases (10 papers)Urticaria and Related Conditions (8 papers)Coagulation, Bradykinin, Polyphosphates, and Angioedema (7 papers)
- Partner nations
- United KingdomUnited StatesThailand
In The Last Decade
Rachel E. Jenkins
19 papers receiving 350 citations
Peers
Comparison fields: 5 of 50
- Pathology and Forensic Medicine 252
- Rheumatology 231
- Genetics 154
- Pharmacology 55
- Dermatology 51
Countries citing papers authored by Rachel E. Jenkins
This map shows the geographic impact of Rachel E. Jenkins's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rachel E. Jenkins with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rachel E. Jenkins more than expected).
Fields of papers citing papers by Rachel E. Jenkins
This network shows the impact of papers produced by Rachel E. Jenkins. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rachel E. Jenkins. The network helps show where Rachel E. Jenkins may publish in the future.
Co-authorship network of co-authors of Rachel E. Jenkins
This figure shows the co-authorship network connecting the top 25 collaborators of Rachel E. Jenkins. A scholar is included among the top collaborators of Rachel E. Jenkins based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rachel E. Jenkins. Rachel E. Jenkins is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 72 | |
| 3 | 18 | |
| 4 | 3 | |
| 5 | 21 | |
| 6 | Conversion of pemphigoid gestationis to bullous pemphigoid--two refractory cases highlighting this association. | 28 |
| 7 | 26 | |
| 8 | 3 | |
| 9 | 31 | |
| 10 | 14 | |
| 11 | 8 | |
| 12 | 4 | |
| 13 | 21 | |
| 14 | 21 | |
| 15 | 33 | |
| 16 | 53 | |
| 17 | 10 | |
| 18 | 2 | |
| 19 | 1 |
About Rachel E. Jenkins
Rachel E. Jenkins is a scholar working on Rheumatology, Genetics and Pathology and Forensic Medicine, having authored 19 papers that have together received 372 indexed citations. Recurring topics across this work include Autoimmune Bullous Skin Diseases (10 papers), Urticaria and Related Conditions (8 papers) and Coagulation, Bradykinin, Polyphosphates, and Angioedema (7 papers). The work is most often cited by research in Rheumatology (231 citations), Genetics (154 citations) and Pathology and Forensic Medicine (252 citations). Rachel E. Jenkins has collaborated with scholars based in United Kingdom, United States and Thailand. Frequent co-authors include Martin M. Black, Stefanie A Jones, Jeff K. Shornick, David Briggs, Susan Kelly, S.E. HANDFIELD-JONES, D. McGibbon, Céline Besse, Sean Whittaker and Wang Sh. Their work appears in journals such as Journal of the American Academy of Dermatology, British Journal of Dermatology and Lara D. Veeken.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.