Qiang Meng

692 total citations
28 papers, 408 citations indexed

About

Qiang Meng is a scholar working on Molecular Biology, Neurology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Qiang Meng has authored 28 papers receiving a total of 408 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 8 papers in Neurology and 6 papers in Cellular and Molecular Neuroscience. Recurrent topics in Qiang Meng's work include S100 Proteins and Annexins (6 papers), Neuroscience and Neuropharmacology Research (4 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). Qiang Meng is often cited by papers focused on S100 Proteins and Annexins (6 papers), Neuroscience and Neuropharmacology Research (4 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). Qiang Meng collaborates with scholars based in China, United States and Netherlands. Qiang Meng's co-authors include Liang Cheng, Liming Yu, Shiqiang Yu, Zhenxiao Jin, Yang Yang, Haifeng Zhang, Mengen Zhai, Yu Zhang, Weixun Duan and Yang Sun and has published in prestigious journals such as SHILAP Revista de lepidopterología, Brain Research and Neurobiology of Disease.

In The Last Decade

Qiang Meng

24 papers receiving 402 citations

Peers

Countries citing papers authored by Qiang Meng

Since Specialization

Citations

This map shows the geographic impact of Qiang Meng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Qiang Meng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Qiang Meng more than expected).

Fields of papers citing papers by Qiang Meng

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Qiang Meng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Qiang Meng. The network helps show where Qiang Meng may publish in the future.

