Philomena Kamya

825 total citations
7 papers, 311 citations indexed

About

Philomena Kamya is a scholar working on Immunology, Virology and Molecular Biology. According to data from OpenAlex, Philomena Kamya has authored 7 papers receiving a total of 311 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Immunology, 3 papers in Virology and 1 paper in Molecular Biology. Recurrent topics in Philomena Kamya's work include Immune Cell Function and Interaction (7 papers), T-cell and B-cell Immunology (6 papers) and Reproductive System and Pregnancy (4 papers). Philomena Kamya is often cited by papers focused on Immune Cell Function and Interaction (7 papers), T-cell and B-cell Immunology (6 papers) and Reproductive System and Pregnancy (4 papers). Philomena Kamya collaborates with scholars based in Canada, United States and Spain. Philomena Kamya's co-authors include Salix Boulet, Nicole F. Bernard, Christos Tsoukas, Jean‐Pierre Routy, Rujun Song, Julie Bruneau, Marianna Kleyman, Julie Bruneau, Naglaa H. Shoukry and Cécile Tremblay and has published in prestigious journals such as The Journal of Immunology, Journal of Virology and AIDS.

In The Last Decade

Philomena Kamya

6 papers receiving 305 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Philomena Kamya Canada 6 278 179 45 30 12 7 311
James Tang United States 4 117 0.4× 171 1.0× 75 1.7× 40 1.3× 5 0.4× 5 204
Philippe Rascle France 9 200 0.7× 159 0.9× 54 1.2× 53 1.8× 5 0.4× 12 268
Eshia Moodley United States 6 178 0.6× 210 1.2× 79 1.8× 42 1.4× 4 0.3× 7 244
Jean-Pierre Routy Canada 7 193 0.7× 196 1.1× 95 2.1× 75 2.5× 7 0.6× 12 310
Anne Nolting United States 4 276 1.0× 140 0.8× 18 0.4× 31 1.0× 12 1.0× 5 295
Z W Chen United States 12 308 1.1× 225 1.3× 69 1.5× 80 2.7× 11 0.9× 13 380
Thalía García-Téllez France 5 160 0.6× 123 0.7× 39 0.9× 44 1.5× 2 0.2× 6 213
Nina Censoplano United States 6 95 0.3× 89 0.5× 34 0.8× 38 1.3× 17 1.4× 7 172
Natalia Olejniczak United Kingdom 9 90 0.3× 139 0.8× 82 1.8× 47 1.6× 3 0.3× 12 200
Valerie Varner United States 9 286 1.0× 75 0.4× 38 0.8× 83 2.8× 11 0.9× 13 341

Countries citing papers authored by Philomena Kamya

Since Specialization
Citations

This map shows the geographic impact of Philomena Kamya's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Philomena Kamya with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Philomena Kamya more than expected).

Fields of papers citing papers by Philomena Kamya

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Philomena Kamya. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Philomena Kamya. The network helps show where Philomena Kamya may publish in the future.

Co-authorship network of co-authors of Philomena Kamya

This figure shows the co-authorship network connecting the top 25 collaborators of Philomena Kamya. A scholar is included among the top collaborators of Philomena Kamya based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Philomena Kamya. Philomena Kamya is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Ndhlovu, Zaza M., Philomena Kamya, Thandeka Nkosi, et al.. (2014). Massive Activation, Expansion and Subsequent Apoptosis of CD8 T-cells in Acute HIV Infection. AIDS Research and Human Retroviruses. 30(S1). A74–A75. 1 indexed citations
2.
Kamya, Philomena, Carlos E Melendez-Peña, Matthew S. Parsons, et al.. (2012). Inhibitory Killer Immunoglobulin-like receptors to self HLA-B and HLA-C ligands contribute differentially to Natural Killer cell functional potential in HIV infected slow progressors. Clinical Immunology. 143(3). 246–255. 11 indexed citations
3.
Kamya, Philomena, Salix Boulet, Christos Tsoukas, et al.. (2011). Receptor-Ligand Requirements for Increased NK Cell Polyfunctional Potential in Slow Progressors Infected with HIV-1 Coexpressing KIR3DL1*h/*y and HLA-B*57. Journal of Virology. 85(12). 5949–5960. 77 indexed citations
4.
Kamya, Philomena, Christos Tsoukas, Salix Boulet, et al.. (2011). T cell Activation does not drive CD4 decline in longitudinally followed HIV-infected Elite Controllers. AIDS Research and Therapy. 8(1). 20–20. 18 indexed citations
5.
Kamya, Philomena, Salix Boulet, Yoav Peretz, et al.. (2010). Changes in Function of HIV-Specific T-Cell Responses with Increasing Time from Infection. Viral Immunology. 23(2). 159–168. 5 indexed citations
6.
Boulet, Salix, Rujun Song, Philomena Kamya, et al.. (2010). HIV Protective KIR3DL1 and HLA-B Genotypes Influence NK Cell Function Following Stimulation with HLA-Devoid Cells. The Journal of Immunology. 184(4). 2057–2064. 84 indexed citations
7.
Boulet, Salix, Marianna Kleyman, Philomena Kamya, et al.. (2008). A combined genotype of KIR3DL1 high expressing alleles and HLA-B*57 is associated with a reduced risk of HIV infection. AIDS. 22(12). 1487–1491. 115 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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