Pierre Fons

1.4k total citations
15 papers, 595 citations indexed

About

Pierre Fons is a scholar working on Molecular Biology, Oncology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Pierre Fons has authored 15 papers receiving a total of 595 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Oncology and 4 papers in Cellular and Molecular Neuroscience. Recurrent topics in Pierre Fons's work include Angiogenesis and VEGF in Cancer (7 papers), Adenosine and Purinergic Signaling (4 papers) and Cancer, Hypoxia, and Metabolism (4 papers). Pierre Fons is often cited by papers focused on Angiogenesis and VEGF in Cancer (7 papers), Adenosine and Purinergic Signaling (4 papers) and Cancer, Hypoxia, and Metabolism (4 papers). Pierre Fons collaborates with scholars based in France, United Kingdom and Austria. Pierre Fons's co-authors include Françoise Bono, Pierre Savi, Jean-Pascal Hérault, Jean‐Marc Herbert, Benoı̂t Favier, Antoine Alam, Jacques Bonnin, Pauline Barron, Marie Mandron and Gera Neufeld and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Oncology and Blood.

In The Last Decade

Pierre Fons

15 papers receiving 580 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Pierre Fons France 9 318 228 198 126 69 15 595
L. M. Gardner United States 13 497 1.6× 305 1.3× 138 0.7× 91 0.7× 52 0.8× 13 1.0k
Kayo Fujisawa Japan 12 243 0.8× 104 0.5× 143 0.7× 45 0.4× 57 0.8× 18 508
Rika Okamoto Japan 9 156 0.5× 229 1.0× 170 0.9× 18 0.1× 66 1.0× 14 469
Laura Bald United States 14 487 1.5× 159 0.7× 175 0.9× 19 0.2× 66 1.0× 16 855
Heiner Renneberg Germany 15 213 0.7× 109 0.5× 85 0.4× 51 0.4× 102 1.5× 22 562
Swathi Yada Singapore 8 270 0.8× 107 0.5× 232 1.2× 8 0.1× 92 1.3× 11 586
G. de Gregorio Italy 12 387 1.2× 44 0.2× 141 0.7× 40 0.3× 43 0.6× 27 624
Xingjiang Yu China 14 479 1.5× 229 1.0× 148 0.7× 14 0.1× 30 0.4× 29 801
Eric C. Yang Canada 8 182 0.6× 92 0.4× 132 0.7× 13 0.1× 42 0.6× 14 411
Antonia Boyer United States 8 782 2.5× 212 0.9× 134 0.7× 19 0.2× 35 0.5× 14 1.0k

Countries citing papers authored by Pierre Fons

Since Specialization
Citations

This map shows the geographic impact of Pierre Fons's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pierre Fons with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pierre Fons more than expected).

Fields of papers citing papers by Pierre Fons

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pierre Fons. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pierre Fons. The network helps show where Pierre Fons may publish in the future.