Co-authorship network of co-authors of Qiang Meng

This figure shows the co-authorship network connecting the top 25 collaborators of Qiang Meng. A scholar is included among the top collaborators of Qiang Meng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Qiang Meng. Qiang Meng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Albiflorin ameliorates neuroinflammation and exerts neuroprotective effects in Parkinson’s disease models 2025 ·Immunopharmacology and Immunotoxicology ·Yuan Gao, Yanmei Chen, Ning Wang, Qiang Meng	2
2	Neurochemical and molecular characteristics of altered brain functional activity in the anti-NMDAR encephalitis 2025 ·Neurobiology of Disease ·(unknown), Wei Li, Cheng‐Liang Luo, Yanghua Tian, Jiaojian Wang, Yuan Gao, (unknown), (unknown), Qiang Meng	0
3	Best1 mitigates ER stress induced by the increased cellular microenvironment stiffness in epilepsy 2024 ·Neurobiology of Disease ·(unknown), (unknown), Qiang Meng, Jingyi Jiang, Bin Gao, (unknown), Yong Liu, (unknown), Changhe Wang, Hua Zhang	1
4	Molecular and Carbon Isotopic Compositions of Crude Oils from the Kekeya Area of the Southwest Depression, Tarim Basin: Implications for Oil Groups and Effective Sources 2024 ·Energies ·(unknown), Qilin Xiao, (unknown), (unknown), Haizhu Zhang, (unknown), (unknown), Zhanghu Wang, (unknown), Qiang Meng	1
5	Brain regions differences in amyloid-β and gene expression in early APP/PS1 mice and identification of<i> </i>Npas4 as a key molecule in Alzheimer's disease 2024 ·SHILAP Revista de lepidopterología ·(unknown), Zhong Zhao, Xiaoyan Wang, (unknown), (unknown), (unknown), Wenli Chen, Qiang Meng	1
6	Analysis of characteristics of movement disorders in patients with anti-N-methyl-D-aspartate receptor encephalitis 2024 ·Frontiers in Neurology ·Hongmei Li, (unknown), (unknown), Qiang Meng	1
7	Cooperation between neurovascular dysfunction and Aβ in Alzheimer’s disease 2023 ·Frontiers in Molecular Neuroscience ·Niya Wang, Yang Xiang, Zhong Zhao, Da Liu, Xiaoyan Wang, Hao Tang, (unknown), (unknown), Wenli Chen, Qiang Meng	3
8	Vanillin attenuates oxidative stress and neurochemical balance in MPTP-induced Parkinson’s disease mice by regulating the TLR-4 inflammatory pathway 2023 ·Folia Neuropathologica ·(unknown), Jiang Liu, Xiaoyan Wang, Qiang Meng	2
9	The role of CNPY2 in endothelial injury and inflammation during the progress of atherosclerosis 2023 ·Journal of Molecular Histology ·Hong Huang, Ning Tang, Yan Li, (unknown), Qiang Chen, Qiang Meng	2
10	Anti-NMDAR antibodies, the blood–brain barrier, and anti-NMDAR encephalitis 2023 ·Frontiers in Neurology ·(unknown), Niya Wang, (unknown), Ning Tang, Kunhua Wu, Qiang Meng	6
11	Bleeding due to successive duodenal and rectal ulcers in an 81-year-old patient with severe COVID-19: a case report 2023 ·BMC Geriatrics ·(unknown), Qiang Meng, Yunwei Li, Yiling Li, (unknown), Hongwen Zhao, Bing Dai	0
12	Integrated Genetics and Micronutrient Data to Inform the Causal Association Between Serum Calcium Levels and Ischemic Stroke 2020 ·Frontiers in Cell and Developmental Biology ·Qiang Meng, Lu Huang, Kai Tao, (unknown), (unknown), Wen Wang, Huaizhou Qin, Dayun Feng, Qing Cai	2
13	TLR2 Ligand Pam3CSK4 Regulates MMP-2/9 Expression by MAPK/NF-κB Signaling Pathways in Primary Brain Microvascular Endothelial Cells 2018 ·Neurochemical Research ·(unknown), (unknown), (unknown), Hao Fu, Qiang Meng	30
14	MMP-9 Upregulation is Attenuated by the Monoclonal TLR2 Antagonist T2.5 After Oxygen–Glucose Deprivation and Reoxygenation in Rat Brain Microvascular Endothelial Cells 2018 ·Journal of Stroke and Cerebrovascular Diseases ·(unknown), (unknown), Hao Fu, Qiang Meng	12
15	Hyperthermia exacerbates the effects of cathepsin L on claudin-1 in a blood-brain barrier model in vitro 2015 ·Brain Research ·Conghua Ji, Limin Wang, (unknown), Shan Li, Hui Yang, (unknown), Qiang Meng	5
16	Decreased Astroglial Monocarboxylate Transporter 4 Expression in Temporal Lobe Epilepsy 2014 ·Molecular Neurobiology ·Bei Liu, Le Niu, Mingzhi Shen, Lei Gao, Chao Wang, Jie Li, (unknown), Ye Tao, Qiang Meng, (unknown), Guodong Gao, Hua Zhang	26
17	Hyperthermia aggravates focal cerebral ischemia-reperfusion injury in rats by down-regulating claudin-1 expression 2013 ·Chunli Zhang, (unknown), Qiang Meng	1
18	Oxidative Parameters in the Rat Brain of Chronic Mild Stress Model for Depression: Relation to Anhedonia-Like Responses 2012 ·The Journal of Membrane Biology ·Chao Wang, Heming Wu, Xiaorong Jing, Qiang Meng, Bei Liu, Hua Zhang, Guodong Gao	24
19	Hyperthermia worsens ischaemic brain injury through destruction of microvessels in an embolic model in rats 2012 ·International Journal of Hyperthermia ·Qiang Meng, Chunyan He, Ashfaq Shuaib, Chen Xu Wang	12
20	Increased expression of urokinase‐type plasminogen activator receptor in the frontal cortex of patients with intractable frontal lobe epilepsy 2010 ·Journal of Neuroscience Research ·Bei Liu, Bin Zhang, Tao Wang, (unknown), Xiaorong Jing, Jun Zheng, Chao Wang, Qiang Meng, Liang Wang, Wei Wang, Heng Guo, Yu You, Hua Zhang, Guodong Gao	23

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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