Qin Yi

745 total citations
42 papers, 510 citations indexed

About

Qin Yi is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Qin Yi has authored 42 papers receiving a total of 510 indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 6 papers in Pulmonary and Respiratory Medicine and 5 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Qin Yi's work include RNA modifications and cancer (7 papers), Mitochondrial Function and Pathology (6 papers) and Pluripotent Stem Cells Research (5 papers). Qin Yi is often cited by papers focused on RNA modifications and cancer (7 papers), Mitochondrial Function and Pathology (6 papers) and Pluripotent Stem Cells Research (5 papers). Qin Yi collaborates with scholars based in China, United States and Spain. Qin Yi's co-authors include Jing Zhu, Bin Tan, Hao Xu, Jie Tian, Qin Zhou, Xuan Jing, Xiangrong Cui, Xinyuan Zhang, Chunlan Long and Yasha Li and has published in prestigious journals such as Scientific Reports, Chemical Engineering Journal and Biochemical and Biophysical Research Communications.

In The Last Decade

Qin Yi

38 papers receiving 504 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Qin Yi China 16 313 130 89 70 56 42 510
Yangguang Yin China 12 293 0.9× 80 0.6× 47 0.5× 108 1.5× 53 0.9× 20 550
Lingling Shu China 14 274 0.9× 125 1.0× 75 0.8× 50 0.7× 87 1.6× 35 596
Guilin Chen China 14 171 0.5× 95 0.7× 59 0.7× 44 0.6× 43 0.8× 36 434
Cheng Gao China 12 192 0.6× 85 0.7× 75 0.8× 141 2.0× 49 0.9× 31 430
Yan Meng China 12 210 0.7× 90 0.7× 130 1.5× 52 0.7× 101 1.8× 27 521
Jari P. Lappalainen Finland 9 276 0.9× 70 0.5× 56 0.6× 81 1.2× 128 2.3× 9 514
Sarah Franco United States 8 307 1.0× 75 0.6× 75 0.8× 49 0.7× 72 1.3× 13 444

Countries citing papers authored by Qin Yi

Since Specialization
Citations

This map shows the geographic impact of Qin Yi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Qin Yi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Qin Yi more than expected).

Fields of papers citing papers by Qin Yi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Qin Yi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Qin Yi. The network helps show where Qin Yi may publish in the future.

Co-authorship network of co-authors of Qin Yi

This figure shows the co-authorship network connecting the top 25 collaborators of Qin Yi. A scholar is included among the top collaborators of Qin Yi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Qin Yi. Qin Yi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Gao, Ziwei, Xiaoying Hou, Jun Tang, et al.. (2025). β-glucan nanotubes improve oral doxorubicin delivery for colorectal cancer by microbiota-mediated colon targeting and reduced toxicity. Carbohydrate Polymers. 374. 124705–124705.
2.
Ge, Gaoran, Xiaoqin Yang, Qin Yi, et al.. (2025). Anthill-Inspired bionic bone cement enhances bone regeneration by guiding angiogenesis and regulating BMSCs metabolism with anti-ROS capacity. Chemical Engineering Journal. 510. 161512–161512. 2 indexed citations
3.
4.
Yi, Qin, et al.. (2024). Use of consensus clustering to identify distinct subtypes of chronic kidney disease and associated mortality risk. Scientific Reports. 14(1). 29893–29893. 1 indexed citations
5.
6.
Li, Fan, et al.. (2024). Calcitonin gene-related peptide attenuated discogenic low back pain in rats possibly via inhibiting microglia activation. Heliyon. 10(3). e25906–e25906. 3 indexed citations
7.
8.
Xu, Hao, Bin Tan, Qin Yi, et al.. (2023). AKAP1 Regulates Mitochondrial Dynamics during the Fatty-Acid-Promoted Maturation of Human-Induced Pluripotent Stem Cell-Derived Cardiomyocytes as Indicated by Proteomics Sequencing. International Journal of Molecular Sciences. 24(9). 8112–8112. 4 indexed citations
9.
Sun, Yanting, Hao Xu, Bin Tan, et al.. (2023). Andrographolide-treated bone marrow mesenchymal stem cells-derived conditioned medium protects cardiomyocytes from injury by metabolic remodeling. Molecular Biology Reports. 50(3). 2651–2662. 2 indexed citations
10.
Liu, Huiwen, Li Wang, Hao Xu, et al.. (2023). Quantitative proteomic and phosphoproteomic analysis reveal the relationship between mitochondrial dysfunction and cytoskeletal remodeling in hiPSC-CMs deficient in PINK1. Journal of Translational Medicine. 21(1). 581–581. 2 indexed citations
11.
Chen, Yunying, et al.. (2023). DISCOVERY OF NOVEL ANTI-SERUM ALBUMIN VHH AS A BUILDING BLOCK FOR PK PROLONGATION. PubMed Central. 6(Supplement_1). 1 indexed citations
12.
Xiao, Fei, Qin Yi, Jing Chen, et al.. (2022). The propofol-induced mitochondrial damage in fetal rat hippocampal neurons via the AMPK/P53 signaling pathway. Annals of Translational Medicine. 10(20). 1106–1106. 7 indexed citations
13.
Wang, Rui, Hao Xu, Bin Tan, et al.. (2022). SIRT3 promotes metabolic maturation of human iPSC-derived cardiomyocytes via OPA1-controlled mitochondrial dynamics. Free Radical Biology and Medicine. 195. 270–282. 28 indexed citations
14.
Sun, Yanting, Hao Xu, Bin Tan, et al.. (2022). Andrographolide protects bone marrow mesenchymal stem cells against glucose and serum deprivation under hypoxia via the NRF2 signaling pathway. Stem Cell Research & Therapy. 13(1). 326–326. 13 indexed citations
15.
Zhang, Xinyuan, Ye Liang, Hao Xu, et al.. (2021). NRF2 is required for structural and metabolic maturation of human induced pluripotent stem cell-derived ardiomyocytes. Stem Cell Research & Therapy. 12(1). 208–208. 17 indexed citations
16.
Liang, Ye, Xinyuan Zhang, Qin Zhou, et al.. (2021). Activation of AMPK Promotes Maturation of Cardiomyocytes Derived From Human Induced Pluripotent Stem Cells. Frontiers in Cell and Developmental Biology. 9. 644667–644667. 27 indexed citations
17.
Zhou, Qin, Min Xie, Jing Zhu, et al.. (2021). PINK1 contained in huMSC-derived exosomes prevents cardiomyocyte mitochondrial calcium overload in sepsis via recovery of mitochondrial Ca2+ efflux. Stem Cell Research & Therapy. 12(1). 269–269. 52 indexed citations
18.
Li, Yiming, Junke Long, Jiaquan Chen, et al.. (2020). Analysis of Spatiotemporal Urine Protein Dynamics to Identify New Biomarkers for Sepsis-Induced Acute Kidney Injury. Frontiers in Physiology. 11. 139–139. 8 indexed citations
19.
Li, Yasha, Haiying Zhong, Bin Tan, et al.. (2019). Decline of p300 contributes to cell senescence and growth inhibition of hUC-MSCs through p53/p21 signaling pathway. Biochemical and Biophysical Research Communications. 515(1). 24–30. 17 indexed citations
20.
Yi, Qin, Beilei Zeng, Yixiao Feng, et al.. (2019). <p>Long noncoding RNA LINC01089 predicts clinical prognosis and inhibits cell proliferation and invasion through the Wnt/β-catenin signaling pathway in breast cancer</p>. OncoTargets and Therapy. Volume 12. 4883–4895. 35 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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