Guihua Tu

525 total citations
18 papers, 472 citations indexed

About

Guihua Tu is a scholar working on Physiology, Neurology and Endocrine and Autonomic Systems. According to data from OpenAlex, Guihua Tu has authored 18 papers receiving a total of 472 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Physiology, 6 papers in Neurology and 5 papers in Endocrine and Autonomic Systems. Recurrent topics in Guihua Tu's work include Adenosine and Purinergic Signaling (12 papers), Neuroscience of respiration and sleep (5 papers) and Nerve injury and regeneration (4 papers). Guihua Tu is often cited by papers focused on Adenosine and Purinergic Signaling (12 papers), Neuroscience of respiration and sleep (5 papers) and Nerve injury and regeneration (4 papers). Guihua Tu collaborates with scholars based in China, United States and Singapore. Guihua Tu's co-authors include Shangdong Liang, Shuangmei Liu, Guilin Li, Haiying Peng, Yun Gao, Hong Xu, Changshui Xu, Jinyan Xie, Chaoran Zheng and Guodong Li and has published in prestigious journals such as Brain Behavior and Immunity, Neurobiology of Disease and Molecular and Cellular Neuroscience.

In The Last Decade

Guihua Tu

18 papers receiving 470 citations

Peers

Guihua Tu

PHYSI

CCM

Mehmet Uğur Türkiye

M. Grohmann Germany

Fiona Hanner United States

Kyohei Tokizane Japan

Ying-Fu Su United States

Hassina Massudi Australia

Jorge E. Guzman United States

Anna Litwiniuk Poland

Mostafa A. Rabie Egypt

Malika Winfield United States

Mehmet Uğur Türkiye

Guihua Tu

170 ×0.9

PHYSI

312 ×2.2

151 ×1.3

PHYSI

204 ×2.1

CCM

11 ×0.1

Citations per year, relative to Guihua Tu Guihua Tu (= 1×) peers Mehmet Uğur

Countries citing papers authored by Guihua Tu

Since Specialization

Citations

This map shows the geographic impact of Guihua Tu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Guihua Tu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Guihua Tu more than expected).

Fields of papers citing papers by Guihua Tu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Guihua Tu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Guihua Tu. The network helps show where Guihua Tu may publish in the future.

Co-authorship network of co-authors of Guihua Tu

This figure shows the co-authorship network connecting the top 25 collaborators of Guihua Tu. A scholar is included among the top collaborators of Guihua Tu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Guihua Tu. Guihua Tu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Tu, Guihua, Lifang Zou, Shuangmei Liu, et al.. (2020). Correction to: Long noncoding NONRATT021972 siRNA normalized abnormal sympathetic activity mediated by the upregulation of P2X7 receptor in superior cervical ganglia after myocardial ischemia. Purinergic Signalling. 16(4). 603–604. 2 indexed citations

Liu, Jianuo, Enquan Xu, Guihua Tu, et al.. (2017). Methamphetamine potentiates HIV-1gp120-induced microglial neurotoxic activity by enhancing microglial outward K + current. Molecular and Cellular Neuroscience. 82. 167–175. 12 indexed citations

Liu, Han, Jianuo Liu, Enquan Xu, et al.. (2016). Human immunodeficiency virus protein Tat induces oligodendrocyte injury by enhancing outward K+ current conducted by KV1.3. Neurobiology of Disease. 97(Pt A). 1–10. 15 indexed citations

Tu, Guihua, Lifang Zou, Shuangmei Liu, et al.. (2016). Long noncoding NONRATT021972 siRNA normalized abnormal sympathetic activity mediated by the upregulation of P2X7 receptor in superior cervical ganglia after myocardial ischemia. Purinergic Signalling. 12(3). 521–535. 44 indexed citations

