R J Murray

1.8k total citations
14 papers, 1.5k citations indexed

About

R J Murray is a scholar working on Oncology, Immunology and Pathology and Forensic Medicine. According to data from OpenAlex, R J Murray has authored 14 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Oncology, 8 papers in Immunology and 7 papers in Pathology and Forensic Medicine. Recurrent topics in R J Murray's work include Viral-associated cancers and disorders (11 papers), Lymphoma Diagnosis and Treatment (7 papers) and Immune Cell Function and Interaction (6 papers). R J Murray is often cited by papers focused on Viral-associated cancers and disorders (11 papers), Lymphoma Diagnosis and Treatment (7 papers) and Immune Cell Function and Interaction (6 papers). R J Murray collaborates with scholars based in United Kingdom, Sweden and Netherlands. R J Murray's co-authors include Alan B. Rickinson, Michael G. Kurilla, Martin Rowe, Jill Brooks, Wendy A. Thomas, Christopher D. Gregory, C. Edwards, E Kieff, Lawrence S. Young and Maria G. Masucci and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Experimental Medicine and Journal of Virology.

In The Last Decade

R J Murray

14 papers receiving 1.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
R J Murray United Kingdom	12	1.1k	776	522	508	311	14	1.5k
Heather M. Long United Kingdom	24	1.2k 1.1×	902 1.2×	553 1.1×	596 1.2×	270 0.9×	37	1.8k
Tracey A. Haigh United Kingdom	19	757 0.7×	702 0.9×	270 0.5×	466 0.9×	107 0.3×	26	1.2k
T B Sculley Australia	26	1.8k 1.6×	937 1.2×	878 1.7×	776 1.5×	564 1.8×	53	2.3k
D Liebowitz United States	13	1.5k 1.4×	414 0.5×	881 1.7×	328 0.6×	362 1.2×	14	1.7k
Maria Teresa Bejarano Sweden	16	510 0.5×	484 0.6×	257 0.5×	190 0.4×	93 0.3×	25	969
Shigehiko Ishihara Japan	18	655 0.6×	557 0.7×	434 0.8×	97 0.2×	166 0.5×	41	1.2k
Wendy A. Thomas United Kingdom	17	1.5k 1.4×	1.1k 1.4×	677 1.3×	645 1.3×	192 0.6×	18	1.9k
Ken‐Ichi Imadome Japan	22	859 0.8×	606 0.8×	409 0.8×	330 0.6×	202 0.6×	103	1.4k
Dai Iwakiri Japan	19	955 0.9×	698 0.9×	309 0.6×	454 0.9×	233 0.7×	26	1.8k
Deanna M. Grote United States	13	546 0.5×	698 0.9×	349 0.7×	225 0.4×	86 0.3×	23	1.4k

Countries citing papers authored by R J Murray

Since Specialization

Citations

This map shows the geographic impact of R J Murray's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by R J Murray with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites R J Murray more than expected).

Fields of papers citing papers by R J Murray

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by R J Murray. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by R J Murray. The network helps show where R J Murray may publish in the future.

Co-authorship network of co-authors of R J Murray

This figure shows the co-authorship network connecting the top 25 collaborators of R J Murray. A scholar is included among the top collaborators of R J Murray based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with R J Murray. R J Murray is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Murray, R J, David A. Boyd, Paul N. Levett, Michael R. Mulvey, & Michelle J. Alfa. (2009). Truncation in the tcdC region of the Clostridium difficilePathLoc of clinical isolates does not predict increased biological activity of Toxin B or Toxin A. BMC Infectious Diseases. 9(1). 103–103. 54 indexed citations

Gregory, Christopher D., et al.. (2009). Inhibitory effects of persistent apoptotic cells on monoclonal antibody production in vitro. mAbs. 1(4). 370–376. 20 indexed citations

Brooks, Jill, R J Murray, Wendy A. Thomas, Michael G. Kurilla, & Alan B. Rickinson. (1993). Different HLA-B27 subtypes present the same immunodominant Epstein-Barr virus peptide.. The Journal of Experimental Medicine. 178(3). 879–887. 89 indexed citations

Gavioli, Riccardo, Michael G. Kurilla, Pedro O. de Campos‐Lima, et al.. (1993). Multiple HLA A11-restricted cytotoxic T-lymphocyte epitopes of different immunogenicities in the Epstein-Barr virus-encoded nuclear antigen 4. Journal of Virology. 67(3). 1572–1578. 146 indexed citations

Thomas, Wendy A., R J Murray, Farhat L. Khanim, et al.. (1993). HLA A2.1-restricted cytotoxic T cells recognizing a range of Epstein-Barr virus isolates through a defined epitope in latent membrane protein LMP2. Journal of Virology. 67(12). 7428–7435. 161 indexed citations

Murray, R J, Michael G. Kurilla, Jill Brooks, et al.. (1992). Identification of target antigens for the human cytotoxic T cell response to Epstein-Barr virus (EBV): implications for the immune control of EBV-positive malignancies.. The Journal of Experimental Medicine. 176(1). 157–168. 428 indexed citations

Masucci, Maria G., Riccardo Gavioli, R J Murray, et al.. (1992). Immune escape by Epstein-Barr virus (EBV) carrying Burkitt's lymphoma: in vitro reconstitution of sensitivity to EBV-specific cytotoxic T cells. International Immunology. 4(11). 1283–1292. 21 indexed citations

Griffin, Heather, Martin Rowe, R J Murray, Jill Brooks, & Alan B. Rickinson. (1992). Restoration of the LFA‐3 adhesion pathway in Burkitt's lymphoma cells using an LFA‐3 recombinant vaccinia virus: consequences for T cell recognition. European Journal of Immunology. 22(7). 1741–1748. 4 indexed citations

Rickinson, Alan B., et al.. (1992). T cell recognition of Epstein-Barr virus associated lymphomas.. PubMed. 13. 53–80. 79 indexed citations

10.

Murray, R J, Jill Brooks, Alan B. Rickinson, & Martin Rowe. (1990). Cross‐recognition of a mouse H‐2‐peptide complex by human HLA‐restricted cytotoxic T cells. European Journal of Immunology. 20(3). 659–664. 6 indexed citations

11.

Murray, R J, Michael G. Kurilla, Jill Brooks, et al.. (1990). Human cytotoxic T-cell responses against Epstein-Barr virus nuclear antigens demonstrated by using recombinant vaccinia viruses.. Proceedings of the National Academy of Sciences. 87(8). 2906–2910. 85 indexed citations

12.

Gregory, Christopher D., R J Murray, C. Edwards, & Alan B. Rickinson. (1988). Downregulation of cell adhesion molecules LFA-3 and ICAM-1 in Epstein-Barr virus-positive Burkitt's lymphoma underlies tumor cell escape from virus-specific T cell surveillance.. The Journal of Experimental Medicine. 167(6). 1811–1824. 274 indexed citations

13.

Murray, R J, et al.. (1988). Epstein-Barr virus-specific cytotoxic T-cell recognition of transfectants expressing the virus-coded latent membrane protein LMP. Journal of Virology. 62(10). 3747–3755. 96 indexed citations

14.

Murray, R J, Lawrence S. Young, Alain Calender, et al.. (1988). Different patterns of Epstein-Barr virus gene expression and of cytotoxic T-cell recognition in B-cell lines infected with transforming (B95.8) or nontransforming (P3HR1) virus strains. Journal of Virology. 62(3). 894–901. 72 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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