Qicai Wu

571 total citations
22 papers, 416 citations indexed

About

Qicai Wu is a scholar working on Molecular Biology, Cancer Research and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Qicai Wu has authored 22 papers receiving a total of 416 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 7 papers in Cancer Research and 6 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Qicai Wu's work include RNA modifications and cancer (6 papers), Cancer-related molecular mechanisms research (6 papers) and Mitochondrial Function and Pathology (4 papers). Qicai Wu is often cited by papers focused on RNA modifications and cancer (6 papers), Cancer-related molecular mechanisms research (6 papers) and Mitochondrial Function and Pathology (4 papers). Qicai Wu collaborates with scholars based in China, Canada and United States. Qicai Wu's co-authors include Xueliang Zhou, Rongrong Zhu, Huang Huang, Jichun Liu, Li Wan, Qirong Xu, Jichun Liu, Jinlong Liu, Shuaishuai Zhao and Xinping Xu and has published in prestigious journals such as Cellular and Molecular Life Sciences, Journal of Cellular Physiology and Journal of Cellular Biochemistry.

In The Last Decade

Qicai Wu

20 papers receiving 415 citations

Peers

Qicai Wu

CCM

PRM

SURGE

Huang Huang China

Bofang Zhang China

Linmao Lyu China

Jingmin Zhou China

Youbin Liu China

Shunlin Qu China

Miyesaier Abudureyimu China

Yuqiong Chen China

Zhaofen Zheng China

Huang Huang China

Qicai Wu

285 ×1.0

128 ×1.0

74 ×0.6

CCM

123 ×2.2

PRM

30 ×0.8

SURGE

Citations per year, relative to Qicai Wu Qicai Wu (= 1×) peers Huang Huang

Countries citing papers authored by Qicai Wu

Since Specialization

Citations

This map shows the geographic impact of Qicai Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Qicai Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Qicai Wu more than expected).

Fields of papers citing papers by Qicai Wu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Qicai Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Qicai Wu. The network helps show where Qicai Wu may publish in the future.

Co-authorship network of co-authors of Qicai Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Qicai Wu. A scholar is included among the top collaborators of Qicai Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Qicai Wu. Qicai Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Zhao, Shuaishuai, et al.. (2025). Building and validating a machine learning model to predict coronary heart disease risk based on non-invasive indicators. Computer Methods and Programs in Biomedicine. 275. 109186–109186.

Zhao, Shuaishuai, et al.. (2025). SETD2 drives METTL14-mediated m6A to suppress Piezo1 Attenuation and activate TGM2 to promote pulmonary hypertension. Cellular and Molecular Life Sciences. 82(1). 302–302.

Zhao, Shuaishuai, et al.. (2024). Lactate regulates pathological cardiac hypertrophy via histone lactylation modification. Journal of Cellular and Molecular Medicine. 28(16). e70022–e70022. 23 indexed citations

Zhao, Shuaishuai, Jinlong Liu, Qicai Wu, & Xueliang Zhou. (2023). Role of histone lactylation interference RNA m6A modification and immune microenvironment homeostasis in pulmonary arterial hypertension. Frontiers in Cell and Developmental Biology. 11. 1268646–1268646. 18 indexed citations

Wang, Fudong, et al.. (2022). Summary of the Experiences and Results of Transesophageal Ultrasound-Guided Ventricular Septal Defect and Atrial Septal Defect Closure Operation. The Heart Surgery Forum. 25(5). E638–E644. 1 indexed citations

Lai, Songqing, Xueliang Zhou, Qing Wan, et al.. (2022). Ischemic preconditioning/ischemic postconditioning alleviates anoxia/reoxygenation injury via the Notch1/Hes1/VDAC1 axis. Journal of Biochemical and Molecular Toxicology. 36(11). e23199–e23199. 8 indexed citations

Wu, Qicai, et al.. (2022). Integrated Single-Cell and RNA Sequencing Analysis Identifies Key Immune Cell and Dendritic Cells Associated Genes Participated in Myocarditis. Journal of Immunology Research. 2022. 1–21. 3 indexed citations

Huang, Huang, Guozhu Ye, Songqing Lai, et al.. (2021). Plasma Lipidomics Identifies Unique Lipid Signatures and Potential Biomarkers for Patients With Aortic Dissection. Frontiers in Cardiovascular Medicine. 8. 757022–757022. 15 indexed citations

Zhou, Xueliang, et al.. (2021). SEDT2/METTL14-mediated m6A methylation awakening contributes to hypoxia-induced pulmonary arterial hypertension in mice. Aging. 13(5). 7538–7548. 40 indexed citations

10.

Zhou, Xueliang, Xia Wu, Rongrong Zhu, et al.. (2020). Notch1–Nrf2 signaling crosstalk provides myocardial protection by reducing ROS formation. Biochemistry and Cell Biology. 98(2). 106–111. 23 indexed citations

11.

Zhu, Rongrong, et al.. (2020). Inhibition of the Notch1 pathway induces peripartum cardiomyopathy. Journal of Cellular and Molecular Medicine. 24(14). 7907–7914. 12 indexed citations

12.

Huang, Huang, Songqing Lai, Yong Luo, et al.. (2019). Nutritional Preconditioning of Apigenin Alleviates Myocardial Ischemia/Reperfusion Injury via the Mitochondrial Pathway Mediated by Notch1/Hes1. Oxidative Medicine and Cellular Longevity. 2019. 1–15. 27 indexed citations

13.

Zhou, Xueliang, Zhibo Liu, Rongrong Zhu, et al.. (2019). NSD2 silencing alleviates pulmonary arterial hypertension by inhibiting trehalose metabolism and autophagy. Clinical Science. 133(9). 1085–1096. 16 indexed citations

14.

Zhou, Xueliang, Li Wan, Qirong Xu, et al.. (2018). Notch signaling inhibits cardiac fibroblast to myofibroblast transformation by antagonizing TGF‐β1/Smad3 signaling. Journal of Cellular Physiology. 234(6). 8834–8845. 40 indexed citations

15.

Zhou, Xueliang, et al.. (2018). miR‐21 promotes cardiac fibroblast‐to‐myofibroblast transformation and myocardial fibrosis by targeting Jagged1. Journal of Cellular and Molecular Medicine. 22(8). 3816–3824. 76 indexed citations

16.

Zhou, Xueliang, et al.. (2018). Notch signaling promotes angiogenesis and improves cardiac function after myocardial infarction. Journal of Cellular Biochemistry. 119(8). 7105–7112. 26 indexed citations

17.

Zhou, Xueliang, Rongrong Zhu, Xia Wu, et al.. (2018). NSD2 promotes ventricular remodelling mediated by the regulation of H3K36me2. Journal of Cellular and Molecular Medicine. 23(1). 568–575. 10 indexed citations

18.

Huang, Huang, Qicai Wu, Liang Zeng, et al.. (2017). Heating paper spray mass spectrometry for enhanced detection of propranolol in dried blood samples. Analytical Methods. 9(29). 4282–4287. 13 indexed citations

19.

Liu, Jichun, et al.. (2015). Genetic risk of lung cancer associated with a single nucleotide polymorphism from EXO1: a meta analysis.. PubMed. 8(7). 11132–8. 11 indexed citations

20.

Yu, Bentong, et al.. (2013). [Roles of targeting Ras/Raf/MEK/ERK signaling pathways in the treatment of esophageal carcinoma].. PubMed. 48(5). 635–41. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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