Zou, Lifang, Guihua Tu, Shuangmei Liu, et al.. (2015). LncRNA NONRATT021972 involved the pathophysiologic processes mediated by P2X7 receptors in stellate ganglia after myocardial ischemic injury. Purinergic Signalling. 12(1). 127–137. 27 indexed citations

Kiyota, Tomomi, et al.. (2015). Presenilin-1 familial Alzheimer’s disease mutation alters hippocampal neurogenesis and memory function in CCL2 null mice. Brain Behavior and Immunity. 49. 311–321. 14 indexed citations

Tang, Xiaoli, Libin Deng, Huangui Xiong, et al.. (2014). Expression profile of mitrogen-activated protein kinase (MAPK) signaling genes in the skeletal muscle & liver of rat with type 2 diabetes: role in disease pathology.. PubMed. 140(6). 744–55. 9 indexed citations

Tu, Guihua, Guilin Li, Haiying Peng, et al.. (2013). P2X7 inhibition in stellate ganglia prevents the increased sympathoexcitatory reflex via sensory-sympathetic coupling induced by myocardial ischemic injury. Brain Research Bulletin. 96. 71–85. 42 indexed citations

Kong, Fanjun, Shuangmei Liu, Changshui Xu, et al.. (2013). Electrophysiological studies of upregulated P2X7 receptors in rat superior cervical ganglia after myocardial ischemic injury. Neurochemistry International. 63(3). 230–237. 31 indexed citations

10.

Li, Guilin, Haiying Peng, Guihua Tu, et al.. (2013). Sensory–sympathetic coupling in superior cervical ganglia after myocardial ischemic injury facilitates sympathoexcitatory action via P2X7 receptor. Purinergic Signalling. 9(3). 463–479. 39 indexed citations

11.

Xu, Hong, Bing Wu, Ping Zhang, et al.. (2013). High fatty acids modulate P2X7 expression and IL-6 release via the p38 MAPK pathway in PC12 cells. Brain Research Bulletin. 94. 63–70. 30 indexed citations

12.

Li, Guilin, Hong Xu, Shuangmei Liu, et al.. (2012). Effects of neferine on CCL5 and CCR5 expression in SCG of type 2 diabetic rats. Brain Research Bulletin. 90. 79–87. 31 indexed citations

13.

Zhang, Jun, Xin Li, Yun Gao, et al.. (2012). Effects of puerarin on the inflammatory role of burn-related procedural pain mediated by P2X7 receptors. Burns. 39(4). 610–618. 27 indexed citations

14.

Liu, Shuangmei, Changshui Xu, Guilin Li, et al.. (2012). Vatalanib decrease the positive interaction of VEGF receptor-2 and P2X2/3 receptor in chronic constriction injury rats. Neurochemistry International. 60(6). 565–572. 20 indexed citations

15.

Li, Guilin, Gaochun Zhu, Yun Gao, et al.. (2012). Neferine Inhibits the Upregulation of CCL5 and CCR5 in Vascular Endothelial Cells During Chronic High Glucose Treatment. Inflammation. 36(2). 300–308. 20 indexed citations

16.

Li, Xin, Guilin Li, Hong Xu, et al.. (2011). Effects of anti-rVEGF on the expression of VEGF receptor-2 and P2X2/3 receptors of the spinal dorsal horn in neuropathic pain rats. Brain Research Bulletin. 87(2-3). 227–233. 11 indexed citations

17.

Li, Guilin, Shuangmei Liu, Yang Yang, et al.. (2011). Effects of oxymatrine on sympathoexcitatory reflex induced by myocardial ischemic signaling mediated by P2X3 receptors in rat SCG and DRG. Brain Research Bulletin. 84(6). 419–424. 41 indexed citations

18.

Xu, Changshui, Hong Xu, Wei Xiong, et al.. (2011). Role of puerarin in the signalling of neuropathic pain mediated by P2X3 receptor of dorsal root ganglion neurons. Brain Research Bulletin. 87(1). 37–43. 57 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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