Co-authorship network of co-authors of Pierre Fons

This figure shows the co-authorship network connecting the top 25 collaborators of Pierre Fons. A scholar is included among the top collaborators of Pierre Fons based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pierre Fons. Pierre Fons is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Fons, Pierre, Mark Whittaker, Klaus Hackner, et al.. (2022). Abstract 4150: Enriching for adenosine antagonist patient responses through deep learning. Cancer Research. 82(12_Supplement). 4150–4150. 1 indexed citations
2.
Payne, Andrew, Pierre Fons, Florie Bertrand, et al.. (2021). Abstract 1731: EXS21546, a non-CNS penetrant A2AR-selective antagonist for anti-cancer immunotherapy. Cancer Research. 81(13_Supplement). 1731–1731. 1 indexed citations
3.
Fons, Pierre, Michaël R. Paillasse, Virgile Visentin, et al.. (2019). Abstract 4079: EVT801: a selective VEGFR3 inhibitor with the potential for combination with immune-checkpoint therapies, preclinical evidences and plans for first-in-human evaluation. Cancer Research. 79(13_Supplement). 4079–4079. 1 indexed citations
4.
Fons, Pierre, Florie Bertrand, Adrian Schreyer, et al.. (2018). Abstract 3768: Identification of a novel non-brain penetrant A2AR inhibitor and proof-of-concept of CD73 and A2AR/CD73 small-molecule inhibitors for cancer immunotherapy. Cancer Research. 78(13_Supplement). 3768–3768. 3 indexed citations
5.
Delaunay, Myriam, Nicolas Guibert, Nadia Boubekeur, et al.. (2018). Baseline circulating myeloid-derived suppressor cells and response to PD-1 inhibitor in non-small cell lung cancer patients.. Journal of Clinical Oncology. 36(5_suppl). 145–145. 10 indexed citations
6.
Fons, Pierre, Pauline Barron, Antoine Alam, et al.. (2017). Abstract 2634: EVT801: Standalone cancer immunotherapy in VEGFR3+ tumors and combination with immune checkpoint therapies in VEGFR3- tumors. Cancer Research. 77(13_Supplement). 2634–2634. 1 indexed citations
7.
Fons, Pierre, Michaël R. Paillasse, Adrian Schreyer, et al.. (2017). Abstract 3970: Targeting the adenosine immunosuppressive pathway for cancer immunotherapy with small molecule agents. Cancer Research. 77(13_Supplement). 3970–3970. 5 indexed citations
8.
Fons, Pierre, Jean-Pascal Hérault, Frédérique Dol, et al.. (2014). Tumor Vasculature Is Regulated by FGF/FGFR Signaling‐Mediated Angiogenesis and Bone Marrow‐Derived Cell Recruitment: This Mechanism Is Inhibited by SSR128129E, the First Allosteric Antagonist of FGFRs. Journal of Cellular Physiology. 230(1). 43–51. 22 indexed citations
9.
Delcourt, Nicolas, Celia Quevedo, Christelle Nonne, et al.. (2014). Targeted Identification of Sialoglycoproteins in Hypoxic Endothelial Cells and Validation in Zebrafish Reveal Roles for Proteins in Angiogenesis. Journal of Biological Chemistry. 290(6). 3405–3417. 24 indexed citations
10.
Fons, Pierre, David Sibrac, Bruno Gautret, et al.. (2013). Automated Image Analysis of In Vitro Angiogenesis Assay. SLAS TECHNOLOGY. 18(5). 411–415. 9 indexed citations
11.
Bouteiller, Philippe Le, Pierre Fons, Françoise Bono, et al.. (2007). Soluble HLA-G and control of angiogenesis. Journal of Reproductive Immunology. 76(1-2). 17–22. 42 indexed citations
12.
Favier, Benoı̂t, Antoine Alam, Pauline Barron, et al.. (2006). Neuropilin-2 interacts with VEGFR-2 and VEGFR-3 and promotes human endothelial cell survival and migration. Blood. 108(4). 1243–1250. 234 indexed citations
13.
Fons, Pierre, Sophie Chabot, Judith E. Cartwright, et al.. (2006). Soluble HLA-G1 inhibits angiogenesis through an apoptotic pathway and by direct binding to CD160 receptor expressed by endothelial cells. Blood. 108(8). 2608–2615. 159 indexed citations
14.
Alam, Antoine, Jean-Pascal Hérault, Pauline Barron, et al.. (2004). Heterodimerization with vascular endothelial growth factor receptor-2 (VEGFR-2) is necessary for VEGFR-3 activity. Biochemical and Biophysical Research Communications. 324(2). 909–915. 54 indexed citations
15.
Fons, Pierre, et al.. (2004). VEGF‐R2 and neuropilin‐1 are involved in VEGF‐A‐induced differentiation of human bone marrow progenitor cells. Journal of Cellular Physiology. 200(3). 351–359. